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Nature Medicine, ISSN 1078-8956, 07/2011, Volume 17, Issue 7, pp. 822 - 829
Journal Article
Journal Article
Experimental Neurology, ISSN 0014-4886, 2007, Volume 203, Issue 1, pp. 42 - 54
Paclitaxel-induced peripheral neuropathy (PN) can be a significant problem for patients receiving chemotherapeutic regimens for the treatment of breast,... 
Axonopathy | Neurotoxicity | Breast cancer | Ganglionopathy | Neuropathic pain | Microglia | axonopathy | TACTILE ALLODYNIA | breast cancer | SPINAL-CORD | SATELLITE CELLS | OVARIAN-CANCER | BONE CANCER PAIN | ACTIVATING TRANSCRIPTION FACTOR-3 | ganglionopathy | DORSAL-ROOT GANGLION | NEUROSCIENCES | NERVE GROWTH-FACTOR | neuropathic pain | neurotoxicity | MESSENGER-RNA | SCIATIC-NERVE | microglia | Hyperalgesia - chemically induced | Microglia - metabolism | Posterior Horn Cells - drug effects | Peripheral Nervous System Diseases - chemically induced | Posterior Horn Cells - metabolism | Injections, Intravenous | Antigens, Differentiation, Myelomonocytic - drug effects | Male | CD11b Antigen | Peripheral Nervous System Diseases - physiopathology | Glial Fibrillary Acidic Protein - metabolism | Peripheral Nerves - drug effects | Satellite Cells, Perineuronal - metabolism | Antigens, CD - metabolism | Chemotaxis, Leukocyte - physiology | Hyperalgesia - pathology | Activating Transcription Factor 3 - drug effects | Paclitaxel - toxicity | Chemotaxis, Leukocyte - drug effects | Microglia - pathology | Neurons, Afferent - pathology | Peripheral Nervous System Diseases - pathology | Disease Models, Animal | Schwann Cells - drug effects | Neurons, Afferent - drug effects | Microglia - drug effects | Rats | Schwann Cells - pathology | Schwann Cells - metabolism | Antigens, CD - drug effects | Activating Transcription Factor 3 - metabolism | Peripheral Nerves - pathology | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Peripheral Nerves - physiopathology | Antineoplastic Agents, Phytogenic - toxicity | Satellite Cells, Perineuronal - drug effects | Macrophages - metabolism | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Glial Fibrillary Acidic Protein - drug effects | Ganglia, Spinal - pathology | Posterior Horn Cells - pathology | Macrophages - drug effects | Neurons, Afferent - metabolism | Ganglia, Spinal - drug effects | Satellite Cells, Perineuronal - pathology | Ganglia, Spinal - metabolism | Antimitotic agents | Chemotherapy | Infiltration (Military science) | Neurosciences | Neurons | Oncology, Experimental | Lung cancer | Research | Macrophages | Antineoplastic agents | Intermediate filament proteins | Cancer
Journal Article
Nature Medicine, ISSN 1078-8956, 03/2017, Volume 23, Issue 3, pp. 355 - 360
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2009, Volume 29, Issue 21, pp. 6945 - 6954
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2009, Volume 106, Issue 27, pp. 11330 - 11335
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e36355
Calcitonin gene-related peptide (CGRP alpha, encoded by Calca) is a classic marker of nociceptive dorsal root ganglia (DRG) neurons. Despite years of research,... 
GENE-RELATED-PEPTIDE | RAT SPINAL-CORD | DIFFERENTIAL EXPRESSION | NOCICEPTIVE NEURONS | PRIMARY AFFERENT NEURONS | BIOLOGY | CENTRAL-NERVOUS-SYSTEM | BETA-CGRP | PROSTATIC ACID-PHOSPHATASE | HORN NEURONS | DORSAL-ROOT GANGLION | Histamine - toxicity | Immunohistochemistry | Chloroquine - toxicity | Posterior Horn Cells - drug effects | Posterior Horn Cells - metabolism | Skin - metabolism | Humans | Male | Ganglia, Spinal - cytology | Green Fluorescent Proteins - genetics | Pain - chemically induced | Brain - metabolism | Pruritus - physiopathology | TRPV Cation Channels - metabolism | Sensory Receptor Cells - physiology | Female | Sensory Receptor Cells - metabolism | Calcitonin Gene-Related Peptide - metabolism | Muscles - metabolism | Pruritus - chemically induced | Green Fluorescent Proteins - metabolism | Mice, Inbred C57BL | Capsaicin - toxicity | Cells, Cultured | Ganglia, Spinal - physiology | Calcitonin Gene-Related Peptide - genetics | Mice, Transgenic | Brain - drug effects | Microscopy, Confocal | Animals | Sensory Receptor Cells - drug effects | Pain - physiopathology | Muscles - drug effects | Muscles - innervation | Plant Oils - toxicity | Skin - innervation | Mice | Mustard Plant - toxicity | TRPM Cation Channels - metabolism | Posterior Horn Cells - physiology | Skin - drug effects | Ganglia, Spinal - metabolism | Cell culture | Brain | Spinal cord | Neurosciences | Circuits | Capsaicin | Chloroquine | Fluorescence | Capsaicin receptors | pH effects | Phosphatase | Pain | Dorsal root ganglia | Transgenic animals | Rodents | Animal tissues | Calcitonin | pH | Sensory neurons | Physiology | Menthol | Diphtheria toxin | Thyroid | Dorsal horn | Nerve endings | Motor neurons | Pain perception | Neurons | Epidermis | Light emitting diodes | Anatomy & physiology | Diphtheria | Ablation | Ganglia | Calcitonin gene-related peptide | Histamine | Green fluorescent protein | In vivo methods and tests | Mustard | Axon guidance | Acid phosphatase | Stimuli | transient receptor potential proteins | Oil | Temperature effects | Cell body
Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 12/2008, Volume 586, Issue 23, pp. 5701 - 5715
Most spinal cord injury (SCI) patients suffer from chronic pain. Effective therapy for this pain is lacking, and the underlying mechanisms are poorly... 
PERIPHERAL-NERVE INJURY | SUBSTANTIA-GELATINOSA NEURONS | LAMINA-I NEURONS | PHYSIOLOGY | MICROGLIA | ANION GRADIENT | SYNAPTIC-TRANSMISSION | UP-REGULATION | CONTRIBUTES | NEUROSCIENCES | AFFERENT-FIBERS | CENTRAL NEUROPATHIC PAIN | Immunohistochemistry | Posterior Horn Cells - drug effects | Posterior Horn Cells - metabolism | Electric Stimulation | Spinal Cord - drug effects | Spinal Cord - metabolism | Synaptic Transmission - physiology | Male | Pain - metabolism | Excitatory Postsynaptic Potentials - drug effects | Excitatory Postsynaptic Potentials - physiology | Nerve Fibers - physiology | Spinal Nerve Roots - physiopathology | Action Potentials - drug effects | Carboxylic Acids - pharmacology | Membrane Potentials - drug effects | Neurons, Afferent - drug effects | Indenes - pharmacology | Spinal Cord Injuries - metabolism | Inhibitory Postsynaptic Potentials - physiology | Rats | gamma-Aminobutyric Acid - pharmacology | Nerve Fibers - drug effects | Bicuculline - pharmacology | Neurons, Afferent - physiology | Rats, Sprague-Dawley | Blotting, Western | Symporters - metabolism | Action Potentials - physiology | Animals | Pain - physiopathology | Receptors, GABA-A - physiology | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Neurons, Afferent - metabolism | Posterior Horn Cells - physiology | Inhibitory Postsynaptic Potentials - drug effects | GABA | Neurons | Stratospheric circulation | Neuroscience
Journal Article
Pain, ISSN 0304-3959, 2008, Volume 135, Issue 3, pp. 232 - 239
Although it has been shown that pro-inflammatory cytokines such as interleukin-1β (IL-1β) facilitate perception of noxious inputs at the spinal level, the... 
Phosphorylation | Spinal cord | Rat | IL-1β | NMDA receptor | Hyperalgesia | rat | GLIAL ACTIVATION | SPINAL-CORD | IL-1 beta | PROINFLAMMATORY CYTOKINES | INTRADERMAL INJECTION | PERIPHERAL INFLAMMATION | GLUTAMATE UPTAKE | NEUROSCIENCES | DORSAL-HORN | CLINICAL NEUROLOGY | NEUROPATHIC PAIN | NITRIC-OXIDE | ANESTHESIOLOGY | hyperalgesia | spinal cord | phosphorylation | BEHAVIORAL HYPERALGESIA | Analgesics - pharmacology | Pain Threshold - physiology | Posterior Horn Cells - drug effects | Receptors, N-Methyl-D-Aspartate - metabolism | Male | Pain - immunology | Gliosis - physiopathology | Astrocytes - immunology | Inflammation - drug therapy | Inflammation Mediators - pharmacology | Pain - drug therapy | Reaction Time - drug effects | Pain Threshold - drug effects | Posterior Horn Cells - physiopathology | Gliosis - immunology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Phosphorylation - drug effects | Disease Models, Animal | Interleukin-1beta - antagonists & inhibitors | Astrocytes - drug effects | Receptors, N-Methyl-D-Aspartate - drug effects | Reaction Time - immunology | Anti-Inflammatory Agents - pharmacology | Receptors, Interleukin-1 - immunology | Proto-Oncogene Proteins c-fos - metabolism | Interleukin-1beta - immunology | Rats | Inflammation - immunology | Rats, Sprague-Dawley | Proto-Oncogene Proteins c-fos - drug effects | Hyperalgesia - physiopathology | Hyperalgesia - immunology | Posterior Horn Cells - immunology | Animals | Hyperalgesia - drug therapy | Pain - physiopathology | Gliosis - drug therapy | Receptors, Interleukin-1 - antagonists & inhibitors | Inflammation - physiopathology | Interleukins | Methyl aspartate
Journal Article