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Molecular Cell, ISSN 1097-2765, 06/2015, Volume 58, Issue 6, pp. 1028 - 1039
The bromodomain and extraterminal (BET) protein BRD4 is a validated drug target in leukemia, yet its regulatory function in this disease is not well... 
SELECTIVE-INHIBITION | RECRUITMENT | C/EBP-ALPHA | CREB-BINDING PROTEIN | CHROMATIN | ACETYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | C-MYC | P-TEFB | BRD4 | CELL BIOLOGY | Transcriptional Regulator ERG | NIH 3T3 Cells | Oncogene Proteins - genetics | Humans | Leukemia, Myeloid - genetics | Proto-Oncogene Proteins c-myb - genetics | Gene Expression Profiling | Leukemia, Myeloid - pathology | RNA Interference | Proto-Oncogene Protein c-fli-1 - metabolism | Protein Binding - drug effects | Trans-Activators - genetics | Nuclear Proteins - genetics | Proto-Oncogene Proteins - metabolism | Acute Disease | Proto-Oncogene Protein c-fli-1 - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Oncogene Proteins - metabolism | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Transcription Factors - antagonists & inhibitors | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | CCAAT-Enhancer-Binding Protein-beta - metabolism | Proto-Oncogene Proteins c-myb - metabolism | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Acetylation - drug effects | Animals | Hematopoietic System - metabolism | Leukemia, Myeloid - metabolism | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Trans-Activators - metabolism | Mice | Histones - metabolism | DNA binding proteins | Medicine, Experimental | Medical research | Chromatin | Lysine
Journal Article
Cell, ISSN 0092-8674, 09/2017, Volume 171, Issue 1, pp. 163 - 178.e19
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 04/2016, Volume 7, Issue 1, p. 11208
Journal Article
Genes and Development, ISSN 0890-9369, 05/2010, Volume 24, Issue 9, pp. 916 - 932
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 6, pp. 2610 - 2615
Prevailing theories suggest that luminal cells are the origin of prostate cancer because it is histologically defined by basal cell loss and malignant luminal... 
Epithelial cells | Stromal cells | Stem cells | Basal cell neoplasms | Lesions | Magnification | Prostate | Prostate cancer | Cells | Tissue grafting | AKT | ERG | ETS | CD49F | PROGENITOR CELLS | TRANSFORMATION | ANDROGEN RECEPTOR | MURINE PROSTATE | MULTIDISCIPLINARY SCIENCES | PTEN | MODEL | CD49f | VENTRAL PROSTATE | SUBPOPULATION | EXPRESSION | Fibroblast Growth Factor 10 - pharmacology | Prostatic Neoplasms - metabolism | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Receptors, Androgen - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Prostatic Neoplasms - physiopathology | Stem Cells - metabolism | Proto-Oncogene Proteins c-akt - genetics | Flow Cytometry | Proto-Oncogene Protein c-fli-1 - metabolism | Female | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Prostatic Neoplasms - pathology | Proto-Oncogene Protein c-fli-1 - genetics | Signal Transduction | Mice, Inbred C57BL | Epithelial Cells - pathology | Mice, Transgenic | Mice, Inbred Strains | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Phosphatidylinositol 3-Kinases - genetics | Regeneration | Animals | Receptors, Androgen - genetics | Stem Cells - drug effects | Stem Cells - pathology | Mice | Development and progression | Genetic aspects | Risk factors | Biological Sciences
Journal Article
Oncogene, ISSN 0950-9232, 2013, Volume 32, Issue 42, pp. 5089 - 5100
Ewing sarcoma provides an important model for transcription-factor-mediated oncogenic transformation because of its reliance on the ETS-type fusion oncoprotein... 
Ewing sarcoma | Repression | Transcription | NuRD | EWS/FLI | PROTEIN-PROTEIN | TARGET | TRANSFORMATION | CELLS | DNA-BINDING | transcription | BIOCHEMISTRY & MOLECULAR BIOLOGY | PEDIATRIC SOLID TUMORS | FUSION GENE | CELL BIOLOGY | ONCOLOGY | RESPONSE ELEMENTS | GENETICS & HEREDITY | repression | EXPRESSION | REVEALS | Oncogene Proteins, Fusion - metabolism | Humans | Gene Expression Regulation, Neoplastic | Sarcoma, Ewing - pathology | Co-Repressor Proteins - metabolism | Mi-2 Nucleosome Remodeling and Deacetylase Complex - genetics | Mi-2 Nucleosome Remodeling and Deacetylase Complex - metabolism | Bone Neoplasms - pathology | Histone Demethylases - genetics | Cell Transformation, Neoplastic - genetics | Proto-Oncogene Protein c-fli-1 - metabolism | Transcription, Genetic | Bone Neoplasms - genetics | Genes, Tumor Suppressor | Protein Structure, Tertiary | Proto-Oncogene Protein c-fli-1 - genetics | RNA-Binding Protein EWS - genetics | Histone Deacetylases - metabolism | RNA-Binding Protein EWS - metabolism | Co-Repressor Proteins - genetics | Xenograft Model Antitumor Assays | Histone Demethylases - metabolism | Animals | Mice, Nude | Oncogene Proteins, Fusion - genetics | Protein-Lysine 6-Oxidase - metabolism | Cell Line, Tumor | Protein-Lysine 6-Oxidase - genetics | Sarcoma, Ewing - genetics | Genetic aspects | Genetic transcription | Research | Carcinogenesis | Ewing's sarcoma | Testing | Cancer | Proteins | Cellular biology | Gene expression | EWS | FLI
Journal Article
Cell Reports, ISSN 2211-1247, 02/2015, Volume 10, Issue 7, pp. 1082 - 1095
Journal Article
Nature, ISSN 0028-0836, 03/2018, Volume 555, Issue 7696, pp. 387 - 391
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2014, Volume 124, Issue 2, pp. 580 - 591
Journal Article
Cancer Research, ISSN 0008-5472, 12/2005, Volume 65, Issue 24, pp. 11459 - 11468
Ewing's sarcoma is a member of Ewing's family tumors (EFTs) and the second most common solid hone and soft tissue malignancy of children and young adults. It... 
TARGET | STEM-CELLS | DNA-BINDING | CDNA MICROARRAY | GENE | ONCOLOGY | MYC | EWS/ETS FUSIONS |