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Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 10/2018, Volume 38, Issue 10, pp. 1715 - 1726
The exact physiological function of amyloid-β precursor protein (APP) in endothelial cells is unknown. Endothelium-specific APP-deficient (eAPP−/−) mice were... 
cerebral arteries | amyloid-β | endothelium | endothelial nitric oxide synthase | Amyloid precursor protein | CELLS | NITRIC-OXIDE SYNTHASE | HPH-1 MICE | HUMAN PLATELETS | RECEPTOR | BETA-PEPTIDE | NEUROSCIENCES | GTP-CYCLOHYDROLASE I | GUANYLYL CYCLASE | amyloid | DISEASE | ENDOCRINOLOGY & METABOLISM | HEMATOLOGY | EXPRESSION | Original
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 02/2008, Volume 104, Issue 3, pp. 683 - 695
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 10/2012, Volume 199, Issue 2, pp. 381 - 399
Journal Article
Oncogene, ISSN 0950-9232, 04/2014, Volume 33, Issue 17, pp. 2264 - 2272
Journal Article
Neuron, ISSN 0896-6273, 2007, Volume 56, Issue 1, pp. 66 - 78
Mutations in the amyloid precursor protein (APP) cause early-onset Alzheimer's disease (AD), but the only genetic risk factor for late-onset AD is the ɛ4... 
CELLBIO | MOLNEURO | SIGNALING | BETA-SECRETASE | DEGRADING ENZYME NEPRILYSIN | INTRAMEMBRANE PROTEOLYSIS | ALZHEIMERS-DISEASE | GENE-EXPRESSION | CENTRAL-NERVOUS-SYSTEM | DEPENDENT TRANSCRIPTIONAL CONTROL | SYNAPTIC PLASTICITY | NEUROSCIENCES | INTRACELLULAR DOMAINS | MICE LACKING | Enzyme-Linked Immunosorbent Assay - methods | Humans | Gene Expression Regulation, Developmental - genetics | Nuclear Proteins - pharmacology | Apolipoproteins E - metabolism | Brain - metabolism | RNA, Messenger - biosynthesis | Transfection - methods | Chromatin Immunoprecipitation | Tumor Suppressor Proteins - deficiency | Reverse Transcriptase Polymerase Chain Reaction - methods | Nerve Tissue Proteins - pharmacology | Receptors, LDL - deficiency | Lysine Acetyltransferase 5 | Amyloid beta-Protein Precursor - physiology | Gene Expression Regulation, Developmental - physiology | Animals, Newborn | Amyloid beta-Protein Precursor - deficiency | Cricetinae | Histone Acetyltransferases - pharmacology | Trans-Activators | Cholesterol - metabolism | Mice, Knockout | Tumor Suppressor Proteins - physiology | Amyloid Precursor Protein Secretases - metabolism | Animals | Cytidine Deaminase - metabolism | Mice | Receptors, LDL - physiology | Cell Line, Transformed | Proteins | Brain | Lipoproteins | Pathogenesis | Neurons | Rodents | Alzheimers disease | Metabolism | Apolipoproteins | Cholesterol | Deoxyribonucleic acid--DNA
Journal Article
Nature, ISSN 0028-0836, 03/2017, Volume 543, Issue 7646, pp. 507 - 512
Maternally inherited 15q11-13 chromosomal triplications cause a frequent and highly penetrant type of autism linked to increased gene dosages of UBE3A, which... 
ENERGY-BALANCE | EXCITATORY NEURONS | UBIQUITIN-PROTEIN LIGASE | SPECTRUM DISORDER | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | COPY NUMBER | ANGELMAN-SYNDROME | SYNAPSE FORMATION | E6-ASSOCIATED PROTEIN | GABAERGIC NEURONS | Autism | Genetic aspects | Seizures (Medicine) | Gene expression | Health aspects | Brain | Convulsions & seizures | Social skills | Neurons | Genes
Journal Article