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Cell Cycle, ISSN 1538-4101, 05/2015, Volume 14, Issue 10, pp. 1507 - 1516
.... The effect of CM was closely mimicked by prostaglandin E 2 (PGE 2 ), a bioactive lipid mediator in various physiological or pathological conditions... 
EP4 | bone-muscle crosstalk | Prostaglandin E | proliferation | myogenesis | reactive oxygen species | Bone-muscle crosstalk | Reactive oxygen species | Proliferation | Myogenesis | Immunohistochemistry | Reactive Oxygen Species - metabolism | Receptors, Prostaglandin E, EP2 Subtype - metabolism | Muscle, Skeletal - metabolism | Muscle, Skeletal - cytology | Alprostadil - analogs & derivatives | Myoblasts - drug effects | Muscle, Skeletal - drug effects | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Receptors, Prostaglandin E, EP4 Subtype - agonists | Receptors, Prostaglandin E, EP4 Subtype - genetics | Dinoprostone - pharmacology | Receptors, Prostaglandin E, EP3 Subtype - metabolism | Mice, Inbred C57BL | Receptors, Prostaglandin E, EP2 Subtype - genetics | Alprostadil - pharmacology | Cells, Cultured | Receptors, Prostaglandin E, EP3 Subtype - genetics | Thiophenes - pharmacology | Receptors, Prostaglandin E, EP1 Subtype - genetics | Receptors, Prostaglandin E, EP1 Subtype - metabolism | Triazoles - pharmacology | Animals | Signal Transduction - drug effects | Acetylcysteine - pharmacology | Receptors, Prostaglandin E, EP1 Subtype - agonists | Cell Proliferation - drug effects | Mice | G1 Phase Cell Cycle Checkpoints - drug effects | Receptors, Prostaglandin E, EP2 Subtype - agonists | Receptors, Prostaglandin E, EP3 Subtype - agonists
Journal Article
Neuroscience Letters, ISSN 0304-3940, 10/2011, Volume 504, Issue 3, pp. 185 - 190
.... Here, we investigated the function of PGE E-prostanoid (EP) receptors in the acute outcome of hypoxic-ischemic (HI... 
Hypoxic ischemic encephalopathy | EP1-4 receptors | G-protein coupled receptors | Misoprostol | PGE | Dinoprostone - physiology | Receptors, Prostaglandin E, EP4 Subtype - biosynthesis | Receptors, Prostaglandin E, EP1 Subtype - physiology | Misoprostol - pharmacology | Receptors, Prostaglandin E, EP4 Subtype - physiology | Hypoxia-Ischemia, Brain - pathology | Oxygen - metabolism | Receptors, Prostaglandin E, EP4 Subtype - antagonists & inhibitors | Receptors, Prostaglandin E, EP2 Subtype - biosynthesis | Neurons - metabolism | Hypoxia-Ischemia, Brain - metabolism | Animals, Newborn | Receptors, Prostaglandin E, EP4 Subtype - genetics | Cells, Cultured - drug effects | Hydrazines - pharmacology | Signal Transduction | Endothelial Cells - metabolism | Receptors, Prostaglandin E, EP2 Subtype - genetics | Receptors, Prostaglandin E, EP3 Subtype - biosynthesis | Receptors, Prostaglandin E, EP3 Subtype - genetics | Rats | Receptors, Prostaglandin E, EP1 Subtype - genetics | Hippocampus - pathology | Receptors, Prostaglandin E, EP2 Subtype - physiology | Rats, Sprague-Dawley | Oxazepines - pharmacology | Receptors, Prostaglandin E, EP3 Subtype - physiology | Hippocampus - metabolism | Animals | Receptors, Prostaglandin E, EP1 Subtype - biosynthesis | Glucose - metabolism | Cells, Cultured - metabolism | Receptors, Prostaglandin E, EP1 Subtype - antagonists & inhibitors | Hypoxia-Ischemia, Brain - physiopathology | Index Medicus | PGE2 | hypoxic ischemic encephalopathy | misoprostol
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 10, p. e0162532
... and microsomal prostaglandin E synthase-1. Administration of celecoxib, a COX-2 inhibitor, suppressed lymphangiogenesis in the granulation... 
SUBTYPE | LYMPHATIC VESSELS | INFLAMMATORY-BOWEL-DISEASE | ANGIOGENESIS | CLONING | MULTIDISCIPLINARY SCIENCES | MOUSE | C-TERMINAL TAIL | TUMOR-GROWTH | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Ear - physiology | Male | Receptors, Prostaglandin E, EP4 Subtype - deficiency | Wound Healing - drug effects | Macrophages - immunology | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Receptors, Prostaglandin E, EP4 Subtype - genetics | Receptors, Prostaglandin E, EP3 Subtype - metabolism | Cyclooxygenase 2 Inhibitors - pharmacology | Prostaglandin-E Synthases - metabolism | Mice, Inbred C57BL | Receptors, Prostaglandin E, EP3 Subtype - genetics | Macrophages - cytology | Gene Knockout Techniques | Vascular Endothelial Growth Factor D - biosynthesis | Lymphangiogenesis - drug effects | Up-Regulation - drug effects | Macrophages - metabolism | Animals | Receptors, Prostaglandin E, EP3 Subtype - deficiency | Signal Transduction - drug effects | Cyclooxygenase 2 - metabolism | Macrophages - drug effects | Mice | CD11b Antigen - metabolism | Vascular Endothelial Growth Factor C - biosynthesis | COX-2 inhibitors | Care and treatment | Wound healing | Analysis | Synthetic prostaglandins E | Hyaluronic acid | Macrophages | Vascular endothelial growth factor | Wounds and injuries | Cell proliferation | Homeostasis | Prostaglandin E2 | Biochemistry | Immunity | Experiments | Recruitment | Angiogenesis | Hyaluronan | Receptors | Prostaglandin E receptors | Rodents | Animal tissues | Tumor necrosis factor-TNF | Physiology | Prostaglandin E | Medical research | CD11b antigen | Granulation | Cytokines | Cloning | Inflammation | Pharmacology | Celecoxib | Metabolism | Prostaglandin-E synthase | Endothelial cells | Medicine | Pathology | Signaling | Ear | Healing | Cyclooxygenase-2
Journal Article
Pharmacology & therapeutics (Oxford), ISSN 0163-7258, 2013, Volume 138, Issue 3, pp. 485 - 502
The large variety of biological functions governed by prostaglandin (PG) E is mediated by signaling through four distinct E-type prostanoid (EP) receptors... 
Vascular disease | Osteoporosis | Prostaglandins | Inflammation | Renal function | Cancerogenesis | HUMAN DENDRITIC CELLS | DIETARY SALT INTAKE | DUODENAL BICARBONATE SECRETION | MICROVASCULAR ENDOTHELIAL-CELLS | PROSTAGLANDIN-E-RECEPTOR | AGONIST-INDUCED INTERNALIZATION | BREAST-CANCER CELLS | INFLAMMATORY-BOWEL-DISEASE | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | PHARMACOLOGY & PHARMACY | NF-KAPPA-B | Neoplasms - metabolism | Receptors, Prostaglandin E, EP4 Subtype - agonists | Lung Diseases - metabolism | Cardiovascular Diseases - metabolism | Humans | Bone Diseases - metabolism | Dinoprostone - metabolism | Animals | Gastrointestinal Diseases - metabolism | Receptors, Prostaglandin E, EP4 Subtype - antagonists & inhibitors | Immunologic Factors - metabolism | Kidney Diseases - metabolism | Receptors, Prostaglandin E, EP4 Subtype - metabolism | CREB, cAMP-response element-binding protein | Associate editor | TX, thromboxane receptor | EPRAP, EP4 receptor-associated protein | PK, protein kinase | COX, cyclooxygenase | AMPK, AMP-activated protein kinase | ICAM-1, intercellular adhesion molecule-1 | PI3K, phosphatidyl insositol 3-kinase | CFTR, cystic fibrosis transmembrane conductance regulator | IP, I-type prostanoid receptor | Epac, exchange protein activated by cAMP | NF-κB, nuclear factor kappa-light-chain-enhancer of activated B cells | NSAID, non-steroidal anti-inflammatory drug | IFN, interferon | MEK, MAP kinase kinase | PG, prostaglandin | DSS, dextran sodium sulfate | eNOS, endothelial nitric oxide synthase | Ig, immunoglobulin | P. Holzer | ICER, inducible cAMP early repressor | EGFR, epidermal growth factor receptor | FEM1a, feminization 1 homolog a | ERK, extracellular signal-regulated kinase | MCP, monocyte chemoattractant protein | 5-HETE, 5-hydroxyeicosatetraenoic acid | IL, interleukin | GRK, G protein-coupled receptor kinase | MAP, mitogen-activated protein kinase | LPS, lipopolysaccharide | ClC, chloride channel | DP, D-type prostanoid receptor | FP, F-type prostanoid receptor | TP, T-type prostanoid receptor | cAMP, cyclic adenylyl monophosphate | EP, E-type prostanoid receptor
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2011, Volume 286, Issue 3, pp. 2331 - 2342
...)-induced spinal neuroinflammation. Interestingly, we found that activation of E-prostanoid (EP... 
RAT SPINAL-CORD | TUMOR-NECROSIS-FACTOR | ISCHEMIC TOLERANCE | LIGAND-BINDING | NEONATAL-RAT | E RECEPTOR SUBTYPES | MESSENGER-RNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | PAIN HYPERSENSITIVITY | LIPOPOLYSACCHARIDE | INNATE IMMUNITY | Inflammation - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Myelitis - chemically induced | Myelitis - enzymology | Tumor Necrosis Factor-alpha - genetics | Interleukin-1beta - genetics | Receptors, Prostaglandin E - genetics | Cyclooxygenase 2 - genetics | Interleukin-1beta - metabolism | Intramolecular Oxidoreductases - metabolism | Prostaglandins - genetics | Disease Models, Animal | Prostaglandin-Endoperoxide Synthases - genetics | Receptors, Epoprostenol - metabolism | Lipopolysaccharides - toxicity | Myelitis - genetics | Receptors, Thromboxane A2, Prostaglandin H2 - metabolism | Cells, Cultured | Rats | Receptors, Thromboxane A2, Prostaglandin H2 - genetics | Rats, Sprague-Dawley | Mice, Knockout | Up-Regulation - drug effects | Receptors, Epoprostenol - genetics | Animals | Intramolecular Oxidoreductases - genetics | Prostaglandin-Endoperoxide Synthases - metabolism | Prostaglandin-E Synthases | Cyclooxygenase 2 - metabolism | Inflammation - genetics | Mice | Inflammation - enzymology | Receptors, Prostaglandin E - metabolism | Prostaglandins - metabolism | Prostaglandins | mPGES-1 | Immunology | Spinal Cord | Innate Immunity | Tumor Necrosis Factor (TNF) | Inflammation | Cyclooxygenase (COX) Pathway | Cyclic AMP (cAMP) | Microglia
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2011, Volume 31, Issue 5, pp. 1049 - 1058
OBJECTIVE—One of the hallmarks of inflammation is lymphangiogesis that drains the interstitial fluids. During chronic inflammation, angiogenesis is induced by... 
lymphangiogenesis | endothelium | prostaglandins | vascular biology | angiogenesis | Membrane Glycoproteins - metabolism | Receptors, Prostaglandin E, EP2 Subtype - metabolism | Granulation Tissue - physiopathology | Glycoproteins - metabolism | Male | Lymphatic Vessels - drug effects | Granulation Tissue - drug effects | Lymphatic Vessels - metabolism | Time Factors | Receptors, Prostaglandin E, EP4 Subtype - deficiency | Injections, Subcutaneous | Intramolecular Oxidoreductases - metabolism | Fibroblasts - metabolism | Pyrazoles - pharmacology | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Receptors, Prostaglandin E, EP4 Subtype - genetics | Receptors, Prostaglandin E, EP3 Subtype - metabolism | Cyclooxygenase 2 Inhibitors - pharmacology | Mice, Inbred C57BL | Receptors, Prostaglandin E, EP2 Subtype - genetics | Cells, Cultured | Receptors, Prostaglandin E, EP3 Subtype - genetics | Receptors, Prostaglandin E, EP1 Subtype - genetics | Celecoxib | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Fibroblast Growth Factor 2 - administration & dosage | Sulfonamides - pharmacology | Granulation Tissue - metabolism | Receptors, Prostaglandin E, EP1 Subtype - metabolism | Mice, Knockout | Dinoprostone - metabolism | Lymphangiogenesis - drug effects | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Receptors, Prostaglandin E, EP3 Subtype - deficiency | Signal Transduction - drug effects | Vascular Endothelial Growth Factor C - metabolism | Fibroblasts - drug effects | Prostaglandin-E Synthases | Cyclooxygenase 2 - metabolism | Macrophages - drug effects | Mice | Lymphatic Vessels - physiopathology
Journal Article
Circulation, ISSN 0009-7322, 07/2016, Volume 134, Issue 4, pp. 328 - 338
Journal Article
Journal Article
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2016, Volume 137, Issue 1, pp. 99 - 107.e7
Journal Article