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Science (American Association for the Advancement of Science), ISSN 1095-9203, 06/2002, Volume 296, Issue 5575, pp. 1991 - 1995
...Neurodegenerative disorders as diverse as Alzheimer's disease, Parkinson's disease, priori diseases, Huntington's disease, frontotemporal dementia, and motor... 
Nervous system diseases | Reviews | Neurodegenerative diseases | Disease models | Huntington disease | Parkinson disease | Prion diseases | Amyotrophic lateral sclerosis | Nervous system heredodegenerative disorders | Alzheimers disease | Genetic mutation | Parkinson Disease - therapy | Neurons - pathology | Prion Diseases - genetics | Humans | Peptides - genetics | Tauopathies - pathology | Alzheimer Disease - pathology | Peptides - metabolism | Tauopathies - therapy | Neurons - metabolism | Parkinson Disease - metabolism | Inclusion Bodies - metabolism | Prion Diseases - therapy | Parkinson Disease - pathology | Neurodegenerative Diseases - pathology | Amyotrophic Lateral Sclerosis - therapy | Amyotrophic Lateral Sclerosis - genetics | Alzheimer Disease - therapy | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - therapy | Prion Diseases - pathology | Neurodegenerative Diseases - metabolism | Parkinson Disease - genetics | Protein Folding | Trinucleotide Repeat Expansion | Proteins - genetics | Amyotrophic Lateral Sclerosis - pathology | Tauopathies - metabolism | Animals | Proteins - metabolism | Alzheimer Disease - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Mutation | Proteins - chemistry | Alzheimer Disease - genetics | Prion Diseases - metabolism | Parkinson's disease | Nervous system | Degeneration | Research | Alzheimer's disease | Proteins | Neurological disorders | Index Medicus
Journal Article
PLoS pathogens, ISSN 1553-7366, 03/2010, Volume 6, Issue 3, pp. e1000800 - e1000800
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Brain pathology (Zurich, Switzerland), ISSN 1015-6305, 03/2015, Volume 25, Issue 2, pp. 121 - 135
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Nature (London), ISSN 1476-4687, 12/2018, Volume 564, Issue 7736, pp. 415 - 419
We previously reported(1) the presence of amyloid-beta protein (A beta) deposits in individuals with Creutzfeldt-Jakob disease (CJD... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Alzheimer Disease - etiology | Humans | tau Proteins - metabolism | Male | Drug Contamination - prevention & control | Case-Control Studies | Disease Transmission, Infectious - prevention & control | Disease Transmission, Infectious - statistics & numerical data | Amyloid beta-Peptides - metabolism | Female | Creutzfeldt-Jakob Syndrome - chemically induced | Iatrogenic Disease | Alzheimer Disease - chemically induced | Growth Hormone - administration & dosage | Disease Models, Animal | Prions - metabolism | Reproducibility of Results | Amyloid beta-Protein Precursor - adverse effects | Growth Hormone - pharmacology | Recombinant Proteins - pharmacology | Recombinant Proteins - administration & dosage | Creutzfeldt-Jakob Syndrome - etiology | Drug Contamination - statistics & numerical data | Animals | Models, Biological | Amyloid beta-Peptides - analysis | Amyloid beta-Protein Precursor - administration & dosage | Mice | tau Proteins - analysis | Cadaver | Brain | Seeds | Peptides | Creutzfeldt-Jakob disease | Proteins | Surgery | Vials | Children | Prion protein | Alzheimer's disease | Deposition | Public health | Plaques | Neurodegenerative diseases | Medical treatment | Patients | Disease control | Amyloid precursor protein | Laboratory tests | Pathology | Hypotheses | Tau protein | Prions | Pituitary | Cadavers | Growth hormones | Growth hormone | Laboratory animals | Alzheimers disease | Index Medicus
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Molecular neurobiology, ISSN 1559-1182, 08/2008, Volume 38, Issue 1, pp. 78 - 100
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