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Nature Genetics, ISSN 1061-4036, 07/2016, Volume 48, Issue 7, pp. 747 - 757
Genome-wide association studies have identified several loci associated with pancreatic cancer risk; however, the mechanisms by which genetic factors influence... 
U-BOX | REGULATORY RNA | RETINOIC ACID | LONG NONCODING RNA | GENETICS & HEREDITY | SQUAMOUS-CELL CARCINOMA | GENE-EXPRESSION | SUSCEPTIBILITY LOCI | UBIQUITIN LIGASES | IDENTIFY MULTIPLE | GENOME-WIDE ASSOCIATION | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - metabolism | Cell Proliferation | Pancreatic Neoplasms - metabolism | Humans | DNA Repair Enzymes - genetics | Gene Expression Regulation, Neoplastic | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Case-Control Studies | RNA Splicing Factors - metabolism | STAT1 Transcription Factor - metabolism | Ubiquitination | Proteolysis | DNA Repair Enzymes - metabolism | src-Family Kinases - metabolism | Nuclear Proteins - genetics | Binding Sites | Genome-Wide Association Study | Signal Transduction | Pancreatic Neoplasms - pathology | Nuclear Proteins - metabolism | Pancreatic Neoplasms - genetics | RNA, Long Noncoding - genetics | Pancreas - metabolism | RNA Splicing Factors - genetics | STAT1 Transcription Factor - genetics | Polymorphism, Single Nucleotide - genetics | MicroRNAs - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - genetics | src-Family Kinases - genetics | MicroRNA | Phosphatases | Genetic variation | Pancreatic cancer | Development and progression | Genetic aspects | Properties | Health aspects | Proteins | Studies | Confidence intervals | Research & development--R&D | Genomes | Mutation | Gene expression | Phosphatase | Cancer | Index Medicus
Journal Article
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 05/2017, Volume 486, Issue 3, pp. 759 - 766
Multiple sclerosis (MS) is an immune-mediated demyelinating disease of the central nervous system, and its pathogenesis remains largely unclear. Much attention... 
Multiple sclerosis | IL-1β | miR-448 | Th17 | PTPN2 | BIOPHYSICS | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUROFILAMENT LIGHT | IL-1 beta | CYTOKINE | DIFFERENTIATION | CANCER | Multiple Sclerosis - diagnosis | CD8-Positive T-Lymphocytes - pathology | Luciferases - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 2 - immunology | Humans | Interleukin-17 - immunology | NF-kappa B - immunology | Interleukin-1beta - genetics | Case-Control Studies | Luciferases - genetics | Cell Differentiation | B-Lymphocytes - pathology | Genes, Reporter | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Neutrophils - pathology | Th17 Cells - pathology | Severity of Illness Index | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Signal Transduction | Gene Expression Regulation | Neutrophils - immunology | Interleukin-17 - genetics | Interleukin-1beta - immunology | Multiple Sclerosis - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 2 - genetics | MicroRNAs - immunology | Remission Induction | B-Lymphocytes - immunology | NF-kappa B - genetics | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Th17 Cells - immunology | MicroRNAs - genetics | Primary Cell Culture | CD8-Positive T-Lymphocytes - immunology | Tyrosine | Medical colleges | Phosphatases | MicroRNA | Interleukins | Phenols | T cells
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2018, Volume 13, Issue 6, p. e0198652
The incidence of type-1 Diabetes Mellitus (T1DM) has increased steadily in Kuwait during recent years and it is now considered amongst the high-incidence... 
DISEASE RISK | CELL FUNCTION | AUTOIMMUNITY | ISLET AUTOANTIBODIES | JAPANESE POPULATION | MULTIDISCIPLINARY SCIENCES | GENERATION SEQUENCING REVEALS | PROMOTER POLYMORPHISM | PTPN22 GENE | GENOME-WIDE ASSOCIATION | CHILDREN | Antibody Specificity | Genes, MHC Class II | Genetic Predisposition to Disease | Autoantibodies - blood | Humans | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - genetics | Child, Preschool | Diabetes Mellitus, Type 1 - genetics | Genotype | Infant | Male | HLA-DR Antigens - genetics | Kuwait - epidemiology | HLA-DQ Antigens - genetics | Adolescent | Alleles | Arabs - genetics | Female | Polymorphism, Single Nucleotide | Diabetes Mellitus, Type 1 - immunology | Gain of Function Mutation | Diabetes Mellitus, Type 1 - ethnology | Child | Infant, Newborn | Autoantibodies | Analysis | Diabetes in children | Genetic aspects | Disease susceptibility | Research | Prevalence studies (Epidemiology) | Haplotypes | Pediatrics | Genes | Arabs | Phosphatase | Incidence | Gene sequencing | Proteins | Medical laboratories | Direct reduction | Children | Genotypes | Deoxyribonucleic acid--DNA | Immune system | Tyrosine | Nucleotide sequence | Diabetes mellitus | Environmental factors | Patients | Insulin | Polymerase chain reaction | Studies | Hospitals | Restriction fragment length polymorphism | Diabetes | Immunoassays | DNA sequencing | Protein-tyrosine-phosphatase | Antigenic determinants | Deoxyribonucleic acid | DNA
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2018, Volume 24, Issue 7, pp. 954 - 960
The ubiquitously expressed non-receptor protein tyrosine phosphatase SHP2, encoded by PTPN11, is involved in signal transduction downstream of multiple growth... 
MEDICINE, RESEARCH & EXPERIMENTAL | PANCREATIC DUCTAL ADENOCARCINOMA | TYROSINE PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | COLON-CANCER | SHP2 | INHIBITION | K-RAS | CELL LUNG-CARCINOMA | MOUSE MODEL | PROGRESSION | PHOSPHOTYROSINE PHOSPHATASE | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - metabolism | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Pancreatic Neoplasms - pathology | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Lung Neoplasms - pathology | Mutation - genetics | Carcinogenesis - metabolism | Disease Progression | Carcinogenesis - pathology | Mitogen-Activated Protein Kinase Kinases - metabolism | Animals | Protein Kinase Inhibitors - therapeutic use | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - deficiency | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Tyrosine | Care and treatment | Cellular signal transduction | Genetic aspects | Research | Gene expression | Cancer | Adenocarcinoma | Animal models | Xenotransplantation | Lung cancer | Phosphatase | Carcinogenesis | K-Ras protein | Proteins | Signal transduction | Carcinogens | Allosteric properties | Clonal deletion | Organoids | Dependence | Xenografts | Deletion | Protein-tyrosine kinase receptors | Inhibition | Pancreas | Protein-tyrosine kinase | MAP kinase | Tumor cell lines | Signaling | Transduction | Tumors | Protein-tyrosine-phosphatase
Journal Article