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International journal of molecular sciences, ISSN 1661-6596, 08/2016, Volume 17, Issue 8, pp. 1320 - 1320
Hippo/YAP | Protease | Wnt/β-catenin | Protease-activated receptors (PARs) | CXCR4 | PH-domain | G protein-coupled receptors (GPCRs) | LPA(1-6) | Protease-activated receptor | Oncogenes | Cancer | Neoplasms - metabolism | Signal Transduction - physiology | Animals | Receptors, G-Protein-Coupled - metabolism | Neoplasms - pathology | Humans | Proteins | Genomes | Proteases | Index Medicus | protease | protease-activated receptors (PARs) | oncogenes | protease-activated receptor | cancer
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 05/2004, Volume 24, Issue 18, pp. 4300 - 4312
Protein kinase C | Inflammation | TRPV1 | Substance P | Hyperalgesia | Protease-activated receptors | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Enzyme Activators - pharmacology | Hyperalgesia - chemically induced | Spinal Cord - drug effects | Spinal Cord - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Receptors, Drug - genetics | Humans | Receptor, PAR-2 - metabolism | Type C Phospholipases - metabolism | Male | Neurons - cytology | RNA, Messenger - metabolism | Receptors, Drug - agonists | Type C Phospholipases - antagonists & inhibitors | Kidney - metabolism | Protein Kinase C - metabolism | Neurons - metabolism | Calcitonin Gene-Related Peptide - metabolism | Neurons - drug effects | Receptor, PAR-2 - genetics | Hyperalgesia - metabolism | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Kidney - cytology | Protein Kinase C - antagonists & inhibitors | Rats, Sprague-Dawley | Receptors, Drug - metabolism | Mice, Knockout | Animals | Receptor, PAR-2 - agonists | Substance P - metabolism | Mice | Index Medicus | protease-activated receptors | Cellular | inflammation | hyperalgesia | Molecular | substance P | protein kinase C
Journal Article
Pharmacology & therapeutics (Oxford), ISSN 0163-7258, 2011, Volume 131, Issue 1, pp. 142 - 170
Transducers | TRPC6 | TRPA1 | Chemesthesis | Gastrointestinal cancer | TRPC4 | Hypersensitivity | Gastrointestinal motility | Inflammation | TRPV1 | Hyperalgesia | Inflammatory bowel disease | Mechanosensation | Pain | TRPV6 | TRPV4 | Taste | TRPM5 | Chemosensation | TRPM6 | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Transient Receptor Potential Channels - antagonists & inhibitors | Animals | Digestive System Diseases - drug therapy | Digestive System Diseases - metabolism | Transient Receptor Potential Channels - agonists | Humans | Index Medicus | GI, gastrointestinal | GPCR, G protein-coupled receptor | TRPA, transient receptor potential ankyrin | TRPC, transient receptor potential canonical (or classical) | DRG, dorsal root ganglion | PKD, polycystic kidney disease | TRPV, transient receptor potential vanilloid | ICC, interstitial cell of Cajal | AITC, allyl isothiocyanate | DSS, dextran sulfate sodium | 5-HT, 5-hydroxytryptamine | mRNA, messenger ribonucleic acid | TRPP, transient receptor potential polycystin | siRNA, small interfering ribonucleic acid | RNA, ribonucleic acid | TNBS, trinitrobenzene sulfonic acid | CCK, cholecystokinin | TRPM, transient receptor potential melastatin | TRP, transient receptor potential | PAR, protease-activated receptor | CGRP, calcitonin gene-related peptide
Journal Article
Pharmacology & therapeutics (Oxford), ISSN 0163-7258, 2011, Volume 130, Issue 3, pp. 248 - 282
PAR | GPCR | Protease | Protease activated receptor | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Receptors, Proteinase-Activated - chemistry | Amino Acid Sequence | Humans | Molecular Sequence Data | Cell Membrane - genetics | Cell Membrane - chemistry | Receptors, Proteinase-Activated - physiology | Animals | Clinical Trials as Topic - trends | Coronary Artery Disease - physiopathology | Coronary Artery Disease - genetics | Signal Transduction - physiology | Cell Membrane - metabolism | Protein Binding - physiology | Index Medicus
Journal Article
American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 12/2009, Volume 297, Issue 6, pp. 1681 - 1690
Calcitonin gene-related peptide | Ischemia-reperfusion | Transient receptor potential vanilloid 1 | Protein kinase C-ε | Substance P | Gene knockout | Protease-activated receptors | Life Sciences & Biomedicine | Physiology | Science & Technology | L-Lactate Dehydrogenase - metabolism | Receptor, PAR-2 - metabolism | Myocardial Contraction - drug effects | Male | Peptide Fragments - pharmacology | Receptors, Calcitonin Gene-Related Peptide - metabolism | Recovery of Function | TRPV Cation Channels - deficiency | Cyclic AMP-Dependent Protein Kinase Catalytic Subunits - metabolism | TRPV Cation Channels - metabolism | Myocardial Reperfusion Injury - pathology | Isoquinolines - pharmacology | Cyclic AMP-Dependent Protein Kinase Catalytic Subunits - antagonists & inhibitors | Myocardial Infarction - pathology | Myocardium - metabolism | Radioimmunoassay | Protein Kinase C-epsilon - antagonists & inhibitors | Myocardial Infarction - physiopathology | Receptors, Neurokinin-1 - metabolism | Calcitonin Gene-Related Peptide - metabolism | Isoindoles - pharmacology | Neurokinin-1 Receptor Antagonists | Disease Models, Animal | Calcitonin Gene-Related Peptide - pharmacology | Receptors, Calcitonin Gene-Related Peptide - antagonists & inhibitors | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Ventricular Pressure - drug effects | Myocardium - pathology | Cardiotonic Agents - pharmacology | Myocardial Infarction - metabolism | Sulfonamides - pharmacology | Myocardial Reperfusion Injury - physiopathology | TRPV Cation Channels - genetics | Mice, Knockout | Myocardial Reperfusion Injury - metabolism | Animals | Protein Kinase C-epsilon - metabolism | Fluorescent Antibody Technique | Receptor, PAR-2 - agonists | Substance P - metabolism | Mice | Myocardial Infarction - prevention & control | Protein Kinase Inhibitors - pharmacology | Coronary Circulation - drug effects | Oligopeptides - pharmacology | Myocardial Reperfusion Injury - prevention & control | Prevention | Cell receptors | Physiological aspects | Myocardial ischemia | Development and progression | Genetic aspects | Protein kinases | Proteins | Hypotheses | Peptides | Ischemia | Proteases | Genes | Mutation | Index Medicus | protease-activated receptors | protein kinase C-ε | gene knockout | transient receptor potential vanilloid 1 | calcitonin gene-related peptide | ischemia-reperfusion | substance P
Journal Article