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Journal Article
Science, ISSN 0036-8075, 2/2011, Volume 331, Issue 6018, pp. 768 - 772
Endocrine tumors such as aldosterone-producing adrenal adenomas (APAs), a cause of severe hypertension, feature constitutive hormone production and... 
Depolarization | Hypertension | Somatic mutation | Cell growth | Hyperaldosteronism | REPORTS | Loss of heterozygosity | Adenoma | Genetic mutation | Sequencing | Tumors | REMEDIABLE ALDOSTERONISM | HUMAN BREAST | COLORECTAL CANCERS | ADRENOCORTICAL-CELLS | MULTIDISCIPLINARY SCIENCES | CONDUCTION | GLOMERULOSA CELLS | IDENTIFICATION | MOLECULAR-BASIS | SELECTIVITY | POTASSIUM CHANNEL | Adrenal Glands - pathology | Potassium - metabolism | Cell Proliferation | Humans | Hyperaldosteronism - pathology | Hyperplasia | Protein Multimerization | Zona Glomerulosa - pathology | Male | Hyperaldosteronism - metabolism | Sodium - metabolism | Aldosterone - metabolism | Adrenocortical Adenoma - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Female | Hypertension - genetics | Adrenal Cortex Neoplasms - metabolism | Cell Line | G Protein-Coupled Inwardly-Rectifying Potassium Channels - chemistry | Hyperaldosteronism - genetics | Mutant Proteins - genetics | Mutant Proteins - metabolism | Zona Glomerulosa - metabolism | Adrenal Cortex Neoplasms - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Adrenocortical Adenoma - pathology | Adrenocortical Adenoma - metabolism | Hypertension - metabolism | Adrenal Cortex Neoplasms - genetics | Mutant Proteins - chemistry | Mutation | Index Medicus | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
Journal Article
Science, ISSN 0036-8075, 5/2012, Volume 336, Issue 6081, pp. 597 - 601
Journal Article
Circulation Research, ISSN 0009-7330, 11/2011, Volume 109, Issue 11, pp. 1259 - 1268
Journal Article
Journal Article
Journal Article
CIRCULATION RESEARCH, ISSN 0009-7330, 08/2012, Volume 111, Issue 4, pp. 446 - 454
Rationale: Activation of the mitochondrial ATP-sensitive potassium channel (mitoK(ATP)) has been implicated in the mechanism of cardiac ischemic... 
SENSITIVE POTASSIUM CHANNELS | CARDIAC & CARDIOVASCULAR SYSTEMS | cytoprotection | mitochondria | K-ATP CHANNELS | apoptosis | IDENTIFICATION | ATP-sensitive potassium channel | preconditioning | ischemia | INFARCT SIZE | ACTION-POTENTIAL DURATION | HIGH-AFFINITY INHIBITOR | DISEASE | renal outer medullary potassium channel | PERIPHERAL VASCULAR DISEASE | MATRIX VOLUME | CARDIOPROTECTION | HEMATOLOGY | EXPRESSION | Diazoxide - pharmacology | Mitochondria, Heart - metabolism | Cricetulus | Mitochondria, Heart - pathology | Humans | Thallium - metabolism | Mitochondria, Heart - drug effects | RNA, Messenger - metabolism | Necrosis | Mitochondrial Membranes - drug effects | Potassium Channels - metabolism | Transfection | RNA Interference | Time Factors | Cattle | Adenosine Triphosphate - metabolism | Mass Spectrometry | Proteomics - methods | Potassium Channel Blockers - pharmacology | CHO Cells | Cytoprotection | Animals, Newborn | Cricetinae | Potassium Channels, Inwardly Rectifying - drug effects | Bee Venoms - pharmacology | Gene Expression Regulation | Rats | Potassium Channels, Inwardly Rectifying - genetics | Potassium Channels - drug effects | Mitochondrial Proton-Translocating ATPases - metabolism | Mitochondrial Membranes - metabolism | Potassium Channels - genetics | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Potassium Channels, Inwardly Rectifying - metabolism | Apoptosis | Index Medicus
Journal Article
Journal Article