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Kidney International, ISSN 0085-2538, 2014, Volume 85, Issue 2, pp. 457 - 470
Renal toxicity constitutes a dose-limiting side effect of anticancer therapies targeting vascular endothelial growth factor (VEGF). In order to study this... 
anti-VEGF | FSGS | NF-κB | c-mip | thrombotic microangiopathy | MCNS | ANGIOGENESIS | PHOSPHORYLATION | PROTEINURIA | PODOCYTES | CANCER | CELL CARCINOMA | DAMAGE | RENAL THROMBOTIC MICROANGIOPATHY | NF-kappa B | AUTOCRINE | UROLOGY & NEPHROLOGY | NF-KAPPA-B | Glomerulosclerosis, Focal Segmental - enzymology | Predictive Value of Tests | Humans | Middle Aged | Male | Protein Kinase Inhibitors - adverse effects | Young Adult | Proteinuria - chemically induced | Renal Insufficiency - chemically induced | Base Sequence | Kidney Diseases - chemically induced | Glomerulosclerosis, Focal Segmental - chemically induced | Transcription, Genetic | Hypertension - enzymology | Angiogenesis Inhibitors - adverse effects | Binding Sites | Biomarkers - metabolism | Niacinamide - adverse effects | Kidney Glomerulus - pathology | Thrombotic Microangiopathies - enzymology | Mice, Knockout | Glomerulosclerosis, Focal Segmental - diagnosis | Adaptor Proteins, Signal Transducing | Nephrosis, Lipoid - chemically induced | Thrombotic Microangiopathies - diagnosis | Mice | Transcription Factor RelA - deficiency | Niacinamide - analogs & derivatives | Renal Insufficiency - enzymology | Renal Insufficiency - diagnosis | Vascular Endothelial Growth Factors - antagonists & inhibitors | Kidney Diseases - diagnosis | Molecular Sequence Data | Nephrosis, Lipoid - enzymology | Kidney Diseases - enzymology | Transcription Factor RelA - genetics | Case-Control Studies | Transfection | Phenylurea Compounds - adverse effects | Hypertension - chemically induced | Adult | Female | Proteinuria - diagnosis | Hypertension - diagnosis | Kidney Glomerulus - enzymology | Cell Line | Promoter Regions, Genetic | Nephrosis, Lipoid - diagnosis | Kidney Glomerulus - drug effects | Gene Expression Regulation | Carrier Proteins - genetics | Proteinuria - enzymology | Animals | Carrier Proteins - metabolism | Transcription Factor RelA - metabolism | Vascular Endothelial Growth Factors - metabolism | Aged | Thrombotic Microangiopathies - chemically induced | Life Sciences | Biochemistry, Molecular Biology
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2010, Volume 299, Issue 6, pp. H1891 - H1901
Sahan-Firat S, Jennings BL, Yaghini FA, Song CY, Estes AM, Fang XR, Farjana N, Khan AI, Malik KU. 2,3',4,5'-Tetramethoxystilbene prevents... 
Cytochrome P-450 1B1 activity | Endothelial dysfunction | Reactive oxygen species | Renal function | Vascular reactivity | ARACHIDONIC-ACID RELEASE | CYTOSOLIC PHOSPHOLIPASE A | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ACTIVATED PROTEIN-KINASE | SUPEROXIDE-PRODUCTION | NITRIC-OXIDE SYNTHASE | vascular reactivity | ANGIOTENSIN-II | endothelial dysfunction | VASCULAR SMOOTH-MUSCLE | II-INDUCED HYPERTENSION | cytochrome P-450 1B1 activity | reactive oxygen species | renal function | PERIPHERAL VASCULAR DISEASE | Antihypertensive Agents - pharmacology | Kidney - pathology | Kidney - enzymology | Endothelium, Vascular - drug effects | Male | Stilbenes - pharmacology | Proteinuria - prevention & control | Time Factors | p38 Mitogen-Activated Protein Kinases - metabolism | Hypertension - enzymology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Kidney - drug effects | Enzyme Inhibitors - pharmacology | Cardiomegaly - physiopathology | Rats | Cardiomegaly - enzymology | Sodium Chloride, Dietary | Rats, Sprague-Dawley | Vasoconstriction - drug effects | Hypertension - pathology | Proteinuria - physiopathology | Cardiomegaly - prevention & control | Mitogen-Activated Protein Kinase 3 - metabolism | Cytochrome P-450 CYP1B1 | Vasodilation - drug effects | Muscle, Smooth, Vascular - enzymology | Mitogen-Activated Protein Kinase 1 - metabolism | Endothelium, Vascular - enzymology | Muscle, Smooth, Vascular - physiopathology | Hypertension - etiology | Superoxides - metabolism | Hypertension - prevention & control | Blood Pressure - drug effects | NADH, NADPH Oxidoreductases - metabolism | Aorta - enzymology | Kidney - physiopathology | Aryl Hydrocarbon Hydroxylases - antagonists & inhibitors | Aorta - drug effects | Desoxycorticosterone | Myocardium - pathology | Hypertension - physiopathology | NADPH Oxidase 1 | Aryl Hydrocarbon Hydroxylases - metabolism | Hydroxyeicosatetraenoic Acids - blood | Proteinuria - enzymology | Diuresis - drug effects | Myocardium - enzymology | Animals | 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid - blood | Hypertension | Prevention | Cytochrome P-450 | Physiological aspects | Development and progression | Health aspects
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 6, pp. 1991 - 2002
Inflammation plays a key role in the development and progression of diabetic kidney disease; however, the role of the anti-inflammatory molecule netrin-1 in... 
Pathology | ISCHEMIA-REPERFUSION INJURY | CELLS | ACTIVATION | MONOCYTE CHEMOATTRACTANT | INHIBITION | INSULIN-RESISTANCE | PROTECTS KIDNEY | PROTEINURIA | PATHOLOGY | UNC5B RECEPTOR | NEPHROPATHY | Organ Specificity - drug effects | Albuminuria - complications | Inflammation - pathology | Leukocytes - pathology | Gene Expression - drug effects | Apoptosis - drug effects | Diabetes Mellitus, Experimental - enzymology | Dinoprostone - biosynthesis | Netrin-1 | Albuminuria - blood | Epithelial Cells - drug effects | Hyperglycemia - complications | NF-kappa B - metabolism | Nerve Growth Factors - metabolism | Inflammation - blood | Mesangial Cells - drug effects | Necrosis | Diabetes Mellitus, Experimental - blood | Inflammation - complications | Hyperglycemia - pathology | Diabetes Mellitus, Experimental - complications | Nerve Growth Factors - blood | Kidney Tubules, Proximal - enzymology | Albuminuria - pathology | Kidney Tubules, Proximal - pathology | Tumor Suppressor Proteins - metabolism | Transgenes - genetics | Cytokines - secretion | Epithelial Cells - pathology | Mice, Transgenic | Glucose - pharmacology | Mesangial Cells - metabolism | Albuminuria - enzymology | Cell Movement - drug effects | Animals | Tumor Suppressor Proteins - blood | Hyperglycemia - blood | Chickens | Cyclooxygenase 2 - metabolism | Epithelial Cells - enzymology | Mesangial Cells - pathology | Diabetes Mellitus, Experimental - pathology | Hyperglycemia - enzymology | Leukocytes - drug effects | Mice | Inflammation - enzymology | Kidney Tubules, Proximal - drug effects | Regular
Journal Article
Kidney International, ISSN 0085-2538, 01/2018, Volume 93, Issue 1, pp. 110 - 127
Journal Article
Kidney International, ISSN 0085-2538, 09/2012, Volume 82, Issue 5, pp. 509 - 511
Using conditional knockout models, Zhou firmly establish a renoprotective role of β-catenin in acute kidney injury. Although β-catenin is protective at the... 
UROLOGY & NEPHROLOGY | CELLS | RENAL INTERSTITIAL FIBROSIS | PROGRESSION | Glomerulosclerosis, Focal Segmental - enzymology | Proteinuria - enzymology | Animals | Class I Phosphatidylinositol 3-Kinases - metabolism | Signal Transduction | Kidney - enzymology | Male | Smad3 Protein - metabolism | Doxorubicin
Journal Article
Kidney International, ISSN 0085-2538, 11/2013, Volume 84, Issue 5, pp. 920 - 930
Podocytes are highly specialized epithelial cells with complex actin cytoskeletal architecture crucial for maintenance of the glomerular filtration barrier.... 
glomerular filtration barrier | glomerular disease | cytoskeleton | transgenic mouse | podocyte | NEPHRIN | PROTEINURIA | DIRECT TARGET | CELL BIOLOGY | NEURONAL DEVELOPMENT | ACTIN REORGANIZATION | DISEASE | UROLOGY & NEPHROLOGY | KIDNEY PODOCYTES | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | Renal Insufficiency - enzymology | Phosphorylation | Podocytes - enzymology | rac1 GTP-Binding Protein - deficiency | Desoxycorticosterone Acetate | Male | cdc42 GTP-Binding Protein - metabolism | Acute Kidney Injury - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Mice, 129 Strain | Renal Insufficiency - etiology | Actin Depolymerizing Factors - metabolism | Nephrectomy | Time Factors | Cell Shape | Hypertension - chemically induced | Acute Kidney Injury - chemically induced | Albuminuria - metabolism | Membrane Proteins - metabolism | Hypertension - enzymology | Hypertension - genetics | Neuropeptides - genetics | Disease Models, Animal | Protamines | Neuropeptides - deficiency | Renal Insufficiency - genetics | Signal Transduction | Acute Kidney Injury - pathology | Mice, Inbred C57BL | Genotype | Neuropeptides - metabolism | Podocytes - pathology | Acute Kidney Injury - enzymology | Mice, Knockout | Phenotype | Animals | Renal Insufficiency - pathology | cdc42 GTP-Binding Protein - genetics | Mice | rac1 GTP-Binding Protein - metabolism | cdc42 GTP-Binding Protein - deficiency | rac1 GTP-Binding Protein - genetics
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2018, Volume 128, Issue 8, pp. 3445 - 3459
Podocyte malfunction occurs in autoimmune and nonautoimmune kidney disease. Calcium signaling is essential for podocyte injury, but the role of... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | LUPUS | DEPENDENT KINASE IV | PROTEIN-KINASE | RHO GTPASES | SYNAPTOPODIN | ACTIN DYNAMICS | RAC1 | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | CELL BIOLOGY | Glomerulosclerosis, Focal Segmental - enzymology | Kidney Glomerulus - enzymology | Lupus Nephritis - pathology | Proteinuria - immunology | Signal Transduction | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - metabolism | Humans | Male | Lupus Nephritis - immunology | Kidney Glomerulus - pathology | Proteinuria - pathology | Mice, Knockout | Lupus Nephritis - enzymology | Proteinuria - enzymology | Mice, Inbred MRL lpr | Animals | Glomerulosclerosis, Focal Segmental - immunology | Female | Mice | Mice, Inbred BALB C | Kidney Glomerulus - immunology | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - immunology | Cell Line, Transformed | Glomerulosclerosis, Focal Segmental - pathology | Care and treatment | Nephritis | Analysis | Development and progression | Research | Kidney diseases | Autoimmune diseases | Guanosine triphosphatase | Lipopolysaccharides | Lupus | Phosphorylation | Kidneys | Motility | RhoA protein | Rac1 protein | Lupus nephritis | Kinases | Calcium signalling | Proteins | Actin | Ultrastructure | Biopsy | Calcium-binding protein | Rodents | Ca2+/calmodulin-dependent protein kinase | Trends | Calmodulin | Proteinuria | Structure-function relationships
Journal Article
Journal Article
Journal of the American Society of Nephrology : JASN, ISSN 1046-6673, 09/2014, Volume 25, Issue 9, pp. 1966 - 1978
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 2016, Volume 27, Issue 12, pp. 3545 - 3551
Journal Article