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PLoS Pathogens, ISSN 1553-7366, 2015, Volume 11, Issue 9, pp. e1005142 - e1005142
Pharmacologically-induced activation of replication competent proviruses from latency in the presence of antiretroviral treatment (ART) has been proposed as a... 
ACTIVATION | RNA | PERSISTENCE | CD4(+) T-CELLS | MICROBIOLOGY | VIROLOGY | HISTONE DEACETYLASES | ACTIVE ANTIRETROVIRAL THERAPY | IMMUNODEFICIENCY-VIRUS TYPE-1 | STEM-CELL | INFECTION | PARASITOLOGY | VIRAL RESERVOIR | Follow-Up Studies | Humans | Middle Aged | Antiretroviral Therapy, Highly Active - adverse effects | Male | RNA, Viral - blood | AIDS Vaccines - therapeutic use | Virus Latency - drug effects | AIDS Vaccines - adverse effects | Anti-HIV Agents - administration & dosage | HIV Infections - immunology | Drug Interactions | HIV-1 - physiology | Lymphocytes - immunology | Protein Processing, Post-Translational - drug effects | HIV-1 - isolation & purification | Adult | Anti-HIV Agents - therapeutic use | Female | Depsipeptides - therapeutic use | RNA, Viral - metabolism | Depsipeptides - adverse effects | Biomarkers - metabolism | Lymphocytes - metabolism | HIV-1 - drug effects | Anti-HIV Agents - adverse effects | HIV Infections - virology | Biomarkers - blood | Histones - blood | Acetylation - drug effects | HIV-1 - immunology | Virus Activation - drug effects | Lymphocytes - drug effects | HIV Infections - drug therapy | Histones - metabolism | Infusions, Intravenous | Viral Load - drug effects | Cohort Studies | Depsipeptides - administration & dosage | HIV Infections - metabolism | Index Medicus | Studies | Plasma | HIV | Acquired immune deficiency syndrome | Cytokines | Lymphocytes | Curing | AIDS | Infections | Human immunodeficiency virus | Research | Drug therapy
Journal Article
Nature, ISSN 0028-0836, 10/2015, Volume 526, Issue 7575, pp. 666 - 671
Journal Article
Journal Article
Nature, ISSN 0028-0836, 11/2011, Volume 479, Issue 7373, pp. 419 - 422
Journal Article
Plant and Cell Physiology, ISSN 0032-0781, 04/2015, Volume 56, Issue 12, pp. 2297 - 2311
Post-translational modification mechanisms function as switches that mediate the balance between optimum growth and the response to environmental stimuli, by... 
2-Deoxyglucose | SUMO | Glucose | SIZ1 | Post-germination growth arrest | Arabidopsis | PROTEIN | STRESS-RESPONSE | PLANT GLUCOSE | SUMOYLATION | PLANT SCIENCES | CELL BIOLOGY | BIOLOGICAL PROCESSES | INSENSITIVE MUTANTS | E3 LIGASE SIZ1 | ETHYLENE | ABSCISIC-ACID | TRANSCRIPTION FACTOR | Carbohydrate Metabolism - drug effects | Molecular Weight | Arabidopsis - growth & development | Genes, Plant | Ligases - genetics | Protein Processing, Post-Translational - genetics | Starch - metabolism | Carbohydrate Metabolism - genetics | RNA, Messenger - metabolism | Plant Roots - drug effects | Arabidopsis Proteins - metabolism | Protein Processing, Post-Translational - drug effects | Germination - genetics | Sucrose - metabolism | Plant Roots - growth & development | Protein-Serine-Threonine Kinases - metabolism | Germination - drug effects | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Carbohydrates - pharmacology | Small Ubiquitin-Related Modifier Proteins - metabolism | RNA, Messenger - genetics | Ligases - metabolism | Glucose - pharmacology | Signal Transduction - genetics | Mutation - genetics | Arabidopsis - metabolism | Arabidopsis - genetics | Gene Expression Regulation, Plant - drug effects | Phenotype | Signal Transduction - drug effects | Models, Biological | Plant Roots - anatomy & histology | Sumoylation | Hexokinase - metabolism | Index Medicus
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 385, pp. 75 - 86
Abstract The chaperone Hsp60 is pro-carcinogenic in certain tumor types by interfering with apoptosis and with tumor cell death. In these tumors, it is not yet... 
Hematology, Oncology and Palliative Medicine | Ubiquitination | Hsp60 | Acetylation | Replicative senescence | Doxorubicin | p53 | APOPTOSIS | OXIDATIVE STRESS | MOLECULAR CHAPERONES | DNA-DAMAGE | CELLULAR SENESCENCE | CANCER | HEAT-SHOCK PROTEINS | THERAPY | ONCOLOGY | MITOCHONDRIAL CHAPERONES | EXPRESSION | Lung Neoplasms - drug therapy | Antibiotics, Antineoplastic - pharmacology | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Cellular Senescence - drug effects | Lung Neoplasms - pathology | Mitochondrial Proteins - genetics | Carcinoma, Mucoepidermoid - metabolism | Dose-Response Relationship, Drug | Carcinoma, Mucoepidermoid - genetics | G2 Phase Cell Cycle Checkpoints - drug effects | Protein Processing, Post-Translational - drug effects | Mitochondrial Proteins - metabolism | Proteolysis | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Chaperonin 60 - metabolism | Lung Neoplasms - genetics | Cell Survival - drug effects | Chaperonin 60 - genetics | Tumor Suppressor Protein p53 - metabolism | Carcinoma, Mucoepidermoid - drug therapy | Carcinoma, Mucoepidermoid - pathology | Chaperonins - metabolism | Signal Transduction - drug effects | Cell Line, Tumor | Protein Binding | Cell Proliferation - drug effects | Histones - metabolism | Doxorubicin - pharmacology | Medical colleges | Neurosciences | Anthracyclines | Microbiology | Cell death | Heat shock proteins | Development and progression | Tumor proteins | Cancer | Proteins | Cell culture | Chemotherapy | Senescence | Cell cycle | Fibroblasts | Cardiomyocytes | Breast cancer | Drug dosages | Cancer therapies | Apoptosis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 03/2011, Volume 471, Issue 7336, pp. 74 - 79
Protein acetylation is mediated by histone acetyltransferases (HATs) and deacetylases (HDACs), which influence chromatin dynamics, protein turnover and the DNA... 
YEAST | REPAIR | ACTIVATION | ACETYLATION | MULTIDISCIPLINARY SCIENCES | VALPROIC ACID | CHECKPOINT | HOMOLOGOUS RECOMBINATION | END RESECTION | HISTONE DEACETYLASE INHIBITORS | SACCHAROMYCES-CEREVISIAE | Autophagy-Related Proteins | Protein Kinases - genetics | Microtubule-Associated Proteins - metabolism | Saccharomyces cerevisiae - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Endonucleases - metabolism | Valproic Acid - pharmacology | Autophagy - drug effects | DNA Breaks, Double-Stranded - drug effects | Protein Processing, Post-Translational - drug effects | Histone Acetyltransferases - metabolism | Endodeoxyribonucleases - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Aminopeptidases - metabolism | Chromosomal Instability | Protein-Serine-Threonine Kinases - metabolism | DNA Repair - drug effects | Histone Deacetylases - genetics | Saccharomyces cerevisiae Proteins - antagonists & inhibitors | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Histone Deacetylases - metabolism | Saccharomyces cerevisiae Proteins - genetics | Endonucleases - chemistry | Saccharomyces cerevisiae - cytology | Autophagy-Related Protein 8 Family | Acetylation - drug effects | Signal Transduction - drug effects | Exodeoxyribonucleases - metabolism | Saccharomyces cerevisiae Proteins - metabolism | Saccharomyces cerevisiae - enzymology | Histone Deacetylase Inhibitors - pharmacology | Saccharomyces cerevisiae Proteins - chemistry | Proteins | Physiological aspects | Causes of | Enzymes | Genetic aspects | DNA damage | Phosphorylation | Yeast | Kinases | DNA repair | Autophagy | Experiments | Recruitment | Cell cycle | Genetics | Influence | Apoptosis | Index Medicus
Journal Article
Journal Article