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Nature Genetics, ISSN 1061-4036, 12/2014, Volume 47, Issue 1, pp. 31 - 38
Cushing's disease is caused by corticotroph adenomas of the pituitary. To explore the molecular mechanisms of endocrine autonomy in these tumors, we performed... 
UBPY | ENDOSOMES | DOMAIN | PROTEIN | EPIDERMAL-GROWTH-FACTOR | GENETICS & HEREDITY | UBIQUITYLATION | DEGRADATION | RECEPTOR DOWN-REGULATION | ENDOCYTIC TRAFFICKING | PITUITARY-ADENOMAS | Adenoma - genetics | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Cercopithecus aethiops | Endosomal Sorting Complexes Required for Transport - genetics | Molecular Sequence Data | Pituitary Neoplasms - complications | ACTH-Secreting Pituitary Adenoma - complications | Pituitary ACTH Hypersecretion - genetics | ACTH-Secreting Pituitary Adenoma - genetics | Receptor, Epidermal Growth Factor - metabolism | Pituitary Neoplasms - secretion | Neoplasm Proteins - genetics | Pro-Opiomelanocortin - genetics | Amino Acid Sequence | ACTH-Secreting Pituitary Adenoma - metabolism | Pituitary Neoplasms - genetics | Ubiquitin Thiolesterase - physiology | Pituitary Neoplasms - metabolism | Ubiquitin Thiolesterase - genetics | 14-3-3 Proteins - metabolism | Adrenocorticotropic Hormone - secretion | Sequence Homology, Amino Acid | Exome - genetics | Sequence Alignment | Animals | Endopeptidases - genetics | ACTH-Secreting Pituitary Adenoma - secretion | Pro-Opiomelanocortin - biosynthesis | Mutation | COS Cells | Endosomal Sorting Complexes Required for Transport - physiology | Endopeptidases - physiology | Gene mutations | Cushing syndrome | Physiological aspects | Genetic aspects | Research | Ubiquitin-proteasome system | Health aspects | Risk factors | Pituitary gland | Kinases | Rodents | Genes | Tumors | Index Medicus
Journal Article
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2011, Volume 69, Issue 6, pp. 526 - 533
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e45300 - e45300
Journal Article
Neuron, ISSN 0896-6273, 02/2012, Volume 73, Issue 3, pp. 511 - 522
AgRP neuron activity drives feeding and weight gain whereas that of nearby POMC neurons does the opposite. However, the role of excitatory glutamatergic input... 
POMC NEURONS | GLUCOSE-HOMEOSTASIS | OBESITY | ENERGY-EXPENDITURE | WEIGHT HOMEOSTASIS | SK CHANNELS | ARCUATE NUCLEUS | HYPOTHALAMIC NEURONS | NEUROSCIENCES | TERM SYNAPTIC PLASTICITY | NEUROPEPTIDE-Y | Membrane Potentials - genetics | Age Factors | Nerve Tissue Proteins - deficiency | 2-Amino-5-phosphonovalerate - pharmacology | Male | Neurons - cytology | Green Fluorescent Proteins - genetics | Neuropeptide Y - genetics | Excitatory Postsynaptic Potentials - drug effects | Eating - physiology | Time Factors | Body Composition - drug effects | Dendritic Spines - drug effects | Body Composition - genetics | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Female | Agouti-Related Protein - deficiency | Energy Metabolism - genetics | Neurons - drug effects | Pro-Opiomelanocortin - genetics | Excitatory Postsynaptic Potentials - genetics | Membrane Potentials - drug effects | Brain - cytology | Fasting | Gene Expression Regulation - genetics | Dendritic Spines - physiology | Proto-Oncogene Proteins c-fos - metabolism | Mice, Transgenic | Excitatory Amino Acid Antagonists - pharmacology | Agouti-Related Protein - metabolism | Nerve Tissue Proteins - genetics | GABA Antagonists - pharmacology | Gene Expression Regulation - drug effects | Carrier Proteins - genetics | Eating - drug effects | Patch-Clamp Techniques | Animals | Picrotoxin - pharmacology | Receptors, N-Methyl-D-Aspartate - deficiency | Mice | In Vitro Techniques | RNA, Messenger | Energy Metabolism - drug effects | Neuropeptide Y | Methyl aspartate | Messenger RNA | Neurons | RNA | Genetic engineering | Kinases | Rodents | Food | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2009, Volume 29, Issue 37, pp. 11582 - 11593
Leptin-stimulated Stat3 activation in proopiomelanocortin (POMC)-expressing neurons of the hypothalamus plays an important role in maintenance of energy... 
BODY-WEIGHT | GLUCOSE-HOMEOSTASIS | CYTOKINE SIGNALING-3 | ENERGY HOMEOSTASIS | RECEPTOR | MICE | AGRP NEURONS | DIET-INDUCED OBESITY | ARCUATE NUCLEUS | EXPRESSING NEURONS | NEUROSCIENCES | Enzyme-Linked Immunosorbent Assay - methods | Leptin - metabolism | Green Fluorescent Proteins - genetics | Neuropeptide Y - genetics | Phosphatidylinositol 3-Kinases - metabolism | Body Weight - genetics | Obesity - genetics | Patch-Clamp Techniques - methods | Neuropeptide Y - metabolism | Neural Inhibition - physiology | Transfection | Body Composition - genetics | Neurons - physiology | Electrophoretic Mobility Shift Assay | Membrane Proteins - metabolism | Disease Models, Animal | Neural Inhibition - genetics | Glucose Tolerance Test | Gene Expression Regulation - genetics | Membrane Proteins - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Mice, Transgenic | Obesity - physiopathology | Feedback - physiology | Hypothalamus - pathology | Signal Transduction - genetics | Leukemia Inhibitory Factor - pharmacology | Agouti-Related Protein - metabolism | Eating - genetics | Obesity - metabolism | Gene Expression Regulation - drug effects | Pro-Opiomelanocortin - metabolism | Suppressor of Cytokine Signaling 3 Protein | Insulin - metabolism | Animals | Agouti-Related Protein - genetics | Insulin Resistance - genetics | Mice | Suppressor of Cytokine Signaling Proteins - metabolism | In Vitro Techniques | Neural Inhibition - drug effects | Index Medicus
Journal Article
Journal of Experimental Biology, ISSN 0022-0949, 11/2010, Volume 213, Issue 22, pp. 3858 - 3865
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2010, Volume 285, Issue 45, pp. 34718 - 34728
To understand the role of microRNAs (miRNAs) in pituitary development, a group of pituitary-specific miRNAs were identified, and Dicer1 was then conditionally... 
MORPHOGENESIS | ACTIVATION | DOMAIN | INTERACT | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | SECRETION | BETA-CATENIN | PITX2 | REPRESSORS | PROMOTER | Prolactin - biosynthesis | Thyrotropin, beta Subunit - genetics | Endoribonucleases - genetics | Homeodomain Proteins - metabolism | Lymphoid Enhancer-Binding Factor 1 - genetics | Prolactin - genetics | Pituitary Gland, Anterior - growth & development | Transcription Factor Pit-1 - genetics | Mice, Mutant Strains | Pituitary Gland, Anterior - embryology | DEAD-box RNA Helicases - metabolism | Pro-Opiomelanocortin - genetics | Cell Differentiation - physiology | Gene Expression Regulation, Developmental - physiology | Cell Line | Transcription Factor Pit-1 - biosynthesis | Endoribonucleases - metabolism | Thyrotropin, beta Subunit - biosynthesis | Mice, Transgenic | MicroRNAs - biosynthesis | Transcription Factors - genetics | Lymphoid Enhancer-Binding Factor 1 - metabolism | Homeodomain Proteins - genetics | Cell Lineage - physiology | Transcription Factors - metabolism | DEAD-box RNA Helicases - genetics | Animals | Growth Disorders - metabolism | Promoter Regions, Genetic - physiology | Luteinizing Hormone, beta Subunit - genetics | Ribonuclease III | Pro-Opiomelanocortin - biosynthesis | Mice | MicroRNAs - genetics | Growth Disorders - genetics | Luteinizing Hormone, beta Subunit - biosynthesis | Index Medicus | Gene Regulation | Wnt Signaling | Transcription Regulation | MicroRNA | Pit-1 | Antisense RNA | RNA Silencing | Pituitary Gland | Pitx2 | Lef-1
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 48, pp. 12946 - 12955
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2005, Volume 115, Issue 12, pp. 3484 - 3493
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 05/2013, Volume 110, Issue 21, pp. 8555 - 8560
Cushing disease (CD) is a life-threatening disorder attributed to excess pituitary tumor-derived adrenocorticotrophic hormone (ACTH) and adrenal steroid... 
Cell growth | Phosphorylation | Pituitary neoplasms | Secretion | Plasmids | Cell lines | Pituitary ACTH hypersecretion | Mice | Orphan nuclear receptors | Tumors | MANAGEMENT | ACID | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | MODULATOR | EXPRESSION | Transcriptional Activation - genetics | Cell Proliferation | Receptors, Steroid - metabolism | Humans | Neoplasm Proteins - metabolism | ACTH-Secreting Pituitary Adenoma - therapy | Adrenocorticotropic Hormone - genetics | Pituitary ACTH Hypersecretion - genetics | ACTH-Secreting Pituitary Adenoma - genetics | Receptors, Thyroid Hormone - genetics | Response Elements - genetics | Corticosterone - secretion | Neoplasm Proteins - genetics | Pro-Opiomelanocortin - genetics | Pituitary ACTH Hypersecretion - therapy | Gene Expression Regulation, Neoplastic - genetics | Pituitary ACTH Hypersecretion - pathology | ACTH-Secreting Pituitary Adenoma - metabolism | Corticosterone - genetics | Adrenocorticotropic Hormone - secretion | Receptors, Thyroid Hormone - metabolism | Animals | Pituitary ACTH Hypersecretion - metabolism | Receptors, Steroid - genetics | Pro-Opiomelanocortin - biosynthesis | Cell Line, Tumor | ACTH-Secreting Pituitary Adenoma - pathology | Physiological aspects | Pituitary gland | Research | Hydrocortisone | Health aspects | Causes and theories of causation | Diseases | Rodents | Genetics | T cell receptors | Binding sites | Index Medicus | Biological Sciences
Journal Article
PLoS Genetics, ISSN 1553-7390, 01/2013, Volume 9, Issue 1, pp. e1003207 - e1003207
Journal Article