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1973, ISBN 0443009554, viii, 807
Book
Journal Article
Journal Article
American Journal of Transplantation, ISSN 1600-6135, 11/2009, Volume 9, Issue 11, pp. 2532 - 2541
Journal Article
Kidney International, ISSN 0085-2538, 06/2006, Volume 69, Issue 12, pp. 2131 - 2147
The terminally differentiated podocyte, also called glomerular visceral epithelial cell, are highly specialized cells. They function as a critical size and... 
glomerular epithelial cell | apoptosis | nephrin | proliferation | cell cycle | podocyte | Podocyte | Nephrin | Glomerular epithelial cell | Proliferation | Cell cycle | Apoptosis | FOOT PROCESS EFFACEMENT | GLOMERULAR EPITHELIAL-CELL | PASSIVE HEYMANN NEPHRITIS | PUROMYCIN AMINONUCLEOSIDE NEPHROSIS | EXPERIMENTAL MEMBRANOUS NEPHROPATHY | EXPERIMENTAL DIABETIC-NEPHROPATHY | CYCLE REGULATORY PROTEINS | HIV-ASSOCIATED NEPHROPATHY | UROLOGY & NEPHROLOGY | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | ENDOTHELIAL GROWTH-FACTOR | Cell Proliferation | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Cell Count | Humans | Diabetic Nephropathies - drug therapy | Proteinuria - pathology | Adrenal Cortex Hormones - therapeutic use | Hypertrophy - pathology | Angiotensin II Type 1 Receptor Blockers - pharmacology | Hypertrophy - physiopathology | Diabetic Nephropathies - physiopathology | Glomerular Basement Membrane - pathology | Glomerulosclerosis, Focal Segmental - drug therapy | Glomerular Basement Membrane - physiopathology | Glomerular Basement Membrane - ultrastructure | Glomerulosclerosis, Focal Segmental - physiopathology | Tretinoin - pharmacology | Endothelium - pathology | Diabetic Nephropathies - pathology | Cytoskeleton - pathology | Adrenal Cortex Hormones - pharmacology | Cytoskeleton - ultrastructure | Podocytes - pathology | Endothelium - physiopathology | Proteinuria - physiopathology | Proteinuria - drug therapy | Tretinoin - therapeutic use | Podocytes - drug effects | Podocytes - physiology | Cell Adhesion - physiology | Cell Cycle - physiology | Cytoskeleton - physiology | Apoptosis - physiology | DNA Damage | Glomerulosclerosis, Focal Segmental - pathology
Journal Article
Acta Gastro-Enterologica Belgica, ISSN 1784-3227, 07/2018, Volume 81, Issue 3, pp. 381 - 385
Background and study aim : Renal complications are frequent extraintestinal manifestations in inflammatory bowel disease (IBD). We aimed in our study to... 
Amyloidosis | IBD | IgA nephropathy | Renal complications | GLOMERULONEPHRITIS | CROHNS-DISEASE | ULCERATIVE-COLITIS | BIOPSY | IMMUNOGLOBULIN-A NEPHROPATHY | COMPLICATIONS | GASTROENTEROLOGY & HEPATOLOGY | ASSOCIATION | MANIFESTATIONS | Humans | Middle Aged | Nephrosis, Lipoid - epidemiology | Inflammatory Bowel Diseases - physiopathology | Male | Glomerulonephritis, Membranous - epidemiology | Proteinuria - pathology | Egypt - epidemiology | Thrombotic Microangiopathies - epidemiology | Renal Insufficiency, Chronic - epidemiology | Kidney Cortex Necrosis - epidemiology | Nephritis, Interstitial - metabolism | Amyloidosis - epidemiology | Glomerulosclerosis, Focal Segmental - etiology | Kidney Diseases - epidemiology | Amyloidosis - etiology | Nephrosis, Lipoid - etiology | Inflammatory Bowel Diseases - complications | Acute Disease | Amyloidosis - pathology | Kidney Diseases - pathology | Nephrotic Syndrome - pathology | Nephrosis, Lipoid - pathology | Glomerulonephritis - pathology | Nephritis, Interstitial - etiology | Nephrotic Syndrome - epidemiology | Glomerulonephritis, IGA - metabolism | Thrombotic Microangiopathies - metabolism | Kidney Diseases - etiology | Renal Insufficiency, Chronic - etiology | Nephrotic Syndrome - metabolism | Glomerulonephritis - metabolism | Glomerulonephritis, Membranous - metabolism | Nephritis, Interstitial - epidemiology | Renal Insufficiency, Chronic - metabolism | Antibodies, Antineutrophil Cytoplasmic - metabolism | Inflammatory Bowel Diseases - epidemiology | Nephritis, Interstitial - pathology | Kidney Cortex Necrosis - pathology | Adult | Female | Glomerulonephritis, IGA - etiology | Retrospective Studies | Glomerulonephritis - etiology | Glomerulonephritis, Membranous - etiology | Glomerulonephritis, IGA - epidemiology | Glomerulonephritis - epidemiology | Glomerulonephritis, IGA - pathology | Kidney Diseases - metabolism | Nephrosis, Lipoid - metabolism | Proteinuria - etiology | Proteinuria - epidemiology | Thrombotic Microangiopathies - pathology | Glomerulosclerosis, Focal Segmental - epidemiology | Glomerulonephritis, Membranous - pathology | Proteinuria - metabolism | Glomerulosclerosis, Focal Segmental - metabolism | Renal Insufficiency, Chronic - pathology | Kidney Cortex Necrosis - etiology | Biopsy | Kidney Cortex Necrosis - metabolism | Thrombotic Microangiopathies - etiology | Amyloidosis - metabolism | Glomerulosclerosis, Focal Segmental - pathology | Nephrotic Syndrome - etiology
Journal Article
European journal of human genetics : EJHG, ISSN 1018-4813, 09/2015, Volume 23, Issue 9, pp. 1254 - 1258
Coenzyme Q10 deficiency is a clinically and genetically heterogeneous disorder, with manifestations that may range from fatal neonatal multisystem failure, to... 
Mitochondrial Diseases - pathology | Muscle Weakness - diagnosis | Acidosis, Lactic - genetics | Humans | Ubiquinone - deficiency | Muscle Weakness - blood | Proteinuria - pathology | Muscle Weakness - genetics | Intellectual Disability - genetics | Proteinuria - blood | Intellectual Disability - blood | Alkyl and Aryl Transferases - genetics | Ubiquinone - blood | Fatal Outcome | Muscle Weakness - pathology | Acidosis, Lactic - blood | Ataxia - blood | Female | Ataxia - genetics | Alkyl and Aryl Transferases - deficiency | Infant, Newborn | Ataxia - pathology | Mitochondria, Muscle - genetics | Mitochondrial Diseases - genetics | Gene Expression | Muscle, Skeletal - enzymology | Mitochondria, Muscle - enzymology | Mitochondria, Muscle - pathology | Ubiquinone - analogs & derivatives | Intellectual Disability - pathology | Ubiquinone - genetics | Proteinuria - genetics | Acidosis, Lactic - pathology | Ataxia - diagnosis | Hepatic Insufficiency - blood | Sequence Analysis, DNA | Renal Aminoacidurias - pathology | Point Mutation | Hepatic Insufficiency - pathology | Hepatic Insufficiency - genetics | Consanguinity | Renal Aminoacidurias - genetics | Muscle, Skeletal - pathology | Mitochondrial Diseases - blood | Mitochondrial Diseases - diagnosis | Renal Aminoacidurias - blood | Neonates | Cell culture | Intensive care | Laboratories | Genomics | Liver | Childrens health | Aciduria | Ventricles (cerebral) | Biosynthesis | Genetic screening | Electron transport chain | Mitochondria | Coenzyme Q10 | Encephalopathy | Fibroblasts | Genetics | Complementation | Supplementation | Diagnosis | Lactic acidosis | Echocardiography | Electron microscopy | Metabolism | Patients | Pregnancy | Cristae | Mutation | Ventricle | Acidosis | Proteinuria | Life Sciences | Human health and pathology | Short Report
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 6, pp. 1991 - 2002
Inflammation plays a key role in the development and progression of diabetic kidney disease; however, the role of the anti-inflammatory molecule netrin-1 in... 
Pathology | ISCHEMIA-REPERFUSION INJURY | CELLS | ACTIVATION | MONOCYTE CHEMOATTRACTANT | INHIBITION | INSULIN-RESISTANCE | PROTECTS KIDNEY | PROTEINURIA | PATHOLOGY | UNC5B RECEPTOR | NEPHROPATHY | Organ Specificity - drug effects | Albuminuria - complications | Inflammation - pathology | Leukocytes - pathology | Gene Expression - drug effects | Apoptosis - drug effects | Diabetes Mellitus, Experimental - enzymology | Dinoprostone - biosynthesis | Netrin-1 | Albuminuria - blood | Epithelial Cells - drug effects | Hyperglycemia - complications | NF-kappa B - metabolism | Nerve Growth Factors - metabolism | Inflammation - blood | Mesangial Cells - drug effects | Necrosis | Diabetes Mellitus, Experimental - blood | Inflammation - complications | Hyperglycemia - pathology | Diabetes Mellitus, Experimental - complications | Nerve Growth Factors - blood | Kidney Tubules, Proximal - enzymology | Albuminuria - pathology | Kidney Tubules, Proximal - pathology | Tumor Suppressor Proteins - metabolism | Transgenes - genetics | Cytokines - secretion | Epithelial Cells - pathology | Mice, Transgenic | Glucose - pharmacology | Mesangial Cells - metabolism | Albuminuria - enzymology | Cell Movement - drug effects | Animals | Tumor Suppressor Proteins - blood | Hyperglycemia - blood | Chickens | Cyclooxygenase 2 - metabolism | Epithelial Cells - enzymology | Mesangial Cells - pathology | Diabetes Mellitus, Experimental - pathology | Hyperglycemia - enzymology | Leukocytes - drug effects | Mice | Inflammation - enzymology | Kidney Tubules, Proximal - drug effects | Regular
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 10/2013, Volume 210, Issue 11, pp. 2387 - 2401
Journal Article
Journal Article