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Journal Article
Nature Immunology, ISSN 1529-2908, 12/2013, Volume 14, Issue 12, pp. 1285 - 1293
Cell-mediated immunity critically depends on the localization of lymphocytes at sites of infection. While some memory T cells recirculate, a distinct lineage... 
AFFERENT LYMPHATICS | NONLYMPHOID TISSUE | RM CELLS | IN-VIVO | INFECTION | KRUPPEL-LIKE FACTOR-2 | PERIPHERAL-TISSUES | DIFFERENTIATION | IMMUNOLOGY | LYMPHOCYTE EGRESS | CUTTING EDGE | Antigens, CD - immunology | Transcription, Genetic - drug effects | Lectins, C-Type - immunology | Phosphatidylinositol 3-Kinases - metabolism | Lectins, C-Type - genetics | Antigens, CD - genetics | Phosphatidylinositol 3-Kinases - immunology | Proto-Oncogene Proteins c-akt - genetics | Antigens, CD - metabolism | Lectins, C-Type - metabolism | Signal Transduction - immunology | Flow Cytometry | Kruppel-Like Transcription Factors - metabolism | CD8-Positive T-Lymphocytes - metabolism | Receptors, Lysosphingolipid - genetics | Interleukins - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Receptors, Lysosphingolipid - metabolism | Immunologic Memory - immunology | Antigens, Differentiation, T-Lymphocyte - immunology | Interleukin-33 | Mice, Inbred C57BL | Cells, Cultured | Antigens, Differentiation, T-Lymphocyte - metabolism | Signal Transduction - genetics | Down-Regulation - drug effects | Mice, Inbred Strains | Reverse Transcriptase Polymerase Chain Reaction | Phosphatidylinositol 3-Kinases - genetics | Receptors, Lysosphingolipid - immunology | Transforming Growth Factor beta - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Proto-Oncogene Proteins c-akt - immunology | Signal Transduction - drug effects | Down-Regulation - immunology | Antigens, Differentiation, T-Lymphocyte - genetics | Transcription, Genetic - immunology | CD8-Positive T-Lymphocytes - immunology | Kruppel-Like Transcription Factors - genetics | Kruppel-Like Transcription Factors - immunology
Journal Article
Nature Immunology, ISSN 1529-2908, 02/2013, Volume 14, Issue 2, pp. 162 - 171
Signaling through the G protein-coupled receptors for the complement fragments C3a and C5a (C3aR and C5aR, respectively) by dendritic cells and CD4(+) cells... 
Forkhead Transcription Factors - immunology | Cell Communication - immunology | T-Lymphocytes, Regulatory - metabolism | TOR Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - metabolism | Complement C5a - metabolism | Receptors, Complement - metabolism | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Transforming Growth Factor beta1 - immunology | Forkhead Transcription Factors - metabolism | Class Ib Phosphatidylinositol 3-Kinase - metabolism | T-Lymphocytes, Regulatory - cytology | Interleukin-10 - metabolism | Receptor, Anaphylatoxin C5a - immunology | Cell Differentiation | Proto-Oncogene Proteins c-akt - metabolism | Complement C3a - metabolism | Interleukin-6 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Receptors, Chemokine - metabolism | Gene Expression Regulation | Mice, Transgenic | Receptors, Chemokine - immunology | Receptor Cross-Talk - immunology | TOR Serine-Threonine Kinases - immunology | Animals | Proto-Oncogene Proteins c-akt - immunology | Receptors, Complement - immunology | Cyclic AMP-Dependent Protein Kinases - immunology | Interleukin-6 - immunology | Mice | Complement C3a - immunology | Complement C5a - immunology | Class Ib Phosphatidylinositol 3-Kinase - immunology | Interleukin-10 - immunology | Receptor, Anaphylatoxin C5a - metabolism
Journal Article
Immunologic Research, ISSN 0257-277X, 10/2011, Volume 51, Issue 1, pp. 45 - 60
Complement system activation plays an important role in both innate and acquired immunity. Activation of the complement and the subsequent formation of C5b-9... 
Allergology | Signal transduction | Immunology | Medicine & Public Health | Cell cycle | Internal Medicine | Membrane attack complex | C5b-9 terminal complement complex | Transcriptional regulation | Medicine/Public Health, general | Apoptosis | ACTIVATED PROTEIN-KINASE | SYSTEMIC LUPUS-ERYTHEMATOSUS | GUILLAIN-BARRE-SYNDROME | ULCERATIVE-COLITIS | GLOMERULAR EPITHELIAL-CELLS | C5B-9 COMPLEX | IMMUNOLOGY | S-PROTEIN | MANNAN-BINDING LECTIN | ACUTE MYOCARDIAL-INFARCTION | SMOOTH-MUSCLE-CELLS | Forkhead Transcription Factors - immunology | CDC2 Protein Kinase | Complement Membrane Attack Complex - metabolism | Cyclin-Dependent Kinase 4 - genetics | Humans | Caspase 8 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Complement Membrane Attack Complex - immunology | MAP Kinase Signaling System - immunology | Phosphatidylinositol 3-Kinases - immunology | Cell Cycle Proteins - immunology | Cyclin B - immunology | Forkhead Transcription Factors - metabolism | Muscle Proteins - metabolism | bcl-Associated Death Protein - metabolism | Cell Membrane - metabolism | Phosphorylation - immunology | Cyclin-Dependent Kinases | Proto-Oncogene Proteins c-akt - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | Cyclin-Dependent Kinase 2 - metabolism | Mitogen-Activated Protein Kinase 3 - immunology | Muscle Proteins - immunology | G1 Phase - immunology | S Phase - immunology | Nerve Tissue Proteins - immunology | Cell Cycle Proteins - metabolism | bcl-Associated Death Protein - immunology | Cyclin-Dependent Kinase 4 - metabolism | Nerve Tissue Proteins - metabolism | Caspase 8 - immunology | Animals | Apoptosis - immunology | Proto-Oncogene Proteins c-akt - immunology | Mitogen-Activated Protein Kinase 3 - metabolism | BH3 Interacting Domain Death Agonist Protein - immunology | Cyclin B - metabolism | Cell Membrane - immunology | Forkhead Box Protein O1 | Cyclin-Dependent Kinase 2 - immunology | Medical colleges | Membranes | Immune system | apoptosis | membrane attack complex | cell cycle | signal transduction | transcriptional regulation
Journal Article
JOURNAL OF IMMUNOLOGY, ISSN 0022-1767, 01/2015, Volume 194, Issue 2, pp. 584 - 594
AMP-activated protein kinase (AMPK) is a conserved serine/threonine kinase with a critical function in the regulation of metabolic pathways in eukaryotic... 
MAMMALIAN TARGET | CHRONIC ENTEROCOLITIS | PHOSPHATIDYLINOSITOL 3-KINASE | IN-VIVO | GENE-EXPRESSION | MAXIMAL ACTIVATION | IMMUNOLOGY | SERINE PHOSPHORYLATION | CELLULAR-ENERGY | APOLIPOPROTEIN-E | FUNCTIONAL PHENOTYPE | Inflammation - chemically induced | Inflammation - pathology | Multiprotein Complexes - genetics | Enzyme Activation - immunology | Phosphatidylinositol 3-Kinases - immunology | Proto-Oncogene Proteins c-akt - genetics | Mechanistic Target of Rapamycin Complex 1 | Signal Transduction - immunology | Phosphorylation - genetics | TOR Serine-Threonine Kinases - genetics | AMP-Activated Protein Kinases - immunology | Phosphorylation - immunology | Interleukin-4 - genetics | Phosphorylation - drug effects | Macrophages - immunology | STAT3 Transcription Factor - genetics | Transforming Growth Factor beta - immunology | Lipopolysaccharides - toxicity | Macrophages - pathology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Inflammation - immunology | Signal Transduction - genetics | Enzyme Activation - drug effects | Mice, Knockout | Gene Expression Regulation - drug effects | Phosphatidylinositol 3-Kinases - genetics | Interleukin-4 - immunology | TOR Serine-Threonine Kinases - immunology | Animals | Proto-Oncogene Proteins c-akt - immunology | Transforming Growth Factor beta - genetics | Signal Transduction - drug effects | Interleukin-10 - genetics | Inflammation - genetics | Mice | STAT3 Transcription Factor - immunology | Enzyme Activation - genetics | Interleukin-10 - immunology | AMP-Activated Protein Kinases - genetics | Multiprotein Complexes - immunology
Journal Article
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 2, pp. 296 - 305
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 699 - 714
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