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Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 2575 - 2575
Journal Article
Hypertension, ISSN 0194-911X, 09/2009, Volume 54, Issue 3, pp. 598 - 604
Apelin receptors, present on vascular smooth muscle cells, endothelium, and cardiomyocytes, are activated by the family of apelin peptides to elicit... 
Human apelin receptor APJ | Vasodilatation | Positive cardiac inotropic agents | Vasoconstriction | Apelin | apelin | ENDOGENOUS PEPTIDE LIGAND | human apelin receptor APJ | NATURAL LIGAND | VASCULAR SMOOTH-MUSCLE | vasodilatation | IMMUNOCYTOCHEMICAL LOCALIZATION | TISSUE DISTRIBUTION | ENDOTHELIAL-CELLS | IN-VIVO | IMMUNODEFICIENCY-VIRUS TYPE-1 | PERIPHERAL VASCULAR DISEASE | vasoconstriction | ORPHAN RECEPTOR APJ | CARDIAC CONTRACTILITY | positive cardiac inotropic agents | Cardiotonic Agents - metabolism | Humans | Culture Media, Conditioned - chemistry | Male | Chromatography, High Pressure Liquid | Coronary Artery Disease - blood | Intercellular Signaling Peptides and Proteins - blood | Dose-Response Relationship, Drug | Protein Isoforms - blood | Intercellular Signaling Peptides and Proteins - metabolism | Protein Isoforms - metabolism | Spectrometry, Mass, Electrospray Ionization | Coronary Artery Disease - physiopathology | Myocardium - metabolism | Radioimmunoassay | Female | Mammary Arteries - drug effects | Endothelial Cells - metabolism | Coronary Artery Disease - metabolism | Cells, Cultured | Endothelium, Vascular - physiopathology | Myocardium - pathology | Saphenous Vein - physiopathology | Cardiotonic Agents - pharmacology | Protein Isoforms - pharmacology | Cardiotonic Agents - blood | Endothelial Cells - cytology | Intercellular Signaling Peptides and Proteins - pharmacology | Mammary Arteries - physiopathology | Aged | Vasodilation - drug effects | In Vitro Techniques | Saphenous Vein - drug effects | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e78330 - e78330
Background: Myocardial fibrosis is a key process in diabetic cardiomyopathy. However, their underlying mechanisms have not been elucidated, leading to a lack... 
HEART | CELLS | ACTIVATION | DPP-4 INHIBITION | GLUCAGON-LIKE PEPTIDE-1 | MYOCARDIAL-INFARCTION | RAT | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | RECEPTOR | IMPROVES | Diabetic Cardiomyopathies - metabolism | Metformin - therapeutic use | Diabetic Cardiomyopathies - drug therapy | Insulin - physiology | Fibroblasts - physiology | Cardiomegaly - etiology | Glucagon-Like Peptide 1 - physiology | Male | Pyrazines - therapeutic use | Diabetes Mellitus, Type 2 - metabolism | Cardiotonic Agents - therapeutic use | Glucose Intolerance - drug therapy | Sitagliptin Phosphate | Drug Evaluation, Preclinical | Diabetes Mellitus, Type 2 - complications | Disease Models, Animal | Triazoles - therapeutic use | Hypoglycemic Agents - therapeutic use | Metformin - pharmacology | Cardiomegaly - drug therapy | Cells, Cultured | Glucagon-Like Peptide 1 - pharmacology | Rats | Myocardium - pathology | Cardiotonic Agents - pharmacology | Fibronectins - metabolism | Protein Isoforms - physiology | Hypoglycemic Agents - pharmacology | Protein Isoforms - pharmacology | Triazoles - pharmacology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - physiology | Fibrosis | PPAR delta - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Pyrazines - pharmacology | Apoptosis | Cardiomegaly - metabolism | Hyperglycemia | Glucagon | Heart enlargement | Glucose | Insulin | Dextrose | Diabetes therapy | Heart | Plasma | Heart attacks | Peptides | Cardiomyopathy | Deceleration | Cardiovascular disease | Kinases | Palmitic acid | Eutrophication | Proteins | Rodents | Fibroblasts | Extracellular matrix | Heart diseases | Glucagon-like peptide 1 | Heart failure | Cell survival | Hyperlipidemia | Diabetes mellitus | Cardiomyocytes | Metabolism | Signaling | Cell death | Isoforms | Metformin | Diabetes | Laboratory animals | Hypertrophy | Structure-function relationships | Index Medicus
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 04/2006, Volume 79, Issue 4, pp. 803 - 808
Journal Article
Clinical Immunology, ISSN 1521-6616, 2012, Volume 145, Issue 3, pp. 189 - 200
Journal Article
Cells Tissues Organs, ISSN 1422-6405, 08/2010, Volume 192, Issue 3, pp. 158 - 166
Induction of chondrogenesis in mesenchymal stem cells (MSCs) with TGF-β leads to a hypertrophic phenotype. The hypertrophic maturation of the chondrocytes is... 
Original Paper | Bone marrow stromal cell | Cartilage repair | Chondrogenic differentiation | Tissue engineering | Mesenchymal progenitor cell differentiation | Cell biology | Cartilage biology | Hypertrophy | Mesenchymal stem cells | PROGENITOR CELLS | ENDOCHONDRAL OSSIFICATION | ANATOMY & MORPHOLOGY | BONE-MARROW | CHONDROCYTE MATURATION | DEVELOPMENTAL BIOLOGY | CULTURE | MOLECULAR EVENTS | GROWTH | GENE-EXPRESSION | DIFFERENTIATION | VITRO CARTILAGE FORMATION | Chondrocytes - cytology | Chondrogenesis - drug effects | Humans | Alkaline Phosphatase - metabolism | Extracellular Matrix - metabolism | Male | Collagen Type II - metabolism | Young Adult | Mesenchymal Stromal Cells - cytology | Cell Enlargement - drug effects | Collagen Type X - metabolism | Adult | Female | Glycerophosphates - pharmacology | Cell Differentiation - physiology | Chondrocytes - metabolism | Transforming Growth Factor beta1 - pharmacology | Transforming Growth Factor beta3 - pharmacology | Mesenchymal Stromal Cells - drug effects | Collagen Type I - metabolism | Calcification, Physiologic - drug effects | Glycosaminoglycans - metabolism | Mesenchymal Stromal Cells - metabolism | DNA - metabolism | Protein Isoforms - pharmacology | Chondrogenesis - physiology | Transforming Growth Factor beta - pharmacology | Cell Differentiation - drug effects | Culture Media, Conditioned - metabolism | Index Medicus
Journal Article
Cell, ISSN 0092-8674, 09/2016, Volume 167, Issue 1, pp. 275 - 284.e6
The VEGF-A isoforms play a crucial role in vascular development, and the VEGF signaling pathway is a clinically validated therapeutic target for several... 
endothelium | VEGF | angiogenesis | readthrough translation | tyrosine kinase | ISOFORMS | NEUROPILIN-1 | SPLICE VARIANT | BIOCHEMISTRY & MOLECULAR BIOLOGY | VEGFB | RECEPTOR | EXTRACELLULAR-MATRIX | TUMOR-CELLS | MECHANISMS | BINDING | ENDOTHELIAL GROWTH-FACTOR | CELL BIOLOGY | Capillary Permeability - physiology | Neovascularization, Physiologic - drug effects | Protein Biosynthesis | Alternative Splicing | Mitogens - pharmacology | Neuropilin-1 - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Capillary Permeability - genetics | Protein Isoforms - metabolism | Cloning, Molecular | HEK293 Cells | Angiogenesis Inducing Agents - pharmacology | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Recombinant Proteins - metabolism | Amino Acid Sequence | Guinea Pigs | Neovascularization, Physiologic - genetics | RNA, Messenger - genetics | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Recombinant Proteins - genetics | Chemotaxis - drug effects | Recombinant Proteins - pharmacology | Protein Isoforms - pharmacology | Neovascularization, Physiologic - physiology | Tyrosine - metabolism | Animals | Mitosis - drug effects | Mitosis - physiology | Endothelial Cells - cytology | Mice | Endothelial Cells - drug effects | Protein Isoforms - genetics | Tyrosine | Permeability | RNA | Vascular endothelial growth factor | Analysis | Endothelium | Index Medicus
Journal Article
Journal Article
Journal Article