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1. Full Text Urea-induced ROS cause endothelial dysfunction in chronic renal failure
by D'Apolito, Maria and Du, Xueliang and Pisanelli, Daniela and Pettoello-Mantovani, Massimo and Campanozzi, Angelo and Giacco, Ferdinando and Maffione, Angela Bruna and Colia, Anna Laura and Brownlee, Michael and Giardino, Ida
Atherosclerosis, ISSN 0021-9150, 2015, Volume 239, Issue 2, pp. 393 - 400
Cardiovascular | MnTBAP | chronic renal failure | UCP-1 | Endothelial cells | uncoupling protein 1 | Urea | O-linked-N-acetylglucosamine | manganese tetrakis (4-benzoic acid) porphyrin | CRF | ROS | reactive oxygen species | GlcNAc | Prostacyclin synthase | Reactive oxygen species | Chronic renal failure | Manganese tetrakis (4-benzoic acid) porphyrin | Uncoupling protein 1 | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Humans | Cytochrome P-450 Enzyme System - metabolism | Endothelium, Vascular - drug effects | Aorta - metabolism | Urea - chemistry | Antigens, CD - metabolism | Endoglin | Atherosclerosis - enzymology | Kidney Failure, Chronic - chemically induced | Chemokine CCL2 - metabolism | Endothelium - enzymology | Intramolecular Oxidoreductases - metabolism | Interleukin-6 - metabolism | Superoxide Dismutase - metabolism | Atherosclerosis - physiopathology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Random Allocation | Atherosclerosis - metabolism | Kidney Failure, Chronic - metabolism | Catalase - metabolism | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - metabolism | Animals | Endothelium, Vascular - pathology | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Chronic kidney failure | Atherosclerosis | Endothelium | Index Medicus
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2. Full Text XPO1-dependent nuclear export is a druggable vulnerability in KRAS-mutant lung cancer
by Kim, Jimi and McMillan, Elizabeth and Kim, Hyun Seok and Venkateswaran, Niranjan and Makkar, Gurbani and Rodriguez-Canales, Aime and Villalobos, Pamela and Neggers, Jasper Edgar and Mendiratta, Saurabh and Wei, Shuguang and Landesman, Yosef and Senapedis, William and Baloglu, Erkan and Chow, Chi-Wan B and Frink, Robin E and Gao, Boning and Roth, Michael and Minna, John D and Daelemans, Dirk and Wistuba, Ignacio I and Posner, Bruce A and Scaglioni, Pier Paolo and White, Michael A
Nature (London), ISSN 0028-0836, 09/2016, Volume 538, Issue 7623, pp. 114 - 117
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Lung Neoplasms - drug therapy | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Cell Survival - genetics | Lung Neoplasms - pathology | NF-kappa B - metabolism | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Cell Nucleus - metabolism | RNA Interference | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Female | Verteporfin | Protein-Serine-Threonine Kinases - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Follistatin-Related Proteins - genetics | Lung Neoplasms - genetics | Cell Survival - drug effects | NF-kappa B - antagonists & inhibitors | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Porphyrins - pharmacology | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Animals | Karyopherins - metabolism | Active Transport, Cell Nucleus - drug effects | Genes, Lethal - genetics | NF-KappaB Inhibitor alpha - metabolism | Nuclear Proteins - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | RNA, Small Interfering | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Cell Nucleus - drug effects | Karyopherins - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - metabolism | Care and treatment | Genetic aspects | Gene mutations | Health aspects | Guanosine triphosphatase | Lung cancer | Genomics | Genomes | Kinases | Drug resistance | Gene expression | Drug dosages | Index Medicus
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3. Full Text Reactive oxygen species/oxidative stress contributes to progression of kidney fibrosis following transient ischemic injury in mice
by Jinu Kim and Young Mi Seok and Kyong-Jin Jung and Kwon Moo Park
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 08/2009, Volume 297, Issue 2, pp. 461 - 470
Ischemia-reperfusion | α-smooth muscle actin | Superoxide | Manganese(III) tetrakis(1-methyl-4-pyridyl) porphyrin | Interstitial fibrosis | Physiology | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Kidney - blood supply | Reactive Oxygen Species - metabolism | Kidney - pathology | Apoptosis - drug effects | S100 Calcium-Binding Protein A4 | Actins - metabolism | Male | Metalloporphyrins - pharmacology | Tyrosine - analogs & derivatives | Antigens, CD - metabolism | Kidney - metabolism | Time Factors | Lipid Peroxidation - drug effects | Reperfusion Injury - metabolism | Ischemia - pathology | Superoxide Dismutase - metabolism | Disease Models, Animal | Kidney - drug effects | Oxidation-Reduction | Reperfusion Injury - pathology | Ischemia - metabolism | S100 Proteins - metabolism | Antioxidants - pharmacology | Disease Progression | Hydrogen Peroxide - metabolism | Collagen - metabolism | Tyrosine - metabolism | Animals | Reperfusion Injury - prevention & control | Antigens, Differentiation, Myelomonocytic - metabolism | Fibrosis | Glucosephosphate Dehydrogenase - metabolism | HSP47 Heat-Shock Proteins - metabolism | Mice | Mice, Inbred BALB C | Oxidative Stress - drug effects | Proliferating Cell Nuclear Antigen - metabolism | Vitamin E - pharmacology | Proteins | Studies | Rodents | Oxidation | Kidney diseases | Gene expression | Cells | Index Medicus
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4. Full Text TMEM14C is required for erythroid mitochondrial heme metabolism
by Yien, Yvette Y and Robledo, Raymond F and Schultz, Iman J and Takahashi-Makise, Naoko and Gwynn, Babette and Bauer, Daniel E and Dass, Abhishek and Yi, Gloria and Li, Liangtao and Hildick-Smith, Gordon J and Cooney, Jeffrey D and Pierce, Eric L and Mohler, Kyla and Dailey, Tamara A and Miyata, Non and Kingsley, Paul D and Garone, Caterina and Hattangadi, Shilpa M and Huang, Hui and Chen, Wen and Keenan, Ellen M and Shah, Dhvanit I and Schlaeger, Thorsten M and Dimauro, Salvatore and Orkin, Stuart H and Cantor, Alan B and Palis, James and Koehler, Carla M and Lodish, Harvey F and Kaplan, Jerry and Ward, Diane M and Dailey, Harry A and Phillips, John D and Peters, Luanne L and Paw, Barry H
The Journal of clinical investigation, ISSN 0021-9738, 10/2014, Volume 124, Issue 10, pp. 4294 - 4304
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Mitochondria | Cell metabolism | Genetic research | Genetic aspects | Research | Gene expression | Observations | Proteins | Studies | Enzymes | Genes | Rodents | Cloning | Stem cells | Metabolism | Laboratory animals | Index Medicus | Abridged Index Medicus
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5. Full Text Genetic and pharmacological disruption of the TEAD-YAP complex suppresses the oncogenic activity of YAP
by Liu-Chittenden, Yi and Huang, Bo and Shim, Joong Sup and Chen, Qian and Lee, Se-Jin and Anders, Robert A and Liu, Jun O and Pan, Duojia
Genes & development, ISSN 0890-9369, 06/2012, Volume 26, Issue 12, pp. 1300 - 1305
Chemical biology | YAP | Hippo signaling | Oncogene | Genetics & Heredity | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Biology | Hepatomegaly - pathology | Liver - pathology | Humans | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Multiprotein Complexes - metabolism | Liver - drug effects | Protein Binding - drug effects | HEK293 Cells | Verteporfin | Neurofibromin 2 - metabolism | Liver - metabolism | Mice, Transgenic | Phosphoproteins - genetics | Porphyrins - pharmacology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Transcription Factors - metabolism | Phenotype | Animals | Adaptor Proteins, Signal Transducing - genetics | Hepatomegaly - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Cell Transformation, Neoplastic - pathology | Genes, Dominant - genetics | Cell Cycle Proteins | Index Medicus | Research Communication | chemical biology | oncogene
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6. Full Text Synthetic ion transporters can induce apoptosis by facilitating chloride anion transport into cells
by Ko, Sung-Kyun and Kim, Sung Kuk and Share, Andrew and Lynch, Vincent M and Park, Jinhong and Namkung, Wan and Van Rossom, Wim and Busschaert, Nathalie and Gale, Philip A and Sessler, Jonathan L and Shin, Injae
Nature chemistry, ISSN 1755-4330, 10/2014, Volume 6, Issue 10, pp. 885 - 892
Physical Sciences | Chemistry | Chemistry, Multidisciplinary | Science & Technology | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Pyridines - chemistry | Humans | Porphyrins - chemistry | Small Molecule Libraries - metabolism | Chlorides - chemistry | Porphyrins - metabolism | Anion Transport Proteins - chemistry | Calixarenes - chemistry | Caspases - metabolism | Amino Acid Chloromethyl Ketones - metabolism | Ion Transport | Caspases - chemistry | Cell Line | Chlorides - pharmacology | Amino Acid Chloromethyl Ketones - chemistry | Cytochromes c - metabolism | HCT116 Cells | Rats | Mitochondria - metabolism | Anion Transport Proteins - metabolism | Calixarenes - metabolism | Liposomes - chemistry | Animals | Diamide - chemistry | HeLa Cells | Index Medicus
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7. Full Text Verteporfin suppresses cell survival, angiogenesis and vasculogenic mimicry of pancreatic ductal adenocarcinoma via disrupting the YAP‐TEAD complex
by Wei, Honglong and Wang, Fuhai and Wang, Yong and Li, Tao and Xiu, Peng and Zhong, Jingtao and Sun, Xueying and Li, Jie
Cancer science, ISSN 1347-9032, 03/2017, Volume 108, Issue 3, pp. 478 - 487
Angiogenesis | apoptosis | pancreatic cancer | vasculogenic mimicry | verteporfin | Life Sciences & Biomedicine | Oncology | Science & Technology | Human Umbilical Vein Endothelial Cells | Cyclin D1 - metabolism | Apoptosis - drug effects | Humans | Male | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Proto-Oncogene Proteins c-bcl-2 - metabolism | Vesicular Transport Proteins - antagonists & inhibitors | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Verteporfin | Antineoplastic Agents - pharmacology | Cell Survival - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Matrix Metalloproteinase 2 - metabolism | Pancreatic Neoplasms - pathology | Oncogene Proteins - metabolism | Nuclear Proteins - metabolism | Poly (ADP-Ribose) Polymerase-1 - metabolism | Transcription Factors - antagonists & inhibitors | Porphyrins - pharmacology | Carcinoma, Pancreatic Ductal - pathology | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Cadherins - antagonists & inhibitors | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Cyclin E - metabolism | Adaptor Proteins, Signal Transducing - metabolism | G1 Phase Cell Cycle Checkpoints - drug effects | Antigens, CD | Adenocarcinoma | Proteins | Photochemotherapy | Analysis | Pancreatic cancer | Cancer | Cell proliferation | Metastasis | Cancer therapies | Mimicry | Cell adhesion & migration | Macular degeneration | Cell cycle | Xenografts | G1 phase | Cell survival | Photodynamic therapy | Poly(ADP-ribose) polymerase | Gene expression | Cadherin | Gelatinase A | Yes-associated protein | Microscopy | Medical prognosis | Stem cells | Hypoxia | Apoptosis | Tumors | Index Medicus | Original
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8. Full Text YAP activation is an early event and a potential therapeutic target in liver cancer development
by Perra, Andrea and Kowalik, Marta Anna and Ghiso, Elena and Ledda-Columbano, Giovanna Maria and Di Tommaso, Luca and Angioni, Maria Maddalena and Raschioni, Carlotta and Testore, Elena and Roncalli, Massimo and Giordano, Silvia and Columbano, Amedeo
Journal of hepatology, ISSN 0168-8278, 2014, Volume 61, Issue 5, pp. 1088 - 1096
Gastroenterology and Hepatology | Preneoplastic stages | HCC | Verteporfin | TCPOBOP | Hippo pathway | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Adenoma, Liver Cell - drug therapy | Precancerous Conditions - etiology | Adenoma, Liver Cell - etiology | Humans | Middle Aged | Rats, Inbred F344 | Male | MicroRNAs - metabolism | Precancerous Conditions - metabolism | Phosphoproteins - metabolism | Liver Neoplasms - etiology | Precancerous Conditions - drug therapy | Young Adult | Liver Neoplasms, Experimental - metabolism | Carcinoma, Hepatocellular - drug therapy | Apoptosis Regulatory Proteins - genetics | Adult | Female | Antineoplastic Agents - pharmacology | Carcinoma, Hepatocellular - etiology | Protein-Serine-Threonine Kinases - metabolism | Gene Expression | Signal Transduction | Adenoma, Liver Cell - metabolism | Liver Neoplasms - drug therapy | Rats | Porphyrins - pharmacology | Liver Neoplasms, Experimental - etiology | Apoptosis Regulatory Proteins - metabolism | Animals | Liver Neoplasms, Experimental - drug therapy | Apoptosis Regulatory Proteins - antagonists & inhibitors | Liver Neoplasms - metabolism | Cell Line, Tumor | Aged | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Transcription Factors | Adaptor Proteins, Signal Transducing - metabolism | Cell Cycle Proteins | Carcinoma, Hepatocellular - metabolism | Liver cancer | Health aspects | Index Medicus
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9. Full Text Cancer Cell Membrane Camouflaged Cascade Bioreactor for Cancer Targeted Starvation and Photodynamic Therapy
by Li, Shi-Ying and Cheng, Hong and Xie, Bo-Ru and Qiu, Wen-Xiu and Zeng, Jing-Yue and Li, Chu-Xin and Wan, Shuang-Shuang and Zhang, Lu and Liu, Wen-Long and Zhang, Xian-Zheng
ACS nano, ISSN 1936-0851, 07/2017, Volume 11, Issue 7, pp. 7006 - 7018
homotypic targeting | photodynamic therapy | starvation therapy | cell membrane | metal-organic framework | Physical Sciences | Chemistry | Materials Science | Nanoscience & Nanotechnology | Technology | Materials Science, Multidisciplinary | Science & Technology - Other Topics | Chemistry, Multidisciplinary | Chemistry, Physical | Science & Technology | Neoplasms - metabolism | Catalase - therapeutic use | Photosensitizing Agents - chemistry | Photosensitizing Agents - therapeutic use | Humans | Porphyrins - chemistry | Cercopithecus aethiops | Metal-Organic Frameworks - chemistry | Photochemotherapy | Oxygen - metabolism | Cell Membrane - metabolism | Cell Membrane - drug effects | Glucose Oxidase - therapeutic use | Glucose Oxidase - metabolism | Porphyrins - therapeutic use | Hydrogen Peroxide - metabolism | Neoplasms - drug therapy | Catalase - metabolism | Animals | Cell Line, Tumor | Glucose - metabolism | RAW 264.7 Cells | Mice | Mice, Inbred BALB C | COS Cells | Metal-Organic Frameworks - therapeutic use | Index Medicus
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