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Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 02/2015, Volume 146, pp. 48 - 61
Journal Article
Experimental Cell Research, ISSN 0014-4827, 2008, Volume 314, Issue 17, pp. 3162 - 3174
In this study we describe a novel Rho small GTPase dependent pathway that elicits apoptotic responses controlled by actin reorganization in hormone-sensitive... 
mAR activation | Rho-GTPase signaling | Prostate cancer | Actin dynamics | Apoptosis | LIM-KINASE | STEROID-HORMONES | PROGESTIN RECEPTOR | CELL BIOLOGY | IN-VITRO | GLUCOCORTICOID-RECEPTOR | ONCOLOGY | TESTOSTERONE RECEPTORS | FOCAL ADHESION KINASE | ACTIN CYTOSKELETON | NONGENOMIC ACTIONS | COFILIN PHOSPHORYLATION | Focal Adhesion Kinase 1 - genetics | Prostatic Neoplasms - metabolism | Humans | Receptors, Androgen - metabolism | Actins - metabolism | Male | cdc42 GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Testosterone - analogs & derivatives | rhoA GTP-Binding Protein - genetics | rhoB GTP-Binding Protein - metabolism | Lim Kinases - metabolism | Testosterone - metabolism | rho-Associated Kinases - metabolism | Amides - metabolism | rhoB GTP-Binding Protein - genetics | Destrin - metabolism | Focal Adhesion Kinase 1 - metabolism | Prostatic Neoplasms - pathology | Enzyme Inhibitors - metabolism | rho-Associated Kinases - genetics | Serum Albumin, Bovine - metabolism | Phosphatidylinositol 3-Kinases - genetics | Animals | Pyridines - metabolism | Receptors, Androgen - genetics | Cell Line, Tumor | Signal Transduction - physiology | Apoptosis - physiology | Enzyme Activation | rac1 GTP-Binding Protein - metabolism | Muscle proteins | Actin | Cancer cells | Signal transduction | Hormones | Cellular biology
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 04/2012, Volume 94, Issue 1, pp. 96 - 104
GPR30 is a novel oestrogen receptor expressed in various tissues, including the heart. We determined the role of GPR30 in the maintenance of left ventricular... 
Cardiac hypertrophy | GPR30 | Post-menopausal women | Diastolic dysfunction | mRen2.Lewis rat | mRen2 | CARDIAC & CARDIOVASCULAR SYSTEMS | PLUS PROGESTIN | CARDIAC MYOCYTES | RANDOMIZED CONTROLLED-TRIAL | HYPERTENSIVE POSTMENOPAUSAL WOMEN | BLOOD-PRESSURE | Lewis rat | REPLACEMENT THERAPY | COUPLED ESTROGEN-RECEPTOR | SEX-DIFFERENCES | CONTRACTILE FUNCTION | CORONARY-HEART-DISEASE | Renin - metabolism | Receptors, G-Protein-Coupled - metabolism | Calcium - metabolism | Natriuretic Peptide, Brain - metabolism | NADPH Oxidases - metabolism | Rats, Inbred Lew | Receptors, G-Protein-Coupled - agonists | Quinolines - administration & dosage | RNA, Messenger - metabolism | Quinolines - pharmacology | Renin - genetics | Time Factors | Ventricular Dysfunction, Left - genetics | Injections, Subcutaneous | Echocardiography, Doppler | Female | Blood Pressure - drug effects | Estrogens - metabolism | Cell Line | Angiotensin II - metabolism | Atrial Natriuretic Factor - metabolism | Ovariectomy | Rats, Transgenic | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Ventricular Function, Left - drug effects | Rats | Genotype | Cardiotonic Agents - pharmacology | Cyclopentanes - pharmacology | NADPH Oxidase 4 | Ventricular Dysfunction, Left - diagnostic imaging | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Gene Expression Regulation - drug effects | Ventricular Dysfunction, Left - metabolism | Collagen - metabolism | Myocytes, Cardiac - pathology | Phenotype | Animals | Cardiotonic Agents - administration & dosage | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Mice | Receptors, G-Protein-Coupled - genetics | Ventricular Remodeling - drug effects | Cyclopentanes - administration & dosage | Hypertrophy, Left Ventricular - diagnostic imaging | Original
Journal Article
European Journal of Endocrinology, ISSN 0804-4643, 06/2017, Volume 176, Issue 6, pp. R283 - R308
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e31267
This study investigated the impact of chronic exposure to continuous (CoP4) versus cyclic progesterone (CyP4) alone or in combination with 17 beta-estradiol... 
MITOCHONDRIAL ATP-SYNTHASE | CONJUGATED EQUINE ESTROGENS | ALZHEIMERS-DISEASE | HEALTH INITIATIVE MEMORY | BIOLOGY | AMYLOID PRECURSOR PROTEIN | RANDOMIZED CONTROLLED-TRIAL | GROWTH-FACTOR | MILD COGNITIVE IMPAIRMENT | ESTROGEN PLUS PROGESTIN | POSTMENOPAUSAL WOMEN | Estradiol - metabolism | Computational Biology - methods | Receptors, Estrogen - metabolism | Ovariectomy | Oxidation-Reduction | Peroxiredoxins - metabolism | Gene Expression Regulation | Homeostasis | Rats | Gene Expression Profiling | Mitochondria - metabolism | Orphan Nuclear Receptors - metabolism | Mitochondrial Proton-Translocating ATPases - metabolism | Receptors, Progesterone - metabolism | Hippocampus - metabolism | Amyloidogenic Proteins - metabolism | Animals | Liver X Receptors | Progesterone - metabolism | Female | Estrogens - metabolism | Insulin-Like Growth Factor I - metabolism | Oxidative Phosphorylation Coupling Factors - metabolism | Genes | Physiological aspects | Drug therapy, Combination | Progesterone | Hormones | Gene expression | Estradiol | Peroxiredoxin | Brain | Regulations | Insulin-like growth factor I | Menopause | Liver | Trafficking | Estrogen | 17β-Estradiol | Kinases | Chronic exposure | ATP synthase | Mitochondria | Bioenergetics | Rodents | Down-regulation | Aging | Protein transport | Bioinformatics | b-Site APP cleaving enzyme 1 | Liver X receptors | Aging (artificial) | Exposure | Metabolism | Insulin | Cholesterol | Sex hormones | Womens health | β-Amyloid | Aging (natural) | Secretase | Hippocampus
Journal Article
Journal Article