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Nature Medicine, ISSN 1078-8956, 11/2018, Volume 24, Issue 11, pp. 1732 - 1742
Journal Article
Brain, ISSN 0006-8950, 2017, Volume 140, Issue 7, pp. 1885 - 1899
Journal Article
Kidney International, ISSN 0085-2538, 08/2018, Volume 94, Issue 2, pp. 259 - 267
Pax genes encode developmental regulators that are expressed in a variety of tissues and control critical events in morphogenesis. In the kidney, Pax2 and Pax8... 
drug development | renal cancer | Pax8 | polycystic kidney disease | Pax2 | renal epithelia | DOMAIN-DNA COMPLEX | CRYSTAL-STRUCTURE | RENAL-CELL CARCINOMA | PAIRED DOMAIN | REPRESSION DOMAIN | EPITHELIAL-CELLS | GENE | BRCT-DOMAIN | IN-VIVO | TRANSCRIPTION ACTIVATION | UROLOGY & NEPHROLOGY | Polycystic Kidney Diseases - genetics | Kidney Neoplasms - genetics | Urothelium - cytology | Epithelial Cells - metabolism | PAX2 Transcription Factor - metabolism | Urothelium - metabolism | Epithelial Cells - drug effects | Humans | Antineoplastic Agents - therapeutic use | Polycystic Kidney Diseases - drug therapy | PAX8 Transcription Factor - metabolism | Urothelium - pathology | Protein Domains - drug effects | Kidney - metabolism | Polycystic Kidney Diseases - pathology | PAX8 Transcription Factor - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | PAX2 Transcription Factor - antagonists & inhibitors | Molecular Targeted Therapy - methods | PAX8 Transcription Factor - genetics | Cell Proliferation - genetics | Gene Expression Regulation, Developmental - drug effects | Kidney - growth & development | Epithelial Cells - pathology | PAX2 Transcription Factor - genetics | Kidney - cytology | Disease Progression | Animals | Kidney Neoplasms - pathology | Cell Proliferation - drug effects | Kidney Neoplasms - drug therapy | Urothelium - drug effects
Journal Article
Leukemia, ISSN 0887-6924, 10/2018, Volume 32, Issue 10, pp. 2224 - 2239
Bromodomain and extraterminal (BET) domain containing protein (BRD)-4 modulates the expression of oncogenes such as c-myc, and is a promising therapeutic... 
INHIBITION | THERAPEUTIC STRATEGY | ONCOLOGY | POOR-PROGNOSIS | KINASE | C-MYC | DEGRADATION | DRUG-RESISTANCE | HEMATOLOGY | PHASE-I | CANCER | BET BROMODOMAINS | Cell Survival - drug effects | Humans | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Multiple Myeloma - metabolism | Proto-Oncogene Proteins c-myc - metabolism | Xenograft Model Antitumor Assays | Protein Domains - drug effects | Amino Acid Motifs - drug effects | Multiple Myeloma - drug therapy | Animals | Proteins - metabolism | Ubiquitination - drug effects | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Cell Line, Tumor | Female | Mice, Inbred NOD | Antineoplastic Agents - pharmacology | Mice | Proto-Oncogene Proteins c-akt - metabolism | Drug Resistance, Neoplasm - drug effects | Molecular targeted therapy | Care and treatment | Innovations | Multiple myeloma | Development and progression | Genetic aspects | Gene expression | Health aspects | Oncogenes | Bortezomib | Dexamethasone | Immunomodulation | Therapeutic applications | Downstream effects | p53 Protein | Xenotransplantation | c-Myc protein | AKT protein | Myc protein | Tumor cell lines | Patients | Proteins | Time dependence | Ubiquitination | Molecular modelling | Cell lines | Xenografts | Proteasomes | Viability | Cancer | Apoptosis | multiple myeloma | PROTACs | drug resistance | BET protein family | c-MYC | BRD4
Journal Article
Nature, ISSN 0028-0836, 03/2017, Volume 543, Issue 7647, pp. 733 - 737
Journal Article