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Journal of Nutrition, ISSN 0022-3166, 2014, Volume 144, Issue 7, pp. 1002 - 1008
Dietary fiber intake is associated with lower incidence and mortality from disease, but the underlying mechanisms of these protective effects are unclear. We... 
Fructans - chemistry | Intestinal Mucosa - metabolism | Toll-Like Receptor 2 - agonists | Toll-Like Receptor 2 - antagonists & inhibitors | Toll-Like Receptor 2 - genetics | Humans | Colon - drug effects | Kidney - immunology | Protective Agents - metabolism | Membrane Transport Modulators - antagonists & inhibitors | Membrane Transport Modulators - toxicity | Tetradecanoylphorbol Acetate - analogs & derivatives | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Colon - immunology | NF-kappa B - metabolism | Antibodies, Blocking - pharmacology | Protective Agents - chemistry | Transcription Factor AP-1 - agonists | Gastrointestinal Agents - chemistry | Tetradecanoylphorbol Acetate - antagonists & inhibitors | Transcription Factor AP-1 - metabolism | Intestinal Mucosa - drug effects | Kidney - metabolism | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | Diglycerides - pharmacology | Intestinal Mucosa - immunology | Fructans - antagonists & inhibitors | Molecular Structure | Anti-Inflammatory Agents, Non-Steroidal - metabolism | NF-kappa B - agonists | Tight Junctions - drug effects | Recombinant Proteins - metabolism | Tight Junctions - metabolism | Cell Line | Kidney - drug effects | Gastrointestinal Agents - antagonists & inhibitors | Recombinant Proteins - chemistry | Fructans - metabolism | Toll-Like Receptor 2 - metabolism | Colon - metabolism | Prebiotics - analysis | Tetradecanoylphorbol Acetate - toxicity | Tight Junctions - immunology | Ligands | Oligopeptides - pharmacology | Gastrointestinal Agents - metabolism
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 09/2017, Volume 139, pp. 71 - 81
has long been part of the traditional herbal medicine system in Asia and in Scandinavia. Andrographolide was isolated as a major bioactive constituent of in... 
Hepatitis | NF-κB | Autoimmune | Neurodegeneration | Nrf2 | Asthma | OXIDATIVE STRESS | ALZHEIMERS-DISEASE | COGNITIVE IMPAIRMENT | CELL-CYCLE ARREST | PROTECTIVE ACTIVITY | IN-VITRO | NF-kappa B | INDUCED PULMONARY-FIBROSIS | SIGNALING PATHWAY | PHARMACOLOGY & PHARMACY | COMBINATION KAN-JANG | NF-KAPPA-B | Liver Cirrhosis - immunology | Humans | Drugs, Investigational - therapeutic use | NF-E2-Related Factor 2 - agonists | Protective Agents - chemistry | Neurodegenerative Diseases - drug therapy | Hepatitis - drug therapy | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hepatitis - metabolism | Pneumonia - immunology | Autoimmune Diseases - metabolism | Autoimmune Diseases - drug therapy | Diterpenes - pharmacology | Chemical and Drug Induced Liver Injury - prevention & control | NF-kappa B p50 Subunit - antagonists & inhibitors | NF-kappa B p50 Subunit - metabolism | Autoimmune Diseases - prevention & control | Chemical and Drug Induced Liver Injury - immunology | Signal Transduction - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Models, Biological | NF-E2-Related Factor 2 - metabolism | Oxidative Stress - drug effects | Pneumonia - metabolism | Drugs, Investigational - adverse effects | Drugs, Investigational - pharmacology | Dermatitis - immunology | Neurodegenerative Diseases - prevention & control | Protective Agents - metabolism | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Protective Agents - therapeutic use | Neurodegenerative Diseases - immunology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Drugs, Investigational - chemistry | Hepatitis - prevention & control | Drug Design | Liver Cirrhosis - metabolism | Diterpenes - chemistry | Liver Cirrhosis - drug therapy | Diterpenes - therapeutic use | Liver Cirrhosis - prevention & control | Autoimmune Diseases - immunology | Dermatitis - prevention & control | Neurodegenerative Diseases - metabolism | Animals | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Pneumonia - drug therapy | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | NF-kappa B p50 Subunit - chemistry | Hepatitis - immunology | Diterpenes - adverse effects | Dermatitis - drug therapy | Dermatitis - metabolism | Antioxidants | Medical colleges | Lead compounds | Drug discovery | Anti-inflammatory drugs | Analysis | Index Medicus
Journal Article
Journal Article
Laboratory Investigation, ISSN 0023-6837, 01/2018, Volume 98, Issue 1, pp. 79 - 94
Acute kidney injury (AKI), characterized by aggressive inflammatory responses and destruction of renal resident cells, can cause abrupt kidney dysfunction. To... 
FIBROSIS | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | FRATRICIDE | INFLAMMATION | IN-VIVO | KINASE | PROGRAMMED NECROSIS | PATHOLOGY | CONTRIBUTES | EPIDEMIOLOGY | NONAPOPTOTIC CELL-DEATH | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Kidney - pathology | Apoptosis - drug effects | Humans | Kidney - immunology | Protective Agents - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - chemistry | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Male | Protective Agents - chemistry | Protective Agents - therapeutic use | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Kidney - metabolism | Flavanones - chemistry | RNA Interference | Antineoplastic Agents - adverse effects | Protective Agents - pharmacology | Acute Kidney Injury - chemically induced | Antineoplastic Agents - pharmacology | Anti-Inflammatory Agents, Non-Steroidal - metabolism | Cisplatin - antagonists & inhibitors | Kidney Tubules - metabolism | Binding Sites | Biomarkers - metabolism | Kidney Tubules - immunology | Microscopy, Electron, Transmission | Catalytic Domain | Kidney - drug effects | Flavanones - therapeutic use | Kidney Tubules - drug effects | Acute Kidney Injury - pathology | Flavanones - pharmacology | Mice, Inbred C57BL | Kidney Tubules - ultrastructure | Biomarkers - blood | Cisplatin - pharmacology | Antineoplastic Agents - chemistry | Acute Kidney Injury - prevention & control | Animals | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Flavanones - metabolism | Cell Line, Tumor | Cisplatin - adverse effects | Molecular Docking Simulation | Acute Kidney Injury - metabolism | Macrophage Activation - drug effects | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Cell Line, Transformed | Creatinine | NF-κB protein | Therapy | Kidneys | Syngeneic grafts | Inflammation | Hepatoma | Electron microscopy | Macrophages | Cisplatin | Molecular docking | Urea | Rodents | Infiltration | Inhibition | Injuries
Journal Article
Antioxidants and Redox Signaling, ISSN 1523-0864, 12/2010, Volume 13, Issue 11, pp. 1763 - 1811
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 09/2012, Volume 53, Issue 6, pp. 1298 - 1307
Oxidative damage and inflammation are related to the pathogenesis of age-related macular degeneration (AMD). Epidemiologic studies suggest that insufficient... 
Lutein | Zeaxanthin | Inflammation | Photooxidation | RPE | Proteasome | C-REACTIVE PROTEIN | OXIDATIVE STRESS | AGE-RELATED MACULOPATHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACULAR DEGENERATION | COMPLEMENT FACTOR-H | PROTEOLYTIC PATHWAY | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | CONJUGATING ENZYMES | NF-KAPPA-B | UBIQUITIN-PROTEASOME PATHWAY | Radiation-Protective Agents - metabolism | Interleukin-8 - genetics | Retinal Pigment Epithelium - metabolism | Gene Expression - drug effects | Humans | Zeaxanthins | Down-Regulation - radiation effects | Macular Degeneration - drug therapy | Photochemical Processes | Retinal Pigment Epithelium - pathology | Ultraviolet Rays | Culture Media | Inflammation Mediators - metabolism | Radiation-Protective Agents - pharmacology | Chemokine CCL2 - metabolism | Oxidative Stress - radiation effects | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Retinal Pigment Epithelium - drug effects | Interleukin-6 - genetics | Oxidation-Reduction | Cells, Cultured | Chemokine CCL2 - genetics | Lutein - metabolism | Down-Regulation - drug effects | Gene Expression - radiation effects | Complement Factor H - metabolism | Xanthophylls - metabolism | Xanthophylls - pharmacology | Proteasome Endopeptidase Complex - metabolism | Complement Factor H - genetics | Dietary Supplements | Lutein - pharmacology | Macular Degeneration - pathology | Macular degeneration | Analysis | Genes | Genetic research | Universities and colleges | Ophthalmology
Journal Article