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Endocrinology, ISSN 0013-7227, 10/2013, Volume 154, Issue 10, pp. 3643 - 3651
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 08/2016, Volume 311, Issue 2, pp. E380 - E395
Type 1 diabetes (T1D) originates from autoimmune beta-cell destruction. IMT504 is an immunomodulatory oligonucleotide that increases mesenchymal stem cell... 
Insulitis | β-cells | Islet gene expression | Serum cytokines | OLIGODEOXYNUCLEOTIDES | ACTIVATION | PHYSIOLOGY | serum cytokines | islet gene expression | REG FAMILY GENES | PROLIFERATION | MESENCHYMAL STEM-CELLS | IL-6 | insulitis | ENDOCRINOLOGY & METABOLISM | beta-cells | STREPTOZOTOCIN | EXPRESSION | ONSET | Islets of Langerhans - drug effects | Apoptosis - drug effects | Chemokine CXCL1 - drug effects | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Male | Chemokine CXCL1 - genetics | RNA, Messenger - metabolism | Nestin - drug effects | Insulin - genetics | Interleukin-6 - metabolism | Disease Models, Animal | Somatostatin - genetics | Lithostathine - drug effects | Blood Glucose - drug effects | Insulin - metabolism | Protein Precursors - drug effects | Tumor Necrosis Factor-alpha - drug effects | Mice | Mice, Inbred BALB C | Proglucagon - genetics | Blood Glucose - metabolism | Oligodeoxyribonucleotides - pharmacology | Indoleamine-Pyrrole 2,3,-Dioxygenase - metabolism | Tumor Necrosis Factor-alpha - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - genetics | Indoleamine-Pyrrole 2,3,-Dioxygenase - drug effects | Stem Cells - metabolism | Insulin-Secreting Cells - metabolism | Proglucagon - drug effects | Somatostatin - drug effects | Islets of Langerhans - metabolism | Nestin - genetics | Diabetes Mellitus, Experimental - metabolism | RNA, Messenger - drug effects | Glucose Tolerance Test | Protein Precursors - genetics | Cytokines - metabolism | Islets of Langerhans - pathology | Lithostathine - genetics | Diabetes Mellitus, Type 1 - genetics | Transcriptome - drug effects | Pancreatitis-Associated Proteins | Proteins - genetics | Animals | Cytokines - drug effects | Insulin-Secreting Cells - drug effects | Platelet Endothelial Cell Adhesion Molecule-1 - drug effects | Proteins - drug effects | Stem Cells - drug effects | Cell Proliferation - drug effects | Physiological aspects | Models | Diabetes | Oligonucleotides | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 2011, Volume 60, Issue 11, pp. 2775 - 2786
OBJECTIVE To investigate deep and comprehensive analysis of gut microbial communities and biological parameters after prebiotic administration in obese and... 
GLUCAGON-LIKE PEPTIDE-1 | PERMEABILITY | MECHANISM | INULIN-TYPE FRUCTANS | PHYLOGENETIC MICROARRAY | INSULIN-RESISTANCE | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | ENDOTOXEMIA | RATS | ADIPOGENESIS | Obesity - diet therapy | Leptin - metabolism | Diabetes Mellitus, Type 2 - diet therapy | Glucagon-Like Peptide 1 - blood | Cecum - microbiology | Diabetes Mellitus, Type 2 - metabolism | RNA, Messenger - metabolism | Gram-Negative Bacteria - isolation & purification | Obesity - blood | Molecular Typing | Glucose Intolerance - prevention & control | Enteroendocrine Cells - metabolism | Proglucagon - metabolism | Enteroendocrine Cells - pathology | Hyperglycemia - prevention & control | Dietary Fats - adverse effects | Colon - pathology | Mice, Inbred C57BL | Gene Expression Regulation | Hyperlipidemias - prevention & control | Colon - metabolism | Obesity - metabolism | Obesity - pathology | Prebiotics | Diabetes Mellitus, Type 2 - blood | Animals | Gram-Positive Bacteria - classification | Gram-Positive Bacteria - isolation & purification | Mice, Obese | Mice | Proglucagon - genetics | Diabetes Mellitus, Type 2 - pathology | Gram-Negative Bacteria - classification | Glucose metabolism | Obesity | Microbiota (Symbiotic organisms) | Physiological aspects | Development and progression | Research | Gene expression | Metabolism | insulin-resistance | adipogenesis | inflammation | glucagon-like peptide-1 | permeability | endotoxemia | inulin-type fructans | rats | phylogenetic microarray | mechanism
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e64415
Journal Article
Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 7629
Bile acids are signalling molecules, which activate the transmembrane receptor TGR5 and the nuclear receptor FXR. BA sequestrants (BAS) complex bile acids in... 
GLP-1 SECRETION | FXR | BILE-ACID RECEPTORS | GLUCOSE-HOMEOSTASIS | OBESITY | MULTIDISCIPLINARY SCIENCES | RAT SMALL-INTESTINE | MICE | TYPE-2 DIABETES-MELLITUS | METABOLIC-RATE | EXPRESSION | Colon - cytology | Intestinal Mucosa - metabolism | Sequestering Agents - pharmacology | Humans | Ileum - metabolism | RNA, Messenger - metabolism | Colesevelam Hydrochloride - pharmacology | Obesity - genetics | Glucagon-Like Peptide 1 - genetics | Jejunum - metabolism | Insulin-Secreting Cells - metabolism | Proglucagon - drug effects | Diet, High-Fat | Jejunum - cytology | Enteroendocrine Cells - metabolism | Ileum - cytology | Proglucagon - metabolism | Insulin Secretion | Glucagon-Like Peptide 1 - metabolism | Signal Transduction | Bile Acids and Salts - metabolism | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Colon - metabolism | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Glycolysis | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice, Obese | Mice | Proglucagon - genetics | Receptors, G-Protein-Coupled - genetics | Blood Glucose - metabolism | Anticholesteremic Agents - pharmacology | Intestines - cytology | Cell and Molecular Biology | Endokrinologi och diabetes | Multidisciplinary Sciences | Cell- och molekylärbiologi | Endocrinology and Diabetes
Journal Article
Nutrients, ISSN 2072-6643, 10/2018, Volume 10, Issue 10, p. 1347
Journal Article
Journal Article
Gut, ISSN 0017-5749, 02/2018, Volume 67, Issue 2, pp. 271 - 283
ObjectiveTo investigate the beneficial role of prebiotics on endothelial dysfunction, an early key marker of cardiovascular diseases, in an original mouse... 
BILE ACID METABOLISM | INTESTINAL MICROBIOLOGY | PREBIOTIC | CARDIOVASCULAR DISEASE | ENDOCRINE HORMONES | ACID | GLP-1 | DIET | GLUCOSE | AKKERMANSIA-MUCINIPHILA | NITRIC-OXIDE | WEIGHT-LOSS | ATHEROSCLEROSIS | OBESE | GASTROENTEROLOGY & HEPATOLOGY | CARDIOVASCULAR RISK | Mesenteric Arteries - physiology | Bile Acids and Salts - blood | Gene Expression - drug effects | Bacteria - drug effects | Glucagon-Like Peptide 1 - biosynthesis | Endothelium, Vascular - drug effects | Male | Cecum - microbiology | Fructans - pharmacology | Vasodilation | Organic Anion Transporters, Sodium-Dependent - genetics | Fatty Acids, Omega-3 - deficiency | Antimicrobial Cationic Peptides - genetics | Disease Models, Animal | Fatty Acids, Omega-3 - administration & dosage | Aminopeptidases - genetics | Bile Acids and Salts - biosynthesis | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Mice, Knockout, ApoE | Prebiotics | Neurotensin - genetics | Animals | Gastrointestinal Microbiome - drug effects | Symporters - genetics | Carotid Arteries - physiology | Mice | Nitric Oxide Synthase - metabolism | Proglucagon - genetics | Nitric Oxide - metabolism | Dietary Supplements | rRNA 16S | Peptides | Glucagon | Polyunsaturated fatty acids | Liver | Microbiota | Metabolites | Apolipoprotein E | Rodents | Inulin | Physiology | Supplementation | Carotid artery | Glucagon-like peptide 1 | Obesity | Preservation | Dietary supplements | Abundance | Gene expression | Apolipoproteins | Fatty acids | Endothelium | Steatosis | Weight control | Diet | Nitric oxide | Diabetes | Cardiovascular diseases | Metabolic disorders | Veins & arteries | Gut Microbiota | 1506
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e61211
Elucidating the regulation of glucose-stimulated insulin secretion (GSIS) in pancreatic islet beta cells is important for understanding and treating diabetes.... 
ENDOCRINE PANCREAS | SYNAPTOTAGMIN-VII | NEONATAL DIABETES-MELLITUS | DIFFERENTIAL EXPRESSION | DNA METHYLATION | ISLETS | MULTIDISCIPLINARY SCIENCES | MICE | BINDING PROTEIN | IDENTIFICATION | IMPRINTED GENES | Cell Proliferation | Carbonic Anhydrase II - metabolism | Gene Regulatory Networks | Insulin-Secreting Cells - metabolism | Microarray Analysis | Clone Cells | Insulin-Secreting Cells - cytology | Nuclear Proteins - genetics | Proglucagon - metabolism | Carbonic Anhydrase II - genetics | Insulin Secretion | Calcium-Binding Proteins - metabolism | Cell Line | Proto-Oncogene Proteins c-maf - genetics | Nuclear Proteins - metabolism | Glucose - pharmacology | Secretagogins | Transcription Factors - genetics | Organ Specificity | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Synaptotagmins - metabolism | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Glucose - metabolism | Proto-Oncogene Proteins c-maf - metabolism | Mice | Proglucagon - genetics | Synaptotagmins - genetics | Calcium-Binding Proteins - genetics | Short term | Cell culture | Genes | Insulinoma | Glucose | Proteins | Glucose metabolism | Chlorides | Rodents | Cell cycle | DNA methylation | Pancreas | Deoxyribonucleic acid--DNA | University graduates | Antigens | Secretion | Diabetes mellitus | Cloning | Metabolism | Gene expression | Insulin | Potassium chloride | Molecular modelling | DNA microarrays | Stem cells | Epigenetics | Diabetes | Islets of Langerhans | Deoxyribonucleic acid | DNA
Journal Article
Journal Article