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Soft Computing, ISSN 1432-7643, 6/2019, Volume 23, Issue 12, pp. 4051 - 4064
Journal Article
Development, ISSN 0950-1991, 11/2009, Volume 136, Issue 21, pp. 3597 - 3606
.... Here we show that MTJ integrity is affected in talin 2-deficient mice. Concomitant ablation of talin 1 and 2 leads to defects in myoblast fusion and sarcomere assembly, resembling defects in muscle lacking β1 integrins... 
Talin | Mice | Integrin | Dystrophin | Muscular dystrophy | INTEGRIN-LINKED KINASE | I/LWEQ MODULE | ALPHA-7 INTEGRIN | CELL-ADHESION | DEVELOPMENTAL BIOLOGY | MOUSE DEVELOPMENT | SKELETAL-MUSCLE | PROGRESSIVE MYOPATHY | IN-VIVO | DUCHENNE MUSCULAR-DYSTROPHY | LEUKOCYTE ADHESION | Cell Fusion | Animals | Talin - genetics | Muscular Diseases - metabolism | Sarcomeres - metabolism | Cytoskeleton - metabolism | Muscle, Skeletal - embryology | Talin - metabolism | Integrins - metabolism | Mice, Knockout
Journal Article
Journal Article
Dimensions of Critical Care Nursing, ISSN 0730-4625, 07/2016, Volume 35, Issue 4, pp. 190 - 194
.... After cervical laminectomy with fusion, this gentleman was admitted to the medical intensive care unit, in a complete myasthenic crisis. 
Progressive weakness | Neurology | Myasthenia gravis crisis | Humans | Laminectomy - adverse effects | Male | Aged | Spinal Fusion - adverse effects | Chronic Disease | Myasthenia Gravis - etiology
Journal Article
PLoS biology, ISSN 1544-9173, 03/2010, Volume 8, Issue 3, p. e1000325
textabstractIn Bilateria, many axons cross the midline of the central nervous system, forming well-defined commissures. Whereas in mammals the functions of... 
PROGRESSIVE SCOLIOSIS | ROBO3 | SLIT | RESPONSIVENESS | PROTEIN | CHEMOATTRACTANT | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOLOGY | GUIDANCE | MUTATIONS | HORIZONTAL GAZE PALSY | EXPRESSION | Motor Activity - physiology | Humans | Axons - physiology | Recombinant Fusion Proteins - metabolism | Ocular Motility Disorders - genetics | Embryo, Mammalian - anatomy & histology | Axons - ultrastructure | Evoked Potentials, Auditory - physiology | Membrane Proteins - metabolism | Eye Movements - physiology | Rhombencephalon - anatomy & histology | Membrane Proteins - genetics | Mice, Inbred C57BL | Behavior, Animal - physiology | Nerve Tissue Proteins - genetics | Rhombencephalon - physiology | Body Patterning - physiology | Mice, Knockout | Nerve Tissue Proteins - metabolism | Embryo, Mammalian - physiology | Rotarod Performance Test | Animals | Movement Disorders - physiopathology | Recombinant Fusion Proteins - genetics | Mice | Movement Disorders - genetics | Body Patterning - genetics | Physiological aspects | Embryonic development | Axons | Rhombencephalon | Genetic aspects | Research | Ocular Motility Disorders | Embryo, Mammalian | Eye Movements | Nerve Tissue Proteins | Motor Activity | Movement Disorders | Recombinant Fusion Proteins | Evoked Potentials, Auditory | Behavior, Animal | Membrane Proteins | Life Sciences | Human health and pathology | Body Patterning | Sensory Organs | Spinal cord | Life sciences | Scoliosis | Defects
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2016, Volume 113, Issue 50, pp. E8187 - E8196
Tau prions are thought to aggregate in the central nervous system, resulting in neurodegeneration. Among the tauopathies, Alzheimer’s disease (AD) is the most... 
Pick's disease | Tauopathies | Argyrophilic grain disease | Progressive supranuclear palsy | Corticobasal degeneration | MOUSE-BRAIN | NEUROFIBRILLARY TANGLES | corticobasal degeneration | BRAIN-INJURY | MULTIDISCIPLINARY SCIENCES | FRONTOTEMPORAL DEMENTIA | SHARE ANTIGENIC DETERMINANTS | PICKS-DISEASE | ABNORMAL PHOSPHORYLATION | argyrophilic grain disease | MICROTUBULE-ASSOCIATED PROTEIN | PAIRED HELICAL FILAMENTS | tauopathies | progressive supranuclear palsy | TRANSGENIC MICE | Up-Regulation | Chronic Traumatic Encephalopathy - metabolism | Humans | Bacterial Proteins - chemistry | tau Proteins - metabolism | Pick Disease of the Brain - genetics | Recombinant Fusion Proteins - metabolism | tau Proteins - chemistry | Protein Isoforms - metabolism | tau Proteins - genetics | Chronic Traumatic Encephalopathy - genetics | HEK293 Cells | Luminescent Proteins - chemistry | Pick Disease of the Brain - metabolism | Supranuclear Palsy, Progressive - genetics | Cell Line | Bacterial Proteins - genetics | Recombinant Fusion Proteins - chemistry | Alzheimer Disease - metabolism | Recombinant Fusion Proteins - genetics | Supranuclear Palsy, Progressive - metabolism | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mutation | Alzheimer Disease - genetics | Luminescent Proteins - metabolism | Protein Isoforms - genetics | Physiological aspects | Alzheimer's disease | Health aspects | Prions | Encephalopathy | Biological Sciences | PNAS Plus | Pick’s disease
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 06/2010, Volume 160, Issue 4, pp. 810 - 820
Psoriasis is a chronic inflammatory disease affecting 1-3% of the general population. Among psoriatic patients, 5-40% are affected by psoriatic arthritis. Due... 
briakinumab | certolizumab | ustekinumab | psoriatic arthritis | psoriasis | golimumab | Golimumab | Ustekinumab | Briakinumab | Psoriasis | Certolizumab | Psoriatic arthritis | RANDOMIZED CONTROLLED-TRIAL | INTERLEUKIN-12/23 MONOCLONAL-ANTIBODY | PHASE-III TRIAL | TUMOR-NECROSIS-FACTOR | PROGRESSIVE MULTIFOCAL LEUKOENCEPHALOPATHY | CHRONIC PLAQUE PSORIASIS | DOUBLE-BLIND | PHARMACOLOGY & PHARMACY | PLACEBO-CONTROLLED TRIAL | LONG-TERM TREATMENT | CLINICAL-RESPONSE | T-Lymphocytes - physiology | Biological Products - pharmacokinetics | Recombinant Fusion Proteins - pharmacology | Recombinant Fusion Proteins - adverse effects | Arthritis, Psoriatic - physiopathology | Humans | Recombinant Fusion Proteins - therapeutic use | Antibodies, Monoclonal - adverse effects | Psoriasis - etiology | Antibodies, Monoclonal - therapeutic use | Biological Products - pharmacology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Interleukin-23 - antagonists & inhibitors | T-Lymphocytes - drug effects | Arthritis, Psoriatic - drug therapy | Interleukin-12 - antagonists & inhibitors | Immunoglobulin Fragments - therapeutic use | Product Surveillance, Postmarketing | Biological Products - adverse effects | Biological Products - therapeutic use | Psoriasis - immunology | Psoriasis - drug therapy | Antibodies, Monoclonal - pharmacology | Immunoglobulin Fragments - adverse effects | Antibodies, Monoclonal - pharmacokinetics | Immunoglobulin Fragments - pharmacology | Animals | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Arthritis, Psoriatic - immunology | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Psoriasis - physiopathology | Recombinant Fusion Proteins - pharmacokinetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacokinetics | Arthritis, Psoriatic - etiology | Chronic Disease | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Arthritis | Drug therapy | Immune system | Reviews
Journal Article
BMC Biochemistry, ISSN 1471-2091, 04/2014, Volume 15, Issue 1, pp. 8 - 8
Journal Article
mAbs, ISSN 1942-0862, 09/2011, Volume 3, Issue 5, pp. 461 - 466
.... In this review, we focus on infectious complications that occur upon treatment with mAbs or Fc-containing fusion proteins targeting leukocyte membrane proteins, including CD52, CD20, tumor necrosis... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Human | Monoclonal antibodies |