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NATURE COMMUNICATIONS, ISSN 2041-1723, 10/2019, Volume 10, Issue 1, pp. 4768 - 15
B-1a cells are long-lived, self-renewing innate-like B cells that predominantly inhabit the peritoneal and pleural cavities. In contrast to conventional B-2... 
B-CELLS | STEM-CELLS | POSITIVE SELECTION | LIGHT-CHAIN RECOMBINATION | MULTIDISCIPLINARY SCIENCES | TRANSITIONAL CELLS | BONE-MARROW | IMMUNOGLOBULIN-HEAVY | EXPRESSION | T-CELLS | STAT5 ACTIVATION | Immunoglobulin Light Chains, Surrogate - immunology | Bone Marrow - immunology | Pre-B Cell Receptors - immunology | STAT5 Transcription Factor - immunology | Receptors, Antigen, B-Cell - metabolism | Liver - embryology | Cell Differentiation - genetics | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Liver - immunology | Lymphocyte Activation - genetics | Lymphocyte Activation - immunology | Receptors, Interleukin-7 - genetics | Bone Marrow - metabolism | B-Lymphocyte Subsets - immunology | Immunoglobulin Light Chains, Surrogate - genetics | Pre-B Cell Receptors - genetics | Receptors, Interleukin-7 - immunology | B-Lymphocytes - metabolism | Receptors, Interleukin-7 - metabolism | Liver - metabolism | Mice, Inbred C57BL | Receptors, Antigen, B-Cell - immunology | Mice, Knockout | Cell Differentiation - immunology | Animals | B-Lymphocytes - immunology | Immunoglobulin Light Chains, Surrogate - metabolism | B-Lymphocyte Subsets - metabolism | Receptors, Antigen, B-Cell - genetics | Pre-B Cell Receptors - metabolism | Cell culture | Antigens | Recombination | Fetuses | Autoantigens | Stat5 protein | B-cell receptor | Cell differentiation | Receptors | Lymphocytes B | Bone marrow | Peritoneum | Apoptosis
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2013, Volume 19, Issue 8, pp. 1014 - 1022
The B cell-specific transcription factor BACH2 is required for affinity maturation of B cells. Here we show that Bach2-mediated activation of p53 is required... 
MEDICINE, RESEARCH & EXPERIMENTAL | BCL6 | MYC | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACUTE LYMPHOBLASTIC-LEUKEMIA | TRANSCRIPTION | STAT5 | IDENTIFICATION | CHAIN GENE | EXPRESSION | CELL BIOLOGY | Basic-Leucine Zipper Transcription Factors - metabolism | Precursor Cells, B-Lymphoid - metabolism | Cell Survival - genetics | Molecular Sequence Data | RNA, Messenger - metabolism | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | STAT5 Transcription Factor - metabolism | Precursor Cells, B-Lymphoid - pathology | Gene Deletion | Cell Death | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | PAX5 Transcription Factor - metabolism | Green Fluorescent Proteins - metabolism | RNA, Messenger - genetics | Tumor Suppressor Protein p53 - metabolism | Immunoglobulin mu-Chains - metabolism | Treatment Outcome | Basic-Leucine Zipper Transcription Factors - genetics | Gene Expression Regulation, Leukemic | Proto-Oncogene Proteins c-myc - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | V(D)J Recombination - genetics | Animals | Mice | Cell Transformation, Neoplastic - pathology | Pre-B Cell Receptors - metabolism | Proto-Oncogene Proteins c-bcl-6 | Cell receptors | Physiological aspects | Development and progression | Tumor suppressor genes | Lymphomas | Genetic aspects | Research | B cells | Oncology | Genetic testing | Cellular biology | Leukemia | Tumors
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2016, Volume 213, Issue 2, pp. 209 - 223
DNA double-strand breaks (DSBs) activate a canonical DNA damage response, including highly conserved cell cycle checkpoint pathways that prevent cells with... 
MEDICINE, RESEARCH & EXPERIMENTAL | LIGHT-CHAIN RECOMBINATION | SPI-C | ACUTE LYMPHOBLASTIC-LEUKEMIA | BONE-MARROW | V(D)J RECOMBINATION | GENE-EXPRESSION | CYCLE ARREST | DIFFERENTIATION | IMMUNOLOGY | NF-KAPPA-B | PU.1 ETS DOMAIN | Cell Proliferation | Ataxia Telangiectasia Mutated Proteins - metabolism | Protein-Tyrosine Kinases - metabolism | Homeodomain Proteins - metabolism | Precursor Cells, B-Lymphoid - metabolism | Precursor Cells, B-Lymphoid - immunology | Intracellular Signaling Peptides and Proteins - metabolism | DNA Breaks, Double-Stranded | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | NF-kappa B p52 Subunit - deficiency | NF-kappa B p52 Subunit - genetics | Gene Rearrangement, B-Lymphocyte, Light Chain | Cell Cycle Checkpoints - immunology | Syk Kinase | Protein-Serine-Threonine Kinases - metabolism | Proto-Oncogene Proteins - metabolism | Mice, Transgenic | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | NF-kappa B p52 Subunit - metabolism | Mice, Knockout | Animals | Precursor Cells, B-Lymphoid - cytology | Trans-Activators - metabolism | Ataxia Telangiectasia Mutated Proteins - deficiency | Mice | Adaptor Proteins, Signal Transducing - metabolism | Ataxia Telangiectasia Mutated Proteins - genetics | Pre-B Cell Receptors - metabolism
Journal Article
Cancer Cell, ISSN 1535-6108, 03/2015, Volume 27, Issue 3, pp. 409 - 425
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 06/2016, Volume 35, Issue 25, pp. 3227 - 3238
ZFP521 was previously identified as a putative gene involved in induction of B-cell lymphomagenesis. However, the contribution of ZFP521 to lymphomagenesis has... 
BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | LOCUS | CELL BIOLOGY | HEAVY-CHAINS | RETROVIRUS INTEGRATION | GENE | ONCOLOGY | ACUTE LYMPHOBLASTIC-LEUKEMIA | GENETICS & HEREDITY | ZINC-FINGER PROTEIN | BTK | NF-KAPPA-B | BCR CHECKPOINT | Immunohistochemistry | Protein-Tyrosine Kinases - metabolism | Humans | Precursor Cells, B-Lymphoid - metabolism | Immunoblotting | Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Protein-Tyrosine Kinases - genetics | RNA Interference | Cyclin D3 - genetics | Pre-B Cell Receptors - genetics | Cyclin D3 - metabolism | Disease Models, Animal | Cell Line | Cell Proliferation - genetics | Proto-Oncogene Proteins c-jun - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Signal Transduction - genetics | Transcription Factors - genetics | Mice, Inbred Strains | Reverse Transcriptase Polymerase Chain Reaction | Proto-Oncogene Proteins c-myc - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Transcription Factors - metabolism | Animals | Proto-Oncogene Proteins c-jun - metabolism | Adaptor Proteins, Signal Transducing - genetics | Proto-Oncogene Proteins c-myc - genetics | Adaptor Proteins, Signal Transducing - metabolism | Pre-B Cell Receptors - metabolism | Research | B cells | Interleukins | Mitogen-activated protein kinases | Cell division | Lymphomas | Cytokines | Gene expression | Index Medicus
Journal Article
Journal Article