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Journal of Biological Chemistry, ISSN 0021-9258, 02/2015, Volume 290, Issue 8, pp. 5065 - 5077
Journal Article
Journal Article
Journal Article
The American Journal of Surgical Pathology, ISSN 0147-5185, 11/2014, Volume 38, Issue 11, pp. 1494 - 1500
Lynch syndrome (LS) is an autosomal dominant inherited disease that is associated with an increased risk for colorectal and endometrial cancer due to germline... 
Breast | Lynch | Secondary malignancy | Hereditary nonpolyposis colon cancer syndrome | Mismatch repair | SURGERY | hereditary nonpolyposis colon cancer syndrome | NONPOLYPOSIS COLORECTAL-CANCER | secondary malignancy | mismatch repair | GENE MUTATION | COLON | LYNCH SYNDROME | PATHOLOGY | CARRIERS | TUMORS | ENDOMETRIAL CANCER | STRATEGIES | SPECTRUM | breast | REGISTRY | Immunohistochemistry | MutL Protein Homolog 1 | Prospective Studies | Biomarkers, Tumor - deficiency | Colorectal Neoplasms, Hereditary Nonpolyposis - pathology | Humans | Middle Aged | Male | DNA Mismatch Repair - genetics | DNA-Binding Proteins - deficiency | Young Adult | Breast Neoplasms - chemistry | DNA Methylation | Neoplasms, Second Primary | DNA Mutational Analysis | MutS Homolog 2 Protein - deficiency | Nuclear Proteins - deficiency | Aged, 80 and over | Adult | Female | Mismatch Repair Endonuclease PMS2 | Retrospective Studies | DNA Repair Enzymes - deficiency | Adenosine Triphosphatases - deficiency | Colorectal Neoplasms, Hereditary Nonpolyposis - genetics | Risk Factors | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Proto-Oncogene Proteins B-raf - genetics | Adaptor Proteins, Signal Transducing - deficiency | Adolescent | Aged | Biomarkers, Tumor - genetics | Colorectal Neoplasms, Hereditary Nonpolyposis - chemistry | Mutation | Index Medicus
Journal Article
Science, ISSN 0036-8075, 12/2010, Volume 330, Issue 6009, pp. 1390 - 1393
Although the proteins BAX and BAK are required for initiation of apoptosis at the mitochondria, how BAX and BAK are activated remains unsettled. We provide in... 
T lymphocytes | Mitochondria | Cytokines | Thymocytes | Neurons | Cell death | REPORTS | Cytochromes | Mice | Potassium | Apoptosis | NEURONAL APOPTOSIS | CYTOCHROME-C | MITOCHONDRIAL APOPTOSIS | MECHANISM | MULTIDISCIPLINARY SCIENCES | BH3 DOMAINS | RELEASE | JNK PATHWAY | PROTEINS | BCL-2 FAMILY-MEMBERS | MEMBRANE PERMEABILIZATION | BH3 Interacting Domain Death Agonist Protein - deficiency | T-Lymphocytes - physiology | bcl-2-Associated X Protein - chemistry | Protein Multimerization | Stress, Physiological | bcl-2 Homologous Antagonist-Killer Protein - genetics | BH3 Interacting Domain Death Agonist Protein - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Membrane Proteins - deficiency | Caspases - metabolism | Bcl-2-Like Protein 11 | Apoptosis Regulatory Proteins - deficiency | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Neurons - physiology | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cytochromes c - metabolism | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Permeability | Proto-Oncogene Proteins - deficiency | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Models, Biological | Cerebellum - cytology | Intracellular Membranes - metabolism | bcl-2 Homologous Antagonist-Killer Protein - chemistry | Protein research | Genetic aspects | Mitochondrial DNA | Biochemical genetics | Research | Properties | Methods | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 504, Issue 7480, pp. 394 - 400
Journal Article
FASEB Journal, ISSN 0892-6638, 08/2013, Volume 27, Issue 8, pp. 3298 - 3305
Journal Article
Nature, ISSN 0028-0836, 05/2006, Volume 441, Issue 7092, pp. 523 - 527
The proto-oncogene AKT (also known as PKB) is activated in many human cancers, mostly owing to loss of the PTEN tumour suppressor. In such tumours, AKT becomes... 
FORKHEAD TRANSCRIPTION FACTOR | APOPTOSIS | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | MUTATION | PTEN | MICE | FOXO3A | CELL-SURVIVAL | PML | PHOSPHATASES | Phosphorylation | Phosphoprotein Phosphatases - metabolism | Transcription Factors - deficiency | Neoplasm Proteins - metabolism | Cell Nucleus - enzymology | Tumor Suppressor Proteins - deficiency | Nuclear Proteins - deficiency | Tumor Suppressor Proteins - genetics | Female | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | PTEN Phosphohydrolase - genetics | PTEN Phosphohydrolase - deficiency | Tumor Suppressor Proteins - metabolism | Cells, Cultured | Nuclear Proteins - metabolism | Transcription Factors - genetics | Protein Transport | Transcription Factors - metabolism | Animals | Mice | Neoplasm Proteins - deficiency | Promyelocytic Leukemia Protein | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Membranes | Transcription factors | Inhibitor drugs | Pathogenesis | Oncology | AKT protein | Inactivation | Rodents | Cell cycle | Forkhead protein | Tumorigenesis | Null cells | FOXO3 protein | Phenotypes | Deactivation | Medical treatment | Cell membranes | Cell membranes (biology) | Sterility | Mutants | Cyclin-dependent kinase inhibitor p27 | Mutation | Prostate | PTEN protein | Tumors | Cancer | Apoptosis | Index Medicus
Journal Article
New England Journal of Medicine, ISSN 0028-4793, 06/2016, Volume 374, Issue 26, pp. 2553 - 2562
Journal Article
Cell Reports, ISSN 2211-1247, 05/2013, Volume 3, Issue 5, pp. 1339 - 1345
Journal Article
Immunity, ISSN 1074-7613, 02/2008, Volume 28, Issue 2, pp. 206 - 217
Alterations in the stoichiometric balance between members of Bcl-2 and Fas apoptotic pathway could lead to the pathogenesis of systemic lupus erythematosus... 
MOLIMMUNO | APOPTOSIS | PROTEIN | B-CELL | DISEASE | NEGATIVE SELECTION | MURINE LUPUS | LUPUS-ERYTHEMATOSUS | IMMUNOLOGY | THYMOCYTES | T-CELLS | LPR | Autoimmunity | Kidney - pathology | Autoantibodies - blood | Humans | fas Receptor - deficiency | Kidney - immunology | Lupus Erythematosus, Systemic - metabolism | fas Receptor - metabolism | Membrane Proteins - deficiency | fas Receptor - immunology | Mice, Inbred MRL lpr | Bcl-2-Like Protein 11 | Apoptosis Regulatory Proteins - deficiency | Lupus Erythematosus, Systemic - immunology | Proto-Oncogene Proteins - immunology | fas Receptor - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | Macrophages - immunology | Proto-Oncogene Proteins - metabolism | Apoptosis Regulatory Proteins - immunology | Membrane Proteins - genetics | Lymphatic Diseases - immunology | Splenomegaly - immunology | Membrane Proteins - immunology | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins - deficiency | Antigen-Presenting Cells - immunology | Apoptosis Regulatory Proteins - metabolism | Macrophage Activation | Animals | B-Lymphocytes - immunology | Lupus Erythematosus, Systemic - genetics | T-Lymphocytes - immunology | Mice | Lupus Erythematosus, Systemic - pathology | Apoptosis | Lupus | Development and progression | Systemic lupus erythematosus | Statistical analysis | Disease | Pathogenesis | Lymphocytes | Mortality | Arthritis | Autoimmune diseases | Gene expression | Index Medicus
Journal Article