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世界胃肠病学杂志:英文版, ISSN 1007-9327, 2015, Volume 21, Issue 37, pp. 10573 - 10583
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 2014, Volume 111, Issue 3, pp. 518 - 530
Apart from transporting lipids through the body, the human plasma lipoproteins very low-density lipoprotein (VLDL) and low-density lipoprotein (LDL) are also... 
CETP | proteomics | atherothrombosis | LCAT | protein S | Apolipoproteins | coagulation | prenylcysteine oxidase | prothrombin | anti-microbial peptides | cathepcin D | nLC-MS/MS | dermicidin | TFPI-1 | lysozyme C | CD14 | PLTP | lipopolysaccharide binding protein | apoAV | Cardiovascular Biology and Cell Signalling | Prenylcysteine oxidase | Coagulation | Atherothrombosis | Cathepcin D | Lysozyme C | Lipopolysaccharide binding protein | Dermicidin | Antimicrobial peptides | Proteomics | Prothrombin | Protein S | ApoAV | PATTERNS | MASS-SPECTROMETRY | PARAOXONASE-1 | PERIPHERAL VASCULAR DISEASE | DYSLIPIDEMIA | HEMATOLOGY | OXIDATION | apolipoproteins | PROTHROMBINASE | 2-DIMENSIONAL GEL-ELECTROPHORESIS | REVERSE CHOLESTEROL TRANSPORT | ENHANCEMENT | CHYLOMICRONS | Apolipoproteins A - metabolism | Humans | Antimicrobial Cationic Peptides - metabolism | Lipopolysaccharide Receptors - metabolism | Phospholipid Transfer Proteins - metabolism | Protein S - metabolism | Dyslipidemias - blood | Plasma - metabolism | Mass Spectrometry | Blood Coagulation | Lipoproteins - metabolism | Lipoproteins, LDL - metabolism | Prothrombin - metabolism | Apolipoprotein A-V | Computational Biology | Lipid Metabolism | Muramidase - metabolism | Cathepsin D - metabolism | Carbon-Sulfur Lyases - metabolism | Atherosclerosis - blood | Lipoproteins, VLDL - metabolism | Blood Coagulation Disorders - blood | Cholesterol Ester Transfer Proteins - metabolism | Proteome - metabolism | Phosphatidylcholine-Sterol O-Acyltransferase - metabolism
Journal Article
BMC Nephrology, ISSN 1471-2369, 01/2017, Volume 18, Issue 1, p. 35
Background: During chronic kidney disease progression, kidney-specific risk factors for cardiovascular disease come into play. The present study investigated... 
Prothrombotic state | Hemostatic disorder | Indoxyl sulfate | von Willebrand factor | Tissue factor | Tryptophan derivatives | Uremic toxin | Cardiovascular disease | Chronic kidney disease | Monocytes activation | MORTALITY | KAPPA-B ACTIVATION | OXIDATIVE STRESS | RISK-FACTORS | KYNURENINE PATHWAY | ARYL-HYDROCARBON RECEPTOR | INFLAMMATION | ENDOTHELIAL-CELLS | UROLOGY & NEPHROLOGY | CARDIORENAL SYNDROME | EXPRESSION | Prevalence | C-Reactive Protein | Oxidative Stress | Tissue Plasminogen Activator - metabolism | Humans | Middle Aged | Male | Case-Control Studies | Renal Insufficiency, Chronic - epidemiology | Renal Insufficiency, Chronic - metabolism | Plasminogen Activator Inhibitor 1 - metabolism | Hemostasis | Lipoproteins - metabolism | Cardiovascular Diseases - epidemiology | Adult | Female | Prothrombin - metabolism | von Willebrand Factor - metabolism | Peptide Fragments - metabolism | Cardiovascular Diseases - metabolism | Inflammation | Hydrogen Peroxide - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Urokinase-Type Plasminogen Activator - metabolism | Indican - metabolism | Aged | Thrombin - metabolism | Thromboplastin - metabolism | Neopterin - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Physiological aspects | Toxins | Chronic kidney failure | Cardiovascular diseases | Health aspects
Journal Article
世界胃肠病学杂志:英文版, ISSN 1007-9327, 2016, Volume 22, Issue 37, pp. 8271 - 8282
Chronic hepatitis B virus(HBV) infected patients have an almost 100-fold increased risk to develop hepatocellular carcinoma(HCC). HCC is the fifth most common... 
Hepatocellular | B | carcinoma;Hepatitis | NA | virus | infection;Biomarkers;Predictive;Diagnostic;Alphafetoprotein;Validation;Limitations;MicroR | Validation | MicroRNA | Alphafetoprotein | Predictive | Biomarkers | Diagnostic | Hepatocellular carcinoma | Limitations | Hepatitis B virus infection | SERUM INTERLEUKIN-6 | SURFACE-ANTIGEN LEVELS | GAMMA-CARBOXY PROTHROMBIN | OLIGOMERIC MATRIX PROTEIN | Alpha-fetoprotein | ABNORMAL PROTHROMBIN | PROGNOSTIC-SIGNIFICANCE | GASTROENTEROLOGY & HEPATOLOGY | VIRAL-HEPATITIS | REACTIVE ALPHA-FETOPROTEIN | CLINICAL-SIGNIFICANCE | DISEASE PROGRESSION | Predictive Value of Tests | Hepatitis B - metabolism | Humans | Liver Neoplasms - complications | Osteopontin - metabolism | Glypicans - metabolism | Hepatitis B - complications | Intercellular Signaling Peptides and Proteins - metabolism | Carcinoma, Hepatocellular - complications | Ultrasonography | Antigens, Neoplasm - metabolism | Membrane Proteins - metabolism | Hepatitis B virus | Prothrombin - metabolism | alpha-Fetoproteins - metabolism | Biomarkers - metabolism | Enzyme-Linked Immunosorbent Assay | Serpins - metabolism | Carcinoma, Hepatocellular - diagnosis | Protein Precursors - metabolism | Biomarkers, Tumor - blood | Hepatitis B - diagnosis | Liver Neoplasms - diagnosis | Liver Neoplasms - metabolism | Carcinoma, Hepatocellular - metabolism | Review
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, p. e27574
Chronic pancreatitis is a chronic inflammatory disorder of the pancreas. The etiology is multi-fold, but all lead to progressive scarring and loss of... 
CELLS | SPLENIC VEIN-THROMBOSIS | BIOMARKER DISCOVERY | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | CANCER-TISSUE | EXTRACELLULAR-MATRIX | TANDEM MASS-SPECTROMETRY | PROTEINS | PARAFFIN-EMBEDDED TISSUES | STATISTICAL-MODEL | Immunohistochemistry | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Humans | Glycoproteins - metabolism | Adenocarcinoma - metabolism | Lumican | Proteomics - methods | Pancreatic Stellate Cells - pathology | Extracellular Matrix Proteins - metabolism | Acinar Cells - metabolism | Reproducibility of Results | Signal Transduction | Pancreatic Neoplasms - pathology | Chondroitin Sulfate Proteoglycans - metabolism | Keratan Sulfate - metabolism | Pancreas - pathology | Pancreas - metabolism | Pancreatitis, Chronic - pathology | Collagen - metabolism | Acinar Cells - pathology | Proteins - metabolism | Biological Specimen Banks | Staining and Labeling | Versicans - metabolism | Pancreatitis, Chronic - metabolism | Cluster Analysis | Pancreatic Stellate Cells - metabolism | Proteins | Medical research | Pancreatic cancer | Pancreatitis | Medicine, Experimental | Development and progression | Comparative analysis | Formaldehyde | Adenocarcinoma | Pathogenesis | Paraffin | Tissues | Cell activation | Epidermal growth factor | Pathways | Etiology | Extracellular matrix | Lesions | Pancreas | Versican | Ethanol | Glycoproteins | Metabolism | Disease control | Thrombosis | Biological activity | Medicine | Scars | Collagen | Proteomics | Fibrosis | Prothrombin | Functional groups | Cancer
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 04/2016, Volume 777, pp. 1 - 8
Ferulic acid (FA) produces protective effects against cardiovascular dysfunctions. However, the mechanisms of FA is still not known. Here we examined the... 
Antiplatelet | Antithrombotic | Coagulation | Ferulic acid | ESTER | CELLS | OXIDATIVE STRESS | ACTIVATION | PLATELET-AGGREGATION | KINASE | EXPERIMENTAL THROMBOSIS | IN-VITRO | INHIBITION | PHARMACOLOGY & PHARMACY | VASP PHOSPHORYLATION | Calcium - metabolism | Male | Phosphoproteins - metabolism | Superoxides - metabolism | Microfilament Proteins - metabolism | Phosphorylation - drug effects | Cyclic AMP - metabolism | Thrombosis - physiopathology | Intracellular Space - drug effects | Phosphoric Diester Hydrolases - metabolism | Hemorrhage - complications | Rats | Thromboxane B2 - biosynthesis | Cell Adhesion Molecules - metabolism | Fibrinolytic Agents - pharmacology | Thrombosis - pathology | Thrombosis - metabolism | Animals | Blood Coagulation - drug effects | Cyclic GMP - metabolism | Signal Transduction - drug effects | Intracellular Space - metabolism | Mice | Thrombosis - drug therapy | Nitric Oxide - metabolism | Coumaric Acids - pharmacology | Mitogen-Activated Protein Kinases - metabolism | Behavioral health care | Medical examination | Vasodilators | Thrombin | Cyclic adenylic acid | Anticoagulants (Medicine) | Arachidonic acid | Cyclic guanylic acid | Thrombosis | Blood clot | Blood | Hydrolysis | Analysis | Collagen | Nitric oxide | Physiological aspects | Prothrombin | Adenylic acid | Mitogens | Protein kinases | Adenosine triphosphatase
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, p. e74562
Clear cell carcinoma (CCC) is a histologically distinct carcinoma subtype that arises in several organ systems and is marked by cytoplasmic clearing,... 
SURVIVAL | VENOUS THROMBOEMBOLISM | SEROUS CARCINOMA | POOR-PROGNOSIS | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER RISK | OVARIAN CARCINOMAS | PROTHROMBIN GENE | MUTATIONS | EXPRESSION | HEPATOCYTE NUCLEAR FACTOR-1-BETA | Immunohistochemistry | Glucose-6-Phosphatase - genetics | Kidney Neoplasms - genetics | Oligonucleotide Array Sequence Analysis | Humans | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Carcinoma, Renal Cell - genetics | Risk | Kidney Neoplasms - metabolism | Ovarian Neoplasms - complications | Venous Thrombosis - complications | Ovarian Neoplasms - genetics | DNA Methylation | Glycogen - metabolism | Endometrial Neoplasms - genetics | Blood Coagulation | Endometrial Neoplasms - complications | Female | Ovarian Neoplasms - metabolism | Kidney Neoplasms - complications | Hepatocyte Nuclear Factor 1-beta - genetics | Hepatocyte Nuclear Factor 1-beta - metabolism | Adenocarcinoma, Clear Cell - metabolism | Carcinoma, Renal Cell - pathology | Venous Thrombosis - metabolism | Computational Biology | Blood Coagulation Factors - genetics | Blood Coagulation Factors - metabolism | Glucose-6-Phosphatase - metabolism | Carcinoma, Renal Cell - metabolism | Adenocarcinoma, Clear Cell - complications | Endometrial Neoplasms - pathology | Kidney Neoplasms - pathology | Adenocarcinoma, Clear Cell - pathology | Venous Thrombosis - genetics | Carcinoma, Renal Cell - complications | Venous Thrombosis - pathology | Adenocarcinoma, Clear Cell - genetics | Type 2 diabetes | Carcinoma | Glycogen | Genes | Glucose | Blood coagulation factors | Thrombosis | Dextrose | Blood clot | Glucose metabolism | Fibrin | DNA microarrays | Analysis | Physiological aspects | Cancer | Blood coagulation | Cancer therapies | Clotting | Ovarian cancer | Fibrinogen | DNA methylation | Bioindicators | Thromboembolism | Bioinformatics | Hepatocyte nuclear factor 1 | Endometrium | Deoxyribonucleic acid--DNA | Clearing | Cloning | Histology | Metabolism | Patients | Coagulation factors | Studies | Pathology | Medical prognosis | Prothrombin | Biomarkers | Diabetes | Mutation | Binding sites | Clear cell-type renal cell carcinoma | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0158388
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 05/2010, Volume 88, Issue 7, pp. 1537 - 1548
We have shown that prothrombin kringle‐2 (pKr‐2), a domain of human prothrombin distinct from thrombin could activate cultured rat brain microglia in vitro.... 
prothrombin kringle‐2 | substantia nigra | microglia | neuroin‐flammation | dopaminergic neurons | Dopaminergic neurons | Neuroinflammation | Prothrombin kringle-2 | Substantia nigra | Microglia | NADPH OXIDASE | OXIDATIVE STRESS | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | prothrombin kringle-2 | NEUROSCIENCES | MESSENGER-RNA | neuroinflammation | CAPILLARY ENDOTHELIAL-CELLS | RAT-BRAIN | INDUCED NEUROTOXICITY | PARKINSONS-DISEASE | Substantia Nigra - physiopathology | Microglia - metabolism | MAP Kinase Signaling System - physiology | Coculture Techniques | Gliosis - chemically induced | Nitric Oxide Synthase - antagonists & inhibitors | Gliosis - physiopathology | Prothrombin - chemistry | Substantia Nigra - metabolism | RNA, Messenger - metabolism | CD11b Antigen - analysis | Cyclooxygenase 2 - genetics | Nitric Oxide Synthase Type II - antagonists & inhibitors | Inflammation Mediators - metabolism | Female | Neurons - metabolism | Parkinson Disease - metabolism | Neurons - drug effects | Prothrombin - metabolism | Dopamine - metabolism | RNA, Messenger - drug effects | Cyclooxygenase 2 Inhibitors - pharmacology | Microglia - drug effects | Gliosis - metabolism | Cells, Cultured | Prothrombin - toxicity | Rats | Cyclooxygenase 2 - drug effects | Parkinson Disease - physiopathology | Rats, Sprague-Dawley | Substantia Nigra - drug effects | Animals | MAP Kinase Signaling System - drug effects | Nitric Oxide Synthase Type II - genetics | Cyclooxygenase 2 - metabolism | Nitric Oxide Synthase - metabolism | CD11b Antigen - metabolism | Kringles - physiology | Nitric Oxide Synthase Type II - metabolism | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2017, Volume 37, Issue 3, pp. e22 - e32
OBJECTIVE—The drug warfarin blocks carboxylation of vitamin K–dependent proteins and acts as an anticoagulant and an accelerant of vascular calcification. The... 
anticoagulants | myocytes, smooth muscle | exosomes | prothrombin | vascular calcification | FETUIN-MINERAL COMPLEX | CALCIPROTEIN PARTICLES | MATRIX GLA PROTEIN | CARDIOVASCULAR CALCIFICATION | EXTRACELLULAR CALCIUM | smooth muscle | POTENTIAL MECHANISM | myocytes | CHEMICAL SYNTHESIS | HUMAN ATHEROSCLEROTIC PLAQUES | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | TISSUE FACTOR | Exosomes - drug effects | Exosomes - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Middle Aged | Myocytes, Smooth Muscle - pathology | Warfarin - adverse effects | Male | Endosomes - metabolism | Vascular Calcification - metabolism | Endocytosis | Female | Protein Interaction Domains and Motifs | Myocytes, Smooth Muscle - drug effects | Prothrombin - metabolism | Myocytes, Smooth Muscle - metabolism | Extracellular Matrix Proteins - metabolism | Muscle, Smooth, Vascular - drug effects | Calcium-Binding Proteins - metabolism | Signal Transduction | Phosphatidylserines - metabolism | Cells, Cultured | Vascular Calcification - prevention & control | Anticoagulants - adverse effects | Vascular Calcification - pathology | Peptides - pharmacology | Protein Transport | Muscle, Smooth, Vascular - pathology | Blood Coagulation - drug effects | Vascular Calcification - chemically induced | Protein Binding | Aged
Journal Article