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Cell death and differentiation, ISSN 1476-5403, 2006, Volume 13, Issue 6, pp. 962 - 972
.... They encode for multiple p63, p73 or p53 proteins containing different protein domains (isoforms) due to multiple splicing, alternative promoter and alternative initiation of translation... 
Promoter | Development | Zebrafish | Splice | Drosophila | Cancer | development | P53-DEPENDENT APOPTOSIS | splice | BIOCHEMISTRY & MOLECULAR BIOLOGY | HOMOLOG P63 | TUMOR-SUPPRESSOR GENE | P73 | CELL BIOLOGY | DAMAGE RESPONSE | drosophila | TRANSACTIVATION | P53 FAMILY-MEMBER | promoter | cancer | zebrafish | EXPRESSION | ALTERNATIVELY SPLICED FORM | TRANSFORMED-CELLS | Neoplasms - metabolism | Alternative Splicing | Humans | Promoter Regions, Genetic - genetics | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Protein Isoforms - metabolism | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Tumor Suppressor Proteins - genetics | Trans-Activators - genetics | Apoptosis Regulatory Proteins - genetics | Cell Cycle Proteins - genetics | Transcription, Genetic | Nuclear Proteins - genetics | Tumor Suppressor Proteins - metabolism | Cell Cycle Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | DNA - metabolism | DNA-Binding Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | DNA - genetics | Tumor Protein p73 | Animals | Trans-Activators - metabolism | Mice | Transcription Factors | Mutation | Evolution, Molecular | Protein Isoforms - genetics
Journal Article
by Djebali, Sarah and Davis, Carrie A and Merkel, Angelika and Dobin, Alex and Lassmann, Timo and Mortazavi, Ali and Tanzer, Andrea and Lagarde, Julien and Lin, Wei and Schlesinger, Felix and Xue, Chenghai and Marinov, Georgi K and Khatun, Jainab and Williams, Brian A and Zaleski, Chris and Rozowsky, Joel and Röder, Maik and Kokocinski, Felix and Abdelhamid, Rehab F and Alioto, Tyler and Antoshechkin, Igor and Baer, Michael T and Bar, Nadav S and Batut, Philippe and Bell, Kimberly and Bell, Ian and Chakrabortty, Sudipto and Chen, Xian and Chrast, Jacqueline and Curado, Joao and Derrien, Thomas and Drenkow, Jorg and Dumais, Erica and Dumais, Jacqueline and Duttagupta, Radha and Falconnet, Emilie and Fastuca, Meagan and Fejes-Toth, Kata and Ferreira, Pedro and Foissac, Sylvain and Fullwood, Melissa J and Gao, Hui and Gonzalez, David and Gordon, Assaf and Gunawardena, Harsha and Howald, Cedric and Jha, Sonali and Johnson, Rory and Kapranov, Philipp and King, Brandon and Kingswood, Colin and Luo, Oscar J and Park, Eddie and Persaud, Kimberly and Preall, Jonathan B and Ribeca, Paolo and Risk, Brian and Robyr, Daniel and Sammeth, Michael and Schaffer, Lorian and See, Lei-Hoon and Shahab, Atif and Skancke, Jorgen and Suzuki, Ana Maria and Takahashi, Hazuki and Tilgner, Hagen and Trout, Diane and Walters, Nathalie and Wang, Huaien and Wrobel, John and Yu, Yanbao and Ruan, Xiaoan and Hayashizaki, Yoshihide and Harrow, Jennifer and Gerstein, Mark and Hubbard, Tim and Reymond, Alexandre and Antonarakis, Stylianos E and Hannon, Gregory and Giddings, Morgan C and Ruan, Yijun and Wold, Barbara and Carninci, Piero and Guigó, Roderic and Gingeras, Thomas R
Nature (London), ISSN 1476-4687, 2012, Volume 489, Issue 7414, pp. 101 - 108
Journal Article
Journal of cell science, ISSN 1477-9137, 2016, Volume 129, Issue 7, pp. 1287 - 1292
RAS proteins (KRAS4A, KRAS4B, NRAS and HRAS) function as GDP-GTP-regulated binary on-off switches, which regulate cytoplasmic signaling networks that control... 
PI3K | Ral | Rac | GTPase | Oncogene | Raf | Neoplasms - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Codon, Nonsense - genetics | Mutation, Missense - genetics | Protein Isoforms - genetics | Cell Science at A Glance
Journal Article
The EMBO Journal, ISSN 0261-4189, 02/2011, Volume 30, Issue 4, pp. 783 - 795
.... During the EMT process, TGF‐β induced isoform switching of fibroblast growth factor (FGF) receptors, causing the cells to become sensitive to FGF‐2. Addition of FGF‐2 to TGF‐β... 
FGF‐2 | TGF‐β | δEF1 | FGF receptor | EMT | FGF-2 | TGF-β | CELLS | dEF1 | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCTION | IDENTIFICATION | SUPPRESSION | FIBROBLASTS | CELL BIOLOGY | PROSTATE ADENOCARCINOMA | COREPRESSORS | TGF-beta | TUMOR-GROWTH | PROGRESSION | Neoplasms - metabolism | Protein Binding - genetics | Myofibroblasts - physiology | Epithelial-Mesenchymal Transition - physiology | Homeodomain Proteins - metabolism | Humans | Actins - metabolism | Fibroblast Growth Factor 2 - pharmacology | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Actins - genetics | DNA-Binding Proteins - metabolism | Myofibroblasts - metabolism | Cell Differentiation - genetics | Protein Isoforms - metabolism | Neoplasms - genetics | Protein Binding - drug effects | Receptors, Fibroblast Growth Factor - genetics | Fibroblast Growth Factor 2 - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Cell Differentiation - physiology | Neoplasm Invasiveness | Alternative Splicing - genetics | Cells, Cultured | Transforming Growth Factor beta - physiology | Alcohol Oxidoreductases - metabolism | Signal Transduction - genetics | Myofibroblasts - drug effects | Protein Isoforms - physiology | Transcription Factors - metabolism | Transforming Growth Factor beta - pharmacology | Alternative Splicing - drug effects | Cell Differentiation - drug effects | Models, Biological | Receptors, Fibroblast Growth Factor - metabolism | Neoplasms - pathology | Protein Isoforms - genetics | Zinc Finger E-box-Binding Homeobox 1
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 484, Issue 7394, pp. 333 - 338
The prevalence of obesity and type 2 diabetes is increasing worldwide and threatens to shorten lifespan. Impaired insulin action in peripheral tissues is a... 
DIABETES-MELLITUS | FATTY-ACID SYNTHESIS | GENE | INSULIN-RESISTANCE | LIPOGENESIS | MULTIDISCIPLINARY SCIENCES | CARBOHYDRATE-RESPONSE ELEMENT | LIVER | BINDING-PROTEIN CHREBP | UCSC GENOME BROWSER | TRANSGENIC MICE | Transcription Factors - chemistry | Diabetes Mellitus - genetics | Humans | Male | RNA, Messenger - metabolism | Adipose Tissue - metabolism | Protein Isoforms - chemistry | Nuclear Proteins - deficiency | Lipogenesis | Body Mass Index | Glucose Transporter Type 4 - genetics | Adipose Tissue - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - chemistry | Genotype | Glucose - pharmacology | Nuclear Proteins - chemistry | Mice, Knockout | Insulin - metabolism | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Insulin Resistance - genetics | Glucose - metabolism | Mice | Blood Glucose - metabolism | Homeostasis - genetics | Cohort Studies | Diabetes Mellitus - blood | Body Weight | Glucose Transporter Type 4 - metabolism | Transcription Factors - deficiency | Adipose Tissue - cytology | Molecular Sequence Data | Obesity - genetics | Promoter Regions, Genetic - genetics | Protein Isoforms - metabolism | Adiposity | Female | Nuclear Proteins - genetics | Insulin - pharmacology | Glucose Intolerance - genetics | Cross-Sectional Studies | Gene Expression Regulation - genetics | RNA, Messenger - genetics | Cells, Cultured | Diabetes Mellitus - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Obesity - metabolism | Transcription Factors - metabolism | Animals | Glucose Transporter Type 4 - biosynthesis | Adipocytes - metabolism | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Protein Isoforms - genetics
Journal Article