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Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2011, Volume 80, Issue 5, pp. 1512 - 1521
Purpose To investigate vasculatures and microenvironment in tumors growing from preirradiated tissues (pre-IR tumors) and study the vascular responses of... 
Radiology | Hematology, Oncology and Palliative Medicine | Tumor bed effects | Microvascular density | Antiangiogenic therapy | Green fluorescent protein–tagged bone marrow | Radiation | Green fluorescent protein-tagged bone marrow | PROGENITOR CELLS | INDUCED INJURY | ANGIOGENESIS | ADENOCARCINOMA | HYPOXIA | BED STROMA | IRRADIATION | ONCOLOGY | RADIOTHERAPY | NEOVASCULARIZATION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | ENDOTHELIAL GROWTH-FACTOR | Green Fluorescent Proteins | Neoplasm Recurrence, Local - radiotherapy | Cell Hypoxia - physiology | Coloring Agents | Radiation Tolerance | Cell Aggregation - drug effects | Neoplasm Recurrence, Local - drug therapy | Male | Tumor Microenvironment - physiology | Neovascularization, Pathologic - etiology | Nitroimidazoles | Cell Aggregation - physiology | Neoplasms, Radiation-Induced - blood supply | Neovascularization, Pathologic - pathology | Tumor Burden - physiology | Neoplasm Recurrence, Local - pathology | Prostatic Neoplasms | Bone Marrow Transplantation | Angiogenesis Inhibitors - therapeutic use | Neovascularization, Pathologic - radiotherapy | Neoplasms, Radiation-Induced - pathology | Pyrroles - therapeutic use | Neoplasms, Radiation-Induced - radiotherapy | Macrophages - physiology | Tumor Microenvironment - drug effects | Cell Hypoxia - drug effects | Macrophages - pathology | Mice, Inbred C57BL | Neoplasm Recurrence, Local - blood supply | Endothelial Cells - radiation effects | Radiation Dosage | Xenograft Model Antitumor Assays - methods | Neoplasms, Radiation-Induced - drug therapy | Radiation-Sensitizing Agents | Tumor Microenvironment - radiation effects | Animals | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Indoles - therapeutic use | Mice | Drug Resistance, Neoplasm - physiology | Salvage Therapy - methods | Endothelial Cells - drug effects | Antimitotic agents | Antineoplastic agents | Tumors | BONE MARROW | MALE GENITALS | ANIMAL TISSUES | ANIMAL CELLS | MEDICINE | PHOTON EMISSION | THERAPY | GLANDS | LUMINESCENCE | RADIATIONS | RADIOLOGY AND NUCLEAR MEDICINE | PHAGOCYTES | HEMATOPOIETIC SYSTEM | SOMATIC CELLS | CONNECTIVE TISSUE CELLS | RADIOLOGY | PROSTATE | NEOPLASMS | MACROPHAGES | IONIZING RADIATIONS | NUCLEAR MEDICINE | ORGANS | DISEASES | FLUORESCENCE | ANOXIA | CARCINOMAS | EMISSION | BODY
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 03/2003, Volume 95, Issue 6, pp. 458 - 470
Background: Expression of platelet-derived growth factor (PDGF) and activation (by autophosphorylation) of its receptor (PDGF-R), a tyrosine kinase, are... 
BREAST-CANCER | MESSENGER-RNAS | ONCOLOGY | GLIOMA CELL-LINES | FACTOR-BETA-RECEPTOR | ENDOTHELIAL-CELLS | FACTOR PDGF | TYROSINE KINASE INHIBITOR | FACTOR-B-CHAIN | ORGAN MICROENVIRONMENT | FACTOR-ALPHA | Immunohistochemistry | Piperazines - administration & dosage | Prostatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Bone Neoplasms - secondary | Male | Antineoplastic Agents - administration & dosage | Microcirculation - drug effects | Bone Neoplasms - metabolism | Enzyme Inhibitors - administration & dosage | Antineoplastic Agents, Phytogenic - administration & dosage | Bone Neoplasms - blood supply | Neoplasms, Experimental | Platelet-Derived Growth Factor - drug effects | Antineoplastic Agents - pharmacology | Bone Neoplasms - drug therapy | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Tumor Cells, Cultured | Paclitaxel - administration & dosage | Disease Models, Animal | Radiographic Image Enhancement | In Situ Nick-End Labeling | Prostatic Neoplasms - pathology | Bone Neoplasms - enzymology | Administration, Oral | Bone Neoplasms - diagnostic imaging | Pyrimidines - administration & dosage | Enzyme Inhibitors - pharmacology | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Blotting, Western | Cell Division - drug effects | Platelet-Derived Growth Factor - metabolism | Animals | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Mice, Nude | Receptors, Platelet-Derived Growth Factor - metabolism | Fluorescent Antibody Technique | Mice | Benzamides | Receptors, Platelet-Derived Growth Factor - drug effects | Protein-Tyrosine Kinases - antagonists & inhibitors | Development and progression | Bone cancer | Metastasis | Complications | Prostate cancer
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35710
TIMP-2 protein has been intensively studied as a promising anticancer candidate agent, but the in vivo mechanism underlying its anticancer effect has not been... 
APOPTOSIS | ACTIVATION | THERAPY | MATRIX-METALLOPROTEINASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | ESCHERICHIA-COLI | DOWN-REGULATION | TISSUE INHIBITOR | CELL-PROLIFERATION | CANCER | Human Umbilical Vein Endothelial Cells | Humans | Recombinant Fusion Proteins - therapeutic use | Serum Albumin - genetics | Male | Prostate - metabolism | Tissue Inhibitor of Metalloproteinase-2 - genetics | Neovascularization, Pathologic - pathology | Tissue Inhibitor of Metalloproteinase-2 - therapeutic use | Prostate - pathology | Prostatic Neoplasms - genetics | Angiogenesis Inhibitors - therapeutic use | Prostate - drug effects | Prostate - blood supply | Gene Expression Regulation, Neoplastic - drug effects | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - blood supply | Prostatic Neoplasms - pathology | Angiogenesis Inhibitors - genetics | Rats | Serum Albumin - therapeutic use | Matrix Metalloproteinase 2 - genetics | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Recombinant Fusion Proteins - genetics | Neovascularization, Pathologic - genetics | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Care and treatment | RNA | Growth | Albumin | Research | Tumors | Cancer | Laboratories | Science | Clinical trials | mRNA | Tissue inhibitor of metalloproteinase 2 | Tissues | Vascularization | Anticancer properties | Proteins | Angiogenesis | Reduction | Cell growth | Rodents | Penicillin | Inhibition | Fusion protein | Vascular endothelial growth factor | Human serum albumin | Melanoma | Serum albumin | Metabolism | Gene expression | CD31 antigen | Endothelial cells | Gelatinase A | In vivo methods and tests | Gene therapy | Prostate cancer | Umbilical vein | Apoptosis
Journal Article
Journal Article
Neoplasia, ISSN 1476-5586, 2012, Volume 14, Issue 10, pp. 915 - IN12
Abstract The prognosis of patients suffering from pancreatic cancer is still poor and novel therapeutic options are urgently needed. Recently, the... 
Oncology | STAT5b | extracellular signal-regulated kinase | vascular endothelial growth factor-A | HIF-1α | VEGF-D | vascular endothelial growth factor-D | MAPK | VEGF-A | urokinasetype plasminogen activator receptor | Dulbecco's modified Eagle's medium | IL-6 | interleukin-6 | DPAC | mitogen-activated protein kinase | DMEM | caveolin-1 | CAV-1 | hypoxia-inducible factor-1α | FCS | fetal calf serum | uPAR | ductal pancreatic adenocarcinoma | signal transducer and activator of transcription 5b | ERK | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | HEPATOCELLULAR-CARCINOMA | IN-VITRO | ACTIVATION | INVASION | ONCOLOGY | PROSTATE-CANCER | CELL-CYCLE ARREST | EXPRESSION | SIGNAL TRANSDUCER | Carcinoma, Pancreatic Ductal - secondary | RNA, Small Interfering - genetics | Phosphorylation | Cell Proliferation | Pancreatic Neoplasms - metabolism | Humans | Pancreatic Neoplasms - blood supply | Carcinoma, Pancreatic Ductal - metabolism | Neovascularization, Pathologic | Male | Carcinoma, Papillary - metabolism | Carcinoma, Papillary - secondary | Immunoenzyme Techniques | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Adenocarcinoma, Mucinous - blood supply | Carcinoma, Papillary - blood supply | Neoplasm Metastasis | Neoplasm Grading | STAT5 Transcription Factor - antagonists & inhibitors | Tumor Cells, Cultured | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Survival Rate | Adenocarcinoma, Mucinous - secondary | Blotting, Western | Animals | Mice, Nude | Adenocarcinoma, Mucinous - metabolism | Mice | Mice, Inbred BALB C | Carcinoma, Pancreatic Ductal - blood supply | Neoplasm Staging | Cell Movement
Journal Article