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Chemical Research in Toxicology, ISSN 0893-228X, 09/2008, Volume 21, Issue 9, pp. 1814 - 1822
In vitro covalent binding assessments of drugs have been useful in providing retrospective insights into the association between drug metabolism and a... 
ACYL GLUCURONIDES | CHEMISTRY, MEDICINAL | PROTEIN | RAT | REACTIVE METABOLITES | TIENILIC ACID | HEPATIC-NECROSIS | BIOACTIVATION | IDENTIFICATION | CHEMISTRY, MULTIDISCIPLINARY | MASS-SPECTROMETRY | TOXICOLOGY | HUMAN URINE | Buspirone - chemistry | Raloxifene Hydrochloride - pharmacology | Thiazoles - metabolism | Acetaminophen - metabolism | Humans | Microsomes, Liver - metabolism | Indomethacin - metabolism | Simvastatin - pharmacology | Diclofenac - chemistry | Thiazines - metabolism | Propranolol - pharmacology | Toxicity Tests - methods | Binding Sites | Simvastatin - metabolism | Microsomes, Liver - chemistry | Indomethacin - pharmacology | Paroxetine - metabolism | Raloxifene Hydrochloride - metabolism | Triazoles - metabolism | Ticrynafen - metabolism | Paroxetine - pharmacology | Diclofenac - metabolism | Paroxetine - chemistry | Diphenhydramine - pharmacology | Diclofenac - pharmacology | Triazoles - chemistry | Buspirone - metabolism | Ticrynafen - pharmacology | Structure-Activity Relationship | Buspirone - pharmacology | Hepatocytes - metabolism | Dose-Response Relationship, Drug | Carbamazepine - chemistry | Diphenhydramine - metabolism | Acetaminophen - pharmacology | Microsomes, Liver - drug effects | Propranolol - metabolism | Carbamazepine - metabolism | Thiazines - pharmacology | Molecular Structure | Drug Evaluation, Preclinical | Hepatocytes - drug effects | Carbamazepine - pharmacology | Simvastatin - chemistry | Acetaminophen - chemistry | Ticrynafen - chemistry | Propranolol - chemistry | Diphenhydramine - chemistry | Thiazines - chemistry | Triazoles - pharmacology | Indomethacin - chemistry | Thiazoles - chemistry | Thiazoles - pharmacology | Raloxifene Hydrochloride - chemistry | Index Medicus
Journal Article
BIOLOGICAL PSYCHIATRY, ISSN 0006-3223, 10/2011, Volume 70, Issue 8, pp. 712 - 719
Background: Drug-associated cues can elicit stress-like responses in addicted individuals, indicating that cue-and stress-induced drug relapse may share some... 
norepinephrine | ANXIOGENIC DRUG YOHIMBINE | relapse | PSYCHIATRY | NEUROSCIENCES | BED NUCLEUS | corticotropin-releasing factor | CORTICOTROPIN-RELEASING-FACTOR | INDUCED RELAPSE | STRIA TERMINALIS | ALCOHOL-SEEKING | self-administration | STRESS-INDUCED REINSTATEMENT | ALPHA-ADRENERGIC RECEPTORS | Cocaine | PITUITARY-ADRENAL AXIS | I-1-IMIDAZOLINE RECEPTOR | imidazoline | Extinction, Psychological - drug effects | Motor Activity - drug effects | Male | Adrenergic alpha-2 Receptor Agonists - pharmacology | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Drug Interactions | Isoquinolines - pharmacology | Self Administration - psychology | Adrenergic alpha-1 Receptor Antagonists - pharmacology | Prazosin - pharmacology | Clonidine - pharmacology | Cocaine - antagonists & inhibitors | Cues | Guanfacine - pharmacology | Reinforcement (Psychology) | Rats | Brimonidine Tartrate | Imidazoles - pharmacology | Pyrimidines - pharmacology | Adrenergic alpha-2 Receptor Antagonists - pharmacology | Rats, Sprague-Dawley | Drug-Seeking Behavior - drug effects | Clonidine - antagonists & inhibitors | Cocaine - administration & dosage | Cocaine - pharmacology | Pyrroles - pharmacology | Animals | Imidazoles - antagonists & inhibitors | Imidazoline Receptors - agonists | Naphthyridines - pharmacology | Index Medicus
Journal Article
Anesthesia and Analgesia, ISSN 0003-2999, 02/2013, Volume 116, Issue 2, pp. 463 - 472
BACKGROUND: Cannabinoid agonists induce norepinephrine release in central, spinal, and peripheral sites. Previous studies suggest an interaction between the... 
NEUROPATHIC PAIN | INFLAMED TISSUE | RAT FRONTAL-CORTEX | ALPHA-2-ADRENOCEPTOR SUBTYPES | FATTY-ACID AMIDE | SPINAL-CORD | ANESTHESIOLOGY | NONCATECHOLIC PHENYLETHYLAMINE DERIVATIVES | NOREPINEPHRINE RELEASE | FORMALIN TEST | DORSAL-ROOT GANGLION | Analgesics - pharmacology | Ethanolamines - pharmacology | Endocannabinoids - pharmacology | Rats, Wistar | Receptor, Cannabinoid, CB2 - agonists | Norepinephrine - physiology | Sympathetic Nervous System - drug effects | Dinoprostone | Male | Cannabinoid Receptor Agonists - pharmacology | Peripheral Nerves - drug effects | Yohimbine - pharmacology | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Receptor, Cannabinoid, CB1 - agonists | Adrenergic alpha-1 Receptor Antagonists - pharmacology | Prazosin - pharmacology | Palmitic Acids - pharmacology | Palmitic Acids - antagonists & inhibitors | Morpholines - pharmacology | Rats | Pain Measurement - drug effects | Ethanolamines - antagonists & inhibitors | Polyunsaturated Alkamides - antagonists & inhibitors | Adrenergic alpha-2 Receptor Antagonists - pharmacology | Arachidonic Acids - pharmacology | Animals | Arachidonic Acids - antagonists & inhibitors | Polyunsaturated Alkamides - pharmacology | Adrenergic Uptake Inhibitors - pharmacology | Endocannabinoids - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Pain, ISSN 0304-3959, 2005, Volume 117, Issue 1, pp. 171 - 181
Protein kinase C (PKC) is able to phosphorylate several cellular components that serve as key regulatory components in signal transduction pathways of... 
Nociception | Protein kinase C | Substance P | Phorbol ester | MAP kinase | Mice | Glutamate | phorbol ester | PRIMARY AFFERENT NEURONS | INFLAMMATORY HYPERALGESIA | nociception | VANILLOID RECEPTOR | NECROSIS-FACTOR-ALPHA | RAT SENSORY NEURONS | mice | NEUROSCIENCES | CLINICAL NEUROLOGY | NERVE GROWTH-FACTOR | IN-VITRO | SCIATIC-NERVE | ANESTHESIOLOGY | substance P | UP-REGULATION | protein kinase C | glutamate | Bradykinin - analogs & derivatives | Analgesics - pharmacology | Tetradecanoylphorbol Acetate - pharmacology | Capsaicin - pharmacology | Nociceptors - physiology | Tetradecanoylphorbol Acetate - analogs & derivatives | Male | Peptide Fragments - pharmacology | Extracellular Signal-Regulated MAP Kinases - metabolism | Pain - chemically induced | Dose-Response Relationship, Drug | Salicylates - pharmacology | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Drug Interactions | Pain - enzymology | Time Factors | Pain - drug therapy | Protein Kinase C - metabolism | Sympatholytics - pharmacology | Behavior, Animal - drug effects | Indoles - pharmacology | Bradykinin - pharmacology | Blotting, Western - methods | Protein Kinase C - physiology | Calcitonin Gene-Related Peptide - pharmacology | Dipeptides - pharmacology | Enzyme Inhibitors - pharmacology | Capsaicin - analogs & derivatives | Chelating Agents - pharmacology | Enzyme Activation - drug effects | Antibodies - pharmacology | Pain Measurement - methods | Animals | Egtazic Acid - pharmacology | Ruthenium Red - pharmacology | Dizocilpine Maleate - pharmacology | Excitatory Amino Acid Antagonists | Guanethidine - pharmacology | Nociceptors - drug effects | Methyl aspartate | Nerve growth factor | Cell research | Protein kinases | Index Medicus
Journal Article
Journal Article
Journal Article
NEUROPSYCHOPHARMACOLOGY, ISSN 0893-133X, 01/2004, Volume 29, Issue 1, pp. 45 - 58
Intense or chronic stress can produce pathophysiological alterations in the systems involved in the stress response. The amygdala is a key component of the... 
GLUCOCORTICOID RECEPTOR | stress | amygdala | alpha adrenoceptors | NUCLEUS-TRACTUS-SOLITARIUS | BENZODIAZEPINE RECEPTORS | PSYCHIATRY | inhibitory synaptic transmission | ALPHA-ADRENOCEPTOR SUBTYPES | RECEPTOR MESSENGER-RNA | CYCLIC-AMP RESPONSE | GABA release | NOREPINEPHRINE RELEASE | NEUROSCIENCES | TRANSDUCTION PATHWAYS | RAT SENSORIMOTOR CORTEX | norepinelphrine | SYSTEM IN-VITRO | PHARMACOLOGY & PHARMACY | Receptors, Adrenergic, alpha-1 - metabolism | Amygdala - physiopathology | Norepinephrine - pharmacology | Amygdala - drug effects | Body Weight | gamma-Aminobutyric Acid - metabolism | 2-Amino-5-phosphonovalerate - pharmacology | Male | Adrenergic alpha-Antagonists - pharmacology | Anesthetics, Local - pharmacology | Patch-Clamp Techniques - methods | Dose-Response Relationship, Drug | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Drug Interactions | Time Factors | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Restraint, Physical - methods | Stress, Physiological - metabolism | Neurons - physiology | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Aging | Tetrodotoxin - pharmacology | Neurons - drug effects | Animals, Newborn | Isoquinolines - metabolism | Membrane Potentials - drug effects | Amygdala - metabolism | Morpholines - pharmacology | Phosphodiesterase Inhibitors - pharmacology | Rats | Amygdala - cytology | Imidazoles - pharmacology | Bicuculline - pharmacology | Rats, Sprague-Dawley | GABA Antagonists - pharmacology | Animals | Tetrahydronaphthalenes - pharmacology | In Vitro Techniques | Excitatory Amino Acid Antagonists | Neural Inhibition - drug effects | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 06/2007, Volume 74, Issue 3, pp. 406 - 415
Objectives: Canine atrial cardiomyocytes display a constitutively active, acetylcholine-regulated, time-dependent K current (I ) that contributes to atrial... 
Second-messengers | Signal transduction | Ion-channels | PROTEIN-KINASE-C | CARDIAC & CARDIOVASCULAR SYSTEMS | PHOSPHATIDYLINOSITOL 4,5-BISPHOSPHATE | signal transduction | RECTIFYING K+ CHANNELS | FORM STABLE COMPLEXES | VENTRICULAR MYOCYTES | GUINEA-PIG | second-messengers | SIGNALING COMPLEX | TRANSIENT OUTWARD CURRENT | ion-channels | ADENYLYL-CYCLASE | XENOPUS-OOCYTES | Maleimides - pharmacology | Adrenergic alpha-Antagonists - pharmacology | Phorbol 12,13-Dibutyrate - pharmacology | Type C Phospholipases - antagonists & inhibitors | Dose-Response Relationship, Drug | Propranolol - pharmacology | Isoquinolines - pharmacology | Phenylephrine - pharmacology | Prazosin - pharmacology | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Clonidine - pharmacology | Indoles - pharmacology | Receptors, Adrenergic, beta-1 - drug effects | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Action Potentials - drug effects | Acetylcholine - pharmacology | Heart Atria | Receptors, Adrenergic, alpha-1 - drug effects | Bee Venoms - pharmacology | Imidazoles - pharmacology | Protein Kinase C - antagonists & inhibitors | Potassium Channels - drug effects | Sulfonamides - pharmacology | Adrenergic Agents - pharmacology | Piperazines - pharmacology | Clonidine - analogs & derivatives | Patch-Clamp Techniques | Animals | Myocytes, Cardiac - drug effects | Isoproterenol - pharmacology | Dogs | Dihydropyridines - pharmacology | Myocytes, Cardiac - metabolism | Adrenergic alpha-Agonists - pharmacology | Propanolamines - pharmacology | Heart cells | Index Medicus
Journal Article
Physiology & Behavior, ISSN 0031-9384, 2010, Volume 102, Issue 1, pp. 1 - 6
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 11/2013, Volume 38, Issue 12, pp. 2484 - 2497
Activation of kappa-opioid receptors (KORs) in monoamine circuits results in dysphoria-like behaviors and stress-induced reinstatement of drug seeking in both... 
norepinephrine | kappa-opioid receptor | cocaine | reinstatement | locus coeruleus | b-adrenergic receptors | AGONIST SALVINORIN | PRESYNAPTIC INHIBITION | PSYCHIATRY | P38 MAPK | NEUROSCIENCES | BED NUCLEUS | PREFRONTAL CORTEX | CORTICOTROPIN-RELEASING-FACTOR | STRIA TERMINALIS | STRESS-INDUCED REINSTATEMENT | AXON TERMINALS | beta-adrenergic receptors | PHARMACOLOGY & PHARMACY | SALVINORIN-A | Naltrexone - analogs & derivatives | Adrenergic beta-1 Receptor Antagonists - pharmacology | Locus Coeruleus - drug effects | Male | Adrenergic alpha-2 Receptor Agonists - pharmacology | Adrenergic beta-Antagonists - pharmacology | Drug-Seeking Behavior | Naltrexone - pharmacology | Propranolol - pharmacology | Clonidine - pharmacology | Receptors, Adrenergic, beta - metabolism | Receptors, Adrenergic, alpha - metabolism | Receptors, Opioid, kappa - antagonists & inhibitors | Conditioning (Psychology) - drug effects | Mice, Inbred C57BL | Receptors, Opioid, kappa - agonists | Receptors, Opioid, kappa - metabolism | 3,4-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide, (trans)-Isomer - pharmacology | Cocaine - administration & dosage | Cocaine - pharmacology | Animals | Mice | Betaxolol - pharmacology | Propanolamines - pharmacology | Locus Coeruleus - metabolism | Index Medicus | Adrenergic receptors | beta adrenergic receptors | kappa opioid receptor | Catecholamines | Neuropharmacology | behavioral Science | Original | β-adrenergic receptors | opioids
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 02/2012, Volume 418, Issue 1, pp. 161 - 166
► We constructed the pharmacophore model for hERG channel facilitation. ► This model consists of one positively ionizable feature and three hydrophobic... 
Facilitation | Pharmacology | hERG channel | Pharmacophore model | HERG channel | INTERVAL PROLONGATION | K+ CURRENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | BLOCK | CARDIAC-ARRHYTHMIA | PREDICTION | RECTIFIER | BIOPHYSICS | TORSADE-DE-POINTES | DRUGS | LONG-QT-SYNDROME | POTASSIUM CHANNEL | Nortriptyline - chemistry | Metoprolol - pharmacology | Humans | Sotalol - chemistry | Imipramine - chemistry | ERG1 Potassium Channel | Nortriptyline - pharmacology | Propranolol - pharmacology | Chlorpheniramine - chemistry | Ether-A-Go-Go Potassium Channels - physiology | Metoprolol - chemistry | Haloperidol - pharmacology | Potassium Channel Blockers - pharmacology | Quantitative Structure-Activity Relationship | Atenolol - pharmacology | Terfenadine - pharmacology | Fluoxetine - pharmacology | Promethazine - chemistry | Xenopus laevis | Propranolol - chemistry | Fluoxetine - chemistry | Verapamil - pharmacology | Haloperidol - chemistry | Verapamil - chemistry | Ether-A-Go-Go Potassium Channels - antagonists & inhibitors | Animals | Sotalol - pharmacology | Atenolol - chemistry | Hydrophobic and Hydrophilic Interactions | Terfenadine - chemistry | Chlorpheniramine - pharmacology | Imipramine - pharmacology | Potassium Channel Blockers - chemistry | Promethazine - pharmacology | Analysis | Models | Universities and colleges | Index Medicus
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 2009, Volume 616, Issue 1, pp. 73 - 80
Harmaline-induced tremor in rodents is a model of essential tremor. We utilized a novel assay to quantify tremor activity in mice and found that tremor... 
Harmaline | Dopamine | Gabapentin | Primidone | Lithium | Glutamate | Carbamazepine | Valproate | Validity | D 1 receptor | GABA | Propranolol | Essential tremor | ICR mouse | D 2 receptor | γ-Hydroxy-butyrate | receptor | GAMMA-HYDROXYBUTYRIC ACID | NUCLEUS-ACCUMBENS DOPAMINE | D-2 receptor | SUBUNIT MESSENGER-RNAS | AMINO-ACIDS | SODIUM-VALPROATE | PHARMACOLOGY & PHARMACY | gamma-Hydroxy-butyrate | AMINOBUTYRIC-ACID | RECEPTOR SUBUNITS | NEUROTRANSMITTER RELEASE | INFERIOR OLIVE | D-1 receptor | DRUG-INDUCED TREMOR | Receptors, Glutamate - metabolism | Muscimol - pharmacology | gamma-Aminobutyric Acid - metabolism | Glutamates - metabolism | Male | Anticonvulsants - pharmacology | Dose-Response Relationship, Drug | Propranolol - pharmacology | Lithium Chloride - pharmacology | Neurotransmitter Agents - pharmacology | Tremor - chemically induced | Behavior, Animal - drug effects | Raclopride - pharmacology | Chlordiazepoxide - pharmacology | Dopamine - metabolism | Baclofen - pharmacology | Harmaline - pharmacology | Affect - drug effects | Benzazepines - pharmacology | Piperazines - pharmacology | Mice, Inbred ICR | Sodium Oxybate - pharmacology | Animals | Mice | Carbolines - pharmacology | Propranolol hydrochloride | Methyl aspartate | Pyridine | Analysis | Gamma-hydroxybutyrate | Chemical properties | Barbiturates | Index Medicus
Journal Article
FASEB Journal, ISSN 0892-6638, 03/2017, Volume 31, Issue 3, pp. 1193 - 1203
VEGF inhibitors, including receptor tyrosine kinase inhibitors, are used as adjunct therapies in a number of cancer treatments. An emerging issue with these... 
Hypertension | Cancer therapy | Regional hemodynamics | RTKI | VEGF | SIGNALING PATHWAY INHIBITORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TOXICITY | CANCER | RENAL-CELL CARCINOMA | BLOOD-PRESSURE | CELL BIOLOGY | cancer therapy | GROWTH-FACTOR RECEPTOR | INDUCED HYPERTENSION | TARGETED THERAPIES | regional hemodynamics | BIOLOGY | SUNITINIB | hypertension | FACTOR VEGF RECEPTORS | Niacinamide - analogs & derivatives | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Regional Blood Flow - drug effects | Piperidines - pharmacology | Propranolol - administration & dosage | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Quinazolines - administration & dosage | Adrenergic beta-Antagonists - administration & dosage | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Piperidines - administration & dosage | Consciousness | Pyrimidines - administration & dosage | Losartan - pharmacology | Rats | Pyrimidines - pharmacology | Sulfonamides - pharmacology | Rats, Sprague-Dawley | Niacinamide - administration & dosage | Protein Kinase Inhibitors - administration & dosage | Animals | Phenylurea Compounds - administration & dosage | Hemodynamics - drug effects | Phenylurea Compounds - pharmacology | Protein Kinase Inhibitors - pharmacology | Losartan - administration & dosage | Niacinamide - pharmacology | Quinazolines - pharmacology | Sulfonamides - administration & dosage | Drugs | Physiological effects | Drug development | Arteries | Rodents | Aorta | Protein-tyrosine kinase receptors | Blood pressure | Propranolol | Vascular endothelial growth factor | Protein-tyrosine kinase | Tyrosine | Jugular vein | Kidneys | Regional analysis | Catheters | Inhibitors | Coronary vessels | Phentolamine | Hemodynamics | Circulatory system | Cancer | Peritoneum | Index Medicus | Research
Journal Article