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Blood, ISSN 0006-4971, 01/2007, Volume 109, Issue 1, pp. 228 - 234
Life Sciences & Biomedicine | Hematology | Science & Technology | Fundamental and applied biological sciences. Psychology | Fundamental immunology | Biological and medical sciences | Immunobiology | Modulation of the immune response (stimulation, suppression) | CD8-Positive T-Lymphocytes - cytology | Dinoprostone - physiology | Humans | B-Lymphocyte Subsets - cytology | T-Lymphocyte Subsets - drug effects | CD8-Positive T-Lymphocytes - metabolism | Phosphorylation - drug effects | Macrophages - physiology | B-Lymphocyte Subsets - drug effects | Nitric Oxide - biosynthesis | Signal Transduction | CD4-Positive T-Lymphocytes - metabolism | Cell Division - drug effects | Receptors, Antigen, T-Cell - physiology | Mice, Knockout | B-Lymphocyte Subsets - metabolism | Mice | Tryptophan - pharmacology | HeLa Cells | Interleukin-2 - biosynthesis | Indoleamine-Pyrrole 2,3,-Dioxygenase - physiology | Nitric Oxide Synthase Type II - metabolism | Tetradecanoylphorbol Acetate - pharmacology | T-Lymphocyte Subsets - cytology | Concanavalin A - pharmacology | Nitric Oxide - pharmacology | Mesenchymal Stem Cells - physiology | STAT5 Transcription Factor - metabolism | Protein Processing, Post-Translational - drug effects | Nitric Oxide Synthase Type II - antagonists & inhibitors | Tryptophan - analogs & derivatives | Enzyme Induction - drug effects | Transforming Growth Factor beta - antagonists & inhibitors | NG-Nitroarginine Methyl Ester - pharmacology | Transforming Growth Factor beta - immunology | Cell Line | Antigens, CD19 - analysis | CD4-Positive T-Lymphocytes - cytology | Mice, Inbred C57BL | Nitric Oxide Synthase Type II - deficiency | Nitric Oxide - physiology | Cyclooxygenase Inhibitors - pharmacology | Animals | Lymphocyte Activation - drug effects | CD8-Positive T-Lymphocytes - drug effects | Ionomycin - pharmacology | CD4-Positive T-Lymphocytes - drug effects | Indoleamine-Pyrrole 2,3,-Dioxygenase - antagonists & inhibitors | Interferon-gamma - biosynthesis | Prostaglandin-Endoperoxide Synthases - physiology | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 08/2001, Volume 276, Issue 34, pp. 32008 - 32015
Journal Article
Journal Article
FEBS letters, ISSN 0014-5793, 2001, Volume 496, Issue 1, pp. 12 - 18
Lipopolysaccharide | Nitric oxide synthase | Cyclooxygenase | Inflammation | Flavonoid | Peroxisome proliferator-activated receptor-γ | IκB, inhibitor κB | IFN-γ, interferon-γ | Gst, glutathione S-transferase | IKK, IκB kinase | PPAR, peroxisome proliferator-activated receptor | PGE2, prostaglandin E2 | COX, cyclooxygenase | NSAIDs, non-steroidal anti-inflammatory drugs | NF-κB, nuclear factor-κB | NO, nitric oxide | iNOS, inducible nitric oxide synthase | LPS, lipopolysaccharide | 15d-PGJ2, 15-deoxy-Δ12,14 prostaglandin J2 | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Nitric Oxide Synthase - genetics | Promoter Regions, Genetic - drug effects | Nitric Oxide Synthase Type II | Transcription Factors - drug effects | Dose-Response Relationship, Drug | Kaempferols | Apigenin | Protein Binding - drug effects | Flavonoids - pharmacology | Genes, Reporter | Prostaglandin-Endoperoxide Synthases - genetics | Cell Line | Quercetin - analogs & derivatives | Receptors, Cytoplasmic and Nuclear - drug effects | Electrophoresis, Polyacrylamide Gel | Receptors, Cytoplasmic and Nuclear - agonists | Macrophages - cytology | Binding, Competitive - drug effects | Transcription Factors - metabolism | Macrophages - metabolism | Animals | Quercetin - pharmacology | Prostaglandin-Endoperoxide Synthases - metabolism | Flavonoids - metabolism | Rosiglitazone | Macrophages - drug effects | Mice | Nitric Oxide Synthase - metabolism | Thiazoles - pharmacology | Transcription Factors - agonists | Thiazolidinediones | Quercetin - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cell Biology | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | Index Medicus | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
Journal Article