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Biological psychiatry (1969), ISSN 0006-3223, 2010, Volume 68, Issue 12, pp. 1172 - 1181
Background Gene-environment interactions (GEI) are involved in the pathogenesis of mental diseases. We evaluated interaction between mutant human... 
Psychiatry | mouse models | Tet-off system | schizophrenia | DISC1 | gene-environment interactions | mood disorders | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Mood Disorders - genetics | Prenatal Exposure Delayed Effects - psychology | Microtubule-Associated Proteins - metabolism | Glycogen Synthase Kinase 3 beta | Male | Brain - metabolism | Mice, Mutant Strains | Stress, Psychological - metabolism | Atrophy - genetics | Behavior, Animal - drug effects | Female | Corticosterone - blood | Disease Models, Animal | Animals, Newborn | Poly I-C - pharmacology | Cytokine Receptor gp130 - metabolism | Cytokines - metabolism | Mice, Transgenic | Glycogen Synthase Kinase 3 - metabolism | Nerve Tissue Proteins - genetics | Prenatal Exposure Delayed Effects - metabolism | Pregnancy | Prenatal Exposure Delayed Effects - genetics | 1-Alkyl-2-acetylglycerophosphocholine Esterase - metabolism | Animals | Carrier Proteins - metabolism | Prenatal Exposure Delayed Effects - immunology | Mood Disorders - pathology | Dendritic Spines - pathology | Brain - pathology | Mice | Biogenic Monoamines - metabolism | GRB2 Adaptor Protein - metabolism | Atrophy - metabolism | Brain - immunology | Mood Disorders - metabolism | Psychological aspects | Schizophrenia | Universities and colleges | Pregnant women | Analysis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 11/2015, Volume 112, Issue 46, pp. 14348 - 14353
Journal Article
Neuroscience, ISSN 0306-4522, 2010, Volume 167, Issue 3, pp. 741 - 749
.... The aim of the present study was to investigate in the rat the possible organizational effects of low BPA exposure on glucocorticoid-regulated responses... 
Neurology | Endocrine disrupters | Glucocorticoid receptors | Hippocampus | Y-maze | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Adaptation, Psychological - physiology | Memory Disorders - physiopathology | Receptors, Steroid - metabolism | Rats, Wistar | Phenols - toxicity | Memory Disorders - metabolism | Male | Hippocampus - drug effects | Brain - metabolism | Dose-Response Relationship, Drug | Exploratory Behavior - physiology | Exploratory Behavior - drug effects | Neurosecretory Systems - metabolism | Female | Anxiety - metabolism | Hypothalamo-Hypophyseal System - physiopathology | Neurosecretory Systems - physiopathology | Hypothalamo-Hypophyseal System - drug effects | Pituitary-Adrenal System - physiopathology | Benzhydryl Compounds | Maze Learning - physiology | Pituitary-Adrenal System - drug effects | Adaptation, Psychological - drug effects | Brain - physiopathology | Anxiety - physiopathology | Rats | Receptors, Steroid - drug effects | Prenatal Exposure Delayed Effects - physiopathology | Sex Characteristics | Maze Learning - drug effects | Prenatal Exposure Delayed Effects - metabolism | Endocrine Disruptors - toxicity | Brain - drug effects | Environmental Exposure - adverse effects | Pregnancy | Hippocampus - metabolism | Animals | Estrogens, Non-Steroidal - toxicity | Neurosecretory Systems - drug effects | Anxiety - chemically induced | Corticosterone - metabolism | Corticosterone - agonists | Hippocampus - physiopathology | Prenatal Exposure Delayed Effects - chemically induced | Memory Disorders - chemically induced | Index Medicus
Journal Article
Toxicology (Amsterdam), ISSN 0300-483X, 07/2017, Volume 387, pp. 67 - 80
Graphical abstract 
Emergency | Glyphosate-based herbicide | Depressive-like behavior | Oxidative stress | NMDA glutamate receptor | Molecular docking | Glutamate excitotoxicity | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Glycine - analogs & derivatives | Depression - physiopathology | Glycine - metabolism | Prenatal Exposure Delayed Effects | Age Factors | Rats, Wistar | Herbicides - chemistry | Receptors, N-Methyl-D-Aspartate - metabolism | Motor Activity - drug effects | Male | NF-kappa B - metabolism | Structure-Activity Relationship | Hippocampus - drug effects | Cholinergic Fibers - metabolism | Depression - metabolism | Neurotoxicity Syndromes - psychology | Glycine - toxicity | Glycine - chemistry | Depression - chemically induced | Behavior, Animal - drug effects | Female | Synaptic Transmission - drug effects | Molecular Structure | S100 Calcium Binding Protein beta Subunit - metabolism | Binding Sites | Herbicides - metabolism | Cholinergic Fibers - drug effects | Astrocytes - drug effects | Herbicides - toxicity | Gestational Age | GPI-Linked Proteins - metabolism | Neurotoxicity Syndromes - etiology | Pregnancy | Hippocampus - metabolism | Animals | Receptors, N-Methyl-D-Aspartate - agonists | Neurotoxicity Syndromes - physiopathology | Depression - psychology | Neurotoxicity Syndromes - metabolism | Protein Binding | Glutamic Acid - metabolism | Molecular Docking Simulation | Oxidative Stress - drug effects | Hippocampus - physiopathology | Acetylcholinesterase - metabolism | Astrocytes - metabolism | Glutamic Acid - chemistry | Mitogen-Activated Protein Kinases - metabolism | Amino acids | Glutamate | Glyphosate | Depression, Mental | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e63902 - e63902
Exposure of rodent fetuses to low doses of the endocrine disruptor bisphenol A (BPA) causes subtle morphological changes in the prenatal mammary gland and results in pre-cancerous and cancerous lesions during adulthood... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Transcription, Genetic - drug effects | Apoptosis - drug effects | Benzhydryl Compounds - toxicity | Phenols - toxicity | Fetus - metabolism | Mesoderm - drug effects | Apoptosis - genetics | Gene Expression Profiling | RNA, Messenger - metabolism | Mammary Glands, Animal - embryology | Stromal Cells - drug effects | Estrogen Receptor alpha - metabolism | Female | Principal Component Analysis | Epithelium - drug effects | Epithelium - metabolism | Mammary Glands, Animal - pathology | Mice, Inbred C57BL | RNA, Messenger - genetics | Stromal Cells - metabolism | Gene Expression Regulation, Developmental - drug effects | Mammary Glands, Animal - drug effects | Signal Transduction - genetics | Transcriptome - genetics | Ethinyl Estradiol - pharmacology | Troponin C - metabolism | Fetus - drug effects | Pregnancy | Prenatal Exposure Delayed Effects - genetics | Focal Adhesions - drug effects | Animals | Mammary Glands, Animal - metabolism | Signal Transduction - drug effects | Focal Adhesions - metabolism | Mesoderm - metabolism | Mice | Prenatal Exposure Delayed Effects - pathology | Cluster Analysis | Bisphenol-A | RNA | Pregnant women | Genes | Stem cells | Ethinyl estradiol | Dosage and administration | Comparative analysis | Gene expression | Estradiol | Ethinylestradiol | Mesenchyme | Estrogens | Glands | Estrogen | Thyroid gland | Cell adhesion & migration | Morphogenesis | Xenoestrogens | Developmental stages | Rodents | Animal tissues | Mammary gland | Lesions | Drug dosages | Adipogenesis | Urine | Puberty | Fetuses | Stroma | Mammary glands | Exposure | Breast cancer | Epithelium | Embryos | Medicine | Studies | Bisphenol A | Cellular biology | Phenols | Laboratory animals | Apoptosis | Index Medicus | Life Sciences
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