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Artificial Cells, Nanomedicine, and Biotechnology, ISSN 2169-1401, 12/2019, Volume 47, Issue 1, pp. 83 - 89
Journal Article
Lipids in Health and Disease, ISSN 1476-511X, 02/2019, Volume 18, Issue 1, pp. 46 - 46
Journal Article
Lipids in Health and Disease, ISSN 1476-511X, 02/2019, Volume 18, Issue 1, pp. 52 - 52
This study was designed to test the hypothesis that kappa-opioid receptor (kappa-OR) stimulation reduces palmitate-induced HUVECs apoptosis and to investigate... 
κ-Opioid receptor | eNOS | Akt | Palmitate | Apoptosis | NUTRITION & DIETETICS | kappa-Opioid receptor | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOTHELIAL-CELL APOPTOSIS | VASCULAR ENDOTHELIUM | PROTECTS | Naltrexone - analogs & derivatives | RNA, Small Interfering - genetics | Palmitic Acid - pharmacology | Human Umbilical Vein Endothelial Cells - metabolism | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Analgesics, Opioid - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Palmitic Acid - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Naltrexone - pharmacology | Human Umbilical Vein Endothelial Cells - cytology | Caspase 3 - genetics | Nitric Oxide Synthase Type III - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Receptors, Opioid, kappa - genetics | bcl-2-Associated X Protein - genetics | Cell Survival - drug effects | Receptors, Opioid, kappa - antagonists & inhibitors | Human Umbilical Vein Endothelial Cells - drug effects | Signal Transduction | Receptors, Opioid, kappa - agonists | Gene Expression Regulation | Receptors, Opioid, kappa - metabolism | bcl-2-Associated X Protein - metabolism | Nitric Oxide Synthase Type III - genetics | 3,4-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide, (trans)-Isomer - pharmacology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - genetics | Narcotic Antagonists - pharmacology | Proto-Oncogene Proteins c-bcl-2 - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | RNA, Small Interfering - metabolism | Phosphorylation | Bax protein | Bcl-2 protein | Cardiovascular disease | AKT protein | Palmitic acid | Kinases | Caspase-3 | Cell adhesion & migration | Signal transduction | Ischemia | Atherosclerosis | Narcotics | Opioid receptors | Pulmonary arteries | Caspase | Pulmonary hypertension | 1-Phosphatidylinositol 3-kinase | Endothelium | Studies | Sodium | Nitric oxide | Hypoxia | Diabetes
Journal Article
Journal of Experimental and Clinical Cancer Research, ISSN 1756-9966, 02/2019, Volume 38, Issue 1, pp. 59 - 59
Background: Long non-coding RNAs has been reported in tumorigenesis and play important roles in regulating malignant behavior of cancers, including glioma.... 
microRNA | Glioma | Transcription factor | Oncogenes | Long non-coding RNA | KEY REGULATOR | INTERACTIVE DOMAIN 1B | PROLIFERATION | DEMETHYLASE KDM5B | IN-VITRO | INVASION | ONCOLOGY | GASTRIC-CANCER | POOR-PROGNOSIS | CARCINOMA | PROGRESSION | Central Nervous System Neoplasms - metabolism | Forkhead Transcription Factors - biosynthesis | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Nuclear Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | RNA, Long Noncoding - genetics | Carcinogenesis - metabolism | Glioma - metabolism | Feedback, Physiological | Animals | Repressor Proteins - biosynthesis | Jumonji Domain-Containing Histone Demethylases - biosynthesis | Nuclear Proteins - biosynthesis | Forkhead Transcription Factors - metabolism | Survival Analysis | Cell Line, Tumor | Mice | MicroRNAs - genetics | Proto-Oncogene Proteins c-akt - metabolism | Jumonji Domain-Containing Histone Demethylases - metabolism | RNA, Long Noncoding - metabolism | Repressor Proteins - metabolism | Immunohistochemistry | Chromatin | MicroRNA | Gliomas | Analysis | Genetic transcription | Protein binding | Transcription factors | Brain cancer | DNA damage | Genes | Genomes | Breast cancer | Roles | Metastasis | Cancer therapies | Studies | Proteins | Cell growth | Feedback | MicroRNAs | Stem cells | Cell cycle | Tumorigenesis | Behavior | Tumors | Index Medicus
Journal Article
Journal Article
Journal Article
by Wei, R and Xiao, YH and Song, Y and Yuan, HP and Luo, J and Xu, W
JOURNAL OF EXPERIMENTAL & CLINICAL CANCER RESEARCH, ISSN 1756-9966, 03/2019, Volume 38, Issue 1, pp. 112 - 112
Journal Article
CELL COMMUNICATION AND SIGNALING, ISSN 1478-811X, 03/2019, Volume 17, Issue 1, pp. 21 - 21
BackgroundPlatelet-activating factor (PAF) is a potent lipid mediator whose involvement in the onset and progression of atherosclerosis is mediated by, among... 
CLATHRIN-MEDIATED ENDOCYTOSIS | SIGNAL-TRANSDUCTION PATHWAYS | CCAAT-enhancer-binding protein(C/EBP) | GROWTH-FACTOR-RECEPTOR | DENDRITIC CELLS | Platelet-activating factor | GLYCOGEN-SYNTHASE KINASE-3 | GSK-3 | Interleukin-8 | CCAAT/ENHANCER-BINDING-PROTEIN | INTESTINAL EPITHELIAL-CELLS | CELL BIOLOGY | PTP1B | GENE-EXPRESSION | FOCAL ADHESION KINASE | NF-KAPPA-B | Interleukin-8 - genetics | Receptors, G-Protein-Coupled - metabolism | Humans | Platelet Activating Factor - pharmacology | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - antagonists & inhibitors | Glycogen Synthase Kinase 3 - metabolism | Promoter Regions, Genetic - genetics | CCAAT-Enhancer-Binding Protein-beta - metabolism | Phosphothreonine - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - metabolism | Signal Transduction - drug effects | Models, Biological | Platelet Membrane Glycoproteins - metabolism | HEK293 Cells | src-Family Kinases - metabolism | Dendritic Cells - drug effects | Interleukin-8 - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Binding Sites | Dendritic Cells - metabolism | Phosphatases | Atherosclerosis | Physiological aspects | Development and progression | Genetic aspects | Research | Tyrosine | Synthesis | Interleukins | Dendritic cells | Glycogen | Phenols | DNA binding proteins | Protein binding | Phosphorylation | Transcription factors | Lipids | AKT protein | Kinases | Phosphatase | Western blotting | Recruitment | Proteins | Signal transduction | Blood platelets | Rodents | Down-regulation | Fibroblasts | Interleukin 8 | Enzyme-linked immunosorbent assay | Cytokines | Threonine | Glycogen synthase kinase 3 | Gene expression | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Monocytes | Arteriosclerosis | Chemokines | Binding sites | Protein-tyrosine-phosphatase
Journal Article
Journal Article
BMC CANCER, ISSN 1471-2407, 03/2018, Volume 19, Issue 1, pp. 202 - 202
Journal Article
Molecular Cancer, ISSN 1476-4598, 02/2019, Volume 18, Issue 1, pp. 20 - 20
BackgroundCircRNA has emerged as a new non-coding RNA that plays crucial roles in tumour initiation and development. MiRNA sponge' is the most reported role... 
ceRNA | circRNA | AKT1 | miRNA | Organoid | PDX model | Gastric cancer | SURVIVAL | MIR-149-5P | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | EMT | EPITHELIAL-MESENCHYMAL TRANSITION | BIOGENESIS | ONCOLOGY | EXOSOMES | PI3K/AKT/MTOR PATHWAY | EXPRESSION | Exosomes - metabolism | RNA, Small Interfering - genetics | Adenocarcinoma - pathology | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Exosomes - pathology | Male | MicroRNAs - metabolism | Stomach Neoplasms - pathology | Proto-Oncogene Proteins c-akt - genetics | RNA - genetics | Adenocarcinoma - metabolism | Heterografts | TOR Serine-Threonine Kinases - genetics | Female | Adenocarcinoma - genetics | Proto-Oncogene Proteins c-akt - metabolism | RNA - metabolism | Stomach Neoplasms - genetics | RNA - antagonists & inhibitors | Signal Transduction | Lymphatic Metastasis | Disease Progression | Animals | Survival Analysis | Cell Line, Tumor | Aged | Mice | MicroRNAs - genetics | Neoplasm Staging | Stomach Neoplasms - mortality | Adenocarcinoma - mortality | Cell Movement | RNA, Small Interfering - metabolism | Development and progression | MicroRNA | Stomach cancer | Analysis | Cancer | TOR protein | Cell proliferation | Cell culture | Animal models | Transcription | Homeostasis | AKT protein | Biosynthesis | Metastasis | Kinases | Assaying | Autophagy | Cell adhesion & migration | Metastases | Proteins | Liver cancer | Angiogenesis | Signal transduction | Cell growth | Energy | AKT1 protein | MiRNA | Breast cancer | Roles | Metabolism | Gene expression | Ribonucleic acid--RNA | Bladder cancer | Energy balance | Communications systems | MicroRNAs | Medical prognosis | Kidney cancer | Circular RNA | Immunofluorescence | Non-coding RNA | Binding sites | Cell migration | Phagocytosis | Tumors
Journal Article