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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2012, Volume 109, Issue 23, pp. 8983 - 8988
In addition to glycolysis, the oncogenic transcription factor c-MYC (MYC) stimulates glutamine catabolism to fuel growth and proliferation of cancer cells... 
T lymphocytes | Cell growth | Energy metabolism | Cellular metabolism | Small interfering RNA | Catabolism | Biosynthesis | Tumors | Cancer | Apoptosis | Amino acids | miRNA | Reactive oxygen species | Metabolic tumor suppressor | Redox signaling | APOPTOSIS | MULTIDISCIPLINARY SCIENCES | amino acids | CELL-GROWTH | HYPOXIA | CANCER | B-CELLS | PYRROLINE-5-CARBOXYLIC ACID | redox signaling | OXIDASE | GENE | reactive oxygen species | TUMOR-SUPPRESSOR | CARBOXYLATION | metabolic tumor suppressor | Gas Chromatography-Mass Spectrometry | Reactive Oxygen Species - metabolism | Nitrogen Isotopes - metabolism | Oxidation-Reduction | Proline - metabolism | Pyrroline Carboxylate Reductases - metabolism | Humans | Glutamine - metabolism | MicroRNAs - metabolism | Carbon Isotopes - metabolism | Gene Expression Regulation, Enzymologic - physiology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Gene Knockdown Techniques | DNA-Binding Proteins - metabolism | Transcription Factors - metabolism | Chromatin Immunoprecipitation | Gene Expression Regulation, Enzymologic - genetics | Cell Line, Tumor | Nuclear Magnetic Resonance, Biomolecular | Proline Oxidase - metabolism | Real-Time Polymerase Chain Reaction | Cell proliferation | Transcription factors | Proline | Cell metabolism | Genetic aspects | Properties | Glutamine | Biological Sciences
Journal Article
Nature Communications, ISSN 2041-1723, 07/2017, Volume 8, Issue 1, p. 16031
Tissue architecture contributes to pancreatic ductal adenocarcinoma (PDAC) phenotypes. Cancer cells within PDAC form gland-like structures embedded in a... 
MICROENVIRONMENT | RECEPTOR UPARAP/ENDO180 | GLUCOSE-METABOLISM | PATHWAY | BIOSYNTHESIS | MULTIDISCIPLINARY SCIENCES | KRAS | INTRAEPITHELIAL NEOPLASIA | HYPOXIA | CANCER | TUMORS | Neoplasm Transplantation | Cell Proliferation | Pancreatic Neoplasms - metabolism | Proline - metabolism | Humans | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Extracellular Matrix - metabolism | Collagen - chemistry | Male | Extracellular Matrix - chemistry | Carcinoma, Pancreatic Ductal - genetics | Procollagen-Proline Dioxygenase - metabolism | Matrix Metalloproteinase 9 - metabolism | Biological Transport | Matrix Metalloproteinase 9 - genetics | Proline Oxidase - metabolism | Proline Oxidase - genetics | Signal Transduction | Cell Survival | Matrix Metalloproteinase 13 - genetics | Pancreatic Neoplasms - pathology | Mice, Transgenic | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Matrix Metalloproteinase 13 - metabolism | Collagen - metabolism | Animals | Procollagen-Proline Dioxygenase - genetics | Cell Line, Tumor | Mice | Cell proliferation | Adenocarcinoma | Cell survival | Oxidase | Fragments | Proline | In vitro testing | Fuels | Metabolism | Survival | Collagen | Proline oxidase | Extracellular matrix | Nutrients | Reservoirs | Pancreas | Embedded structures | Cancer | Tumors | Life Sciences
Journal Article
Cancer Research, ISSN 0008-5472, 07/2012, Volume 72, Issue 14, pp. 3677 - 3686
Journal Article
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 07/2017, Volume 118, Issue 7, pp. 1678 - 1688
ABSTRACT Pipecolate, an intermediate of the lysine catabolic pathway, is oxidized to Δ1‐piperideine‐6‐carboxylate (P6C) by the flavoenzyme l‐pipecolate oxidase... 
OXIDATIVE STRESS | PIPECOLATE OXIDASE | PIPECOLATE | AMINO ACID METABOLISM | MITOCHONDRIA | PROLINE TRANSPORTER | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEIN IMPORT | ACID OXIDATION | SACCHAROMYCES-CEREVISIAE | CELL-DEATH | CELL BIOLOGY | ALDEHYDE DEHYDROGENASE 7A1 | LIPOIC ACID | AMINO-ACIDS | MOLECULAR-CLONING | RNA, Small Interfering - genetics | TOR Serine-Threonine Kinases - metabolism | Humans | Hydrogen Peroxide - pharmacology | Oxidoreductases Acting on CH-NH Group Donors - genetics | Oxidoreductases Acting on CH-NH Group Donors - metabolism | Pipecolic Acids - pharmacology | Forkhead Box Protein O3 - metabolism | Proto-Oncogene Proteins c-akt - genetics | Forkhead Box Protein O3 - genetics | TOR Serine-Threonine Kinases - genetics | Signal Transduction - drug effects | Glucosephosphate Dehydrogenase - metabolism | Glucosephosphate Dehydrogenase - genetics | NADP - metabolism | Oxidative Stress - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Sarcosine Oxidase - metabolism | Pentose Phosphate Pathway | Cell Survival - physiology | HEK293 Cells - metabolism | Sarcosine Oxidase - genetics | Oxidative stress | Phosphorylation | Hydrogen peroxide | Oxidase | Proline | AKT protein | Biochemistry | Dehydrogenase | Mammalian cells | Signal transduction | Mitochondria | Pathways | Forkhead protein | Inhibition | L-Pipecolate oxidase | FOXO3 protein | Fractionation | Cell survival | RNA-mediated interference | Mortality | Interference | siRNA | Metabolism | Mammals | Ribonucleic acid--RNA | Survival | Inhibitors | Lysine | Cell death | Catabolism | pipecolate | pipecolate oxidase | mitochondria | oxidative stress | amino acid metabolism
Journal Article