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Chinese Medical Journal, ISSN 0366-6999, 06/2017, Volume 130, Issue 11, pp. 1333 - 1341
Journal Article
中华医学杂志:英文版, ISSN 0366-6999, 2016, Volume 129, Issue 10, pp. 1154 - 1160
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2012, Volume 188, Issue 5, pp. 2127 - 2135
It is estimated that 1 billion people around the world are vitamin D deficient. Vitamin D deficiency has been linked to various inflammatory diseases. However,... 
ACTIVATION | GENE | INFLAMMATION | MACROPHAGES | DISEASE | NUCLEAR RECEPTOR SUPERFAMILY | TNF-ALPHA | IMMUNOLOGY | DIFFERENTIAL REGULATION | CANCER | EXPRESSION | Dual Specificity Phosphatase 1 - deficiency | Bone Marrow Cells - enzymology | Humans | L Cells (Cell Line) | Male | Monocytes - immunology | Lipopolysaccharides - antagonists & inhibitors | Mice, 129 Strain | MAP Kinase Signaling System - immunology | Phosphorylation - genetics | MAP Kinase Signaling System - genetics | Bone Marrow Cells - immunology | Inflammation Mediators - metabolism | Monocytes - pathology | Adult | Bone Marrow Cells - drug effects | Inflammation Mediators - antagonists & inhibitors | Phosphorylation - immunology | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Macrophages - immunology | Dual Specificity Phosphatase 1 - genetics | Macrophages - pathology | Mice, Inbred C57BL | Cells, Cultured | Vitamin D - pharmacology | Macrophages - enzymology | Mice, Knockout | Monocytes - drug effects | Monocytes - enzymology | Animals | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Dual Specificity Phosphatase 1 - metabolism | Macrophages - drug effects | Mice | Cytokines - antagonists & inhibitors | Drug Delivery Systems - methods | Cytokines - biosynthesis
Journal Article
BMC Genomics, ISSN 1471-2164, 02/2013, Volume 14, Issue 1, pp. 104 - 104
Journal Article
Journal Article
Cellular Signalling, ISSN 0898-6568, 2012, Volume 24, Issue 2, pp. 560 - 568
The dual-specificity mitogen-activated protein kinase (MAPK) phosphatase-1 (MKP-1) inactivates MAP kinases by dephosphorylation. Here we show that the... 
Cardiac fibrosis | Signal transduction | Cytokines | Interleukins | Migration | Proliferation | Myocardial remodeling | CELLS | ACTIVATED PROTEIN-KINASE | MACROPHAGES | P38 MAPK | BETA | CELL BIOLOGY | NF-KAPPA-B | EXPRESSION | Dual Specificity Phosphatase 1 - deficiency | Dual Specificity Phosphatase 1 - antagonists & inhibitors | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Receptors, Interleukin-17 - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Fibrosis - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Cell Movement - genetics | Proto-Oncogene Proteins c-akt - genetics | Ventricular Remodeling | Transfection | Myocardium - metabolism | Transcription, Genetic | p38 Mitogen-Activated Protein Kinases - metabolism | Receptors, Interleukin-17 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Fibroblasts - metabolism | Dual Specificity Phosphatase 1 - genetics | Fibrosis - genetics | Gene Expression | Enzyme Inhibitors - pharmacology | Genetic Vectors - metabolism | Interleukin-17 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Myocardium - pathology | Genetic Vectors - genetics | Phosphatidylinositol 3-Kinases - genetics | Cell Movement - drug effects | Interleukin-17 - metabolism | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Proliferation - drug effects | Fibroblasts - cytology | Mice | MicroRNAs - genetics | Fibrosis - pathology | Phosphatases | proliferation | cardiac fibrosis | signal transduction | migration | interleukins | myocardial remodeling
Journal Article
Journal Article
Cellular Signalling, ISSN 0898-6568, 06/2017, Volume 34, pp. 1 - 10
Journal Article
Circulation Research, ISSN 0009-7330, 01/2013, Volume 112, Issue 1, pp. 48 - 56
RATIONALE:Mitogen-activated protein kinases (MAPKs) are activated in the heart by disease-inducing and stress-inducing stimuli, where they participate in... 
SIGNALING PATHWAYS | CARDIAC & CARDIOVASCULAR SYSTEMS | signal transduction | PHOSPHORYLATION | HEART-FAILURE | CARDIAC MYOCYTES | genetically altered mice | HYPERTROPHY | INHIBITION | mitogen-activated protein kinases | PROTEIN-KINASE PHOSPHATASE-1 | MKP-1 | IN-VIVO | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | dilated cardiomyopathy | Dual Specificity Phosphatase 1 - deficiency | Fibroblasts - enzymology | Phosphorylation | Calcium - metabolism | Cardiomyopathies - prevention & control | Mice, 129 Strain | Cardiomyopathies - enzymology | Myocytes, Cardiac - enzymology | Cardiomyopathies - genetics | Cardiomyopathies - physiopathology | Time Factors | Cardiomyopathies - diagnosis | p38 Mitogen-Activated Protein Kinases - metabolism | Disease Models, Animal | Myocardial Contraction | Dual Specificity Phosphatase 1 - genetics | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Calcium-Binding Proteins - deficiency | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mice | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Hemodynamics | Protein Tyrosine Phosphatases | Calcium-Binding Proteins - genetics | signaling | Other myocardial biology | Gene regulation | MAPK | DUSP | Animal models of human disease | Dilated cardiomyopathy | Heart failure - basic studies | Physiological and pathological control of gene expression | Congestive | Other heart failure | Hypertrophy | Mitogen-activated protein kinases
Journal Article
Journal Article