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Journal of cellular and molecular medicine, ISSN 1582-1838, 2017, Volume 21, Issue 11, pp. 2884 - 2895
... constituents (such as heat shock proteins and high‐mobility group B1 protein), which can trigger DC maturation . DCs are antigen‐presenting cells that develop directly... 
microRNAs | dendritic cells | immune inflammatory response | cardiomyocyte apoptosis | SIGNAL | MEDICINE, RESEARCH & EXPERIMENTAL | REPERFUSION INJURY | INVOLVEMENT | MICRORNA-150 PROTECTS | CELL BIOLOGY | HEART | NECROTIC CELLS | ACUTE MYOCARDIAL-INFARCTION | DIFFERENTIATION | NF-KAPPA-B | LYMPHOCYTES | Antigens, CD - immunology | Myocardial Infarction - genetics | B7-2 Antigen - genetics | Immunoglobulins - immunology | Myocytes, Cardiac - immunology | Dendritic Cells - immunology | Dendritic Cells - pathology | Apoptosis - genetics | Immunoglobulins - genetics | Antigens, CD - genetics | Necrosis - pathology | Transcription Factor RelA - genetics | Myocardial Infarction - immunology | Necrosis - immunology | Bone Marrow Cells - immunology | Janus Kinase 1 - genetics | Myocardial Infarction - pathology | Transcription Factor RelA - immunology | CD40 Antigens - genetics | Membrane Glycoproteins - immunology | B7-2 Antigen - immunology | Signal Transduction | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - immunology | MicroRNAs - immunology | STAT1 Transcription Factor - genetics | Janus Kinase 1 - immunology | STAT1 Transcription Factor - immunology | Membrane Glycoproteins - genetics | Myocytes, Cardiac - pathology | Animals | Models, Biological | Proto-Oncogene Proteins c-fos - genetics | Mice | MicroRNAs - genetics | Necrosis - genetics | Primary Cell Culture | CD40 Antigens - immunology | Immune response | MicroRNA | Dendritic cells | Analysis | Apoptosis | Original
Journal Article
Cell reports (Cambridge), ISSN 2211-1247, 2014, Volume 8, Issue 1, pp. 75 - 83
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2008, Volume 28, Issue 29, pp. 7344 - 7349
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 1480 - 11
...) rats, thus contributing to Muller cell gliosis, characterized by upregulated expression of glial fibrillary acidic protein (GFAP... 
ACTIVATED PROTEIN-KINASE | MESSENGER-RNA | POTASSIUM CHANNELS | MULTIDISCIPLINARY SCIENCES | GANGLION-CELLS | MEMBRANE CONDUCTANCE | INTRAOCULAR-PRESSURE | OCULAR HYPERTENSION MODEL | GLIAL-CELLS | EXPERIMENTAL GLAUCOMA | NUCLEUS-ACCUMBENS | Retina - drug effects | Glial Fibrillary Acidic Protein - genetics | Retina - metabolism | Nitriles - pharmacology | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Ependymoglial Cells - metabolism | Gliosis - chemically induced | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Ocular Hypertension - metabolism | Glial Fibrillary Acidic Protein - metabolism | Ocular Hypertension - drug therapy | Gliosis - pathology | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | JNK Mitogen-Activated Protein Kinases - genetics | Ependymoglial Cells - pathology | Disease Models, Animal | Barium Compounds - administration & dosage | Butadienes - pharmacology | Gliosis - genetics | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Ocular Hypertension - pathology | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - metabolism | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Potassium Channels, Inwardly Rectifying - genetics | Ependymoglial Cells - drug effects | Rats, Sprague-Dawley | Ocular Hypertension - genetics | Cyclic AMP Response Element-Binding Protein - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Chlorides - administration & dosage | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cyclic AMP Response Element-Binding Protein - metabolism | Proto-Oncogene Proteins c-fos - genetics | Gliosis - drug therapy | Protein Kinase Inhibitors - pharmacology | Chronic Disease | Potassium Channels, Inwardly Rectifying - metabolism | Retina - pathology | Mitogen-Activated Protein Kinase 1 - metabolism | Transcription factors | Potassium channels (inwardly-rectifying) | P elements | Glial fibrillary acidic protein | c-Jun protein | Extracellular signal-regulated kinase | MAP kinase | Retina | Injection | Cyclic AMP response element-binding protein | Kinases | Proteins | Signal transduction | Gliosis | Protein kinase | Rodents | c-Fos protein | Glutamic acid receptors (metabotropic)
Journal Article
by Chan, QKY and Lam, HM and Ng, CF and Lee, AYY and Chan, ESY and Ng, HK and Ho, SM and Lau, KM
Cell Death and Differentiation, ISSN 1350-9047, 09/2010, Volume 17, Issue 9, pp. 1511 - 1523
G-protein-coupled receptor-30 (GPR30) shows estrogen-binding affinity and mediates non-genomic signaling of estrogen to regulate cell growth... 
COUPLED RECEPTOR GPR30 | ER-BETA | signal transduction | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | G | G-protein-coupled receptor-30 | G2/M | PROLIFERATION | G-PROTEIN | non-genomic estrogen signaling | ESTROGEN-RECEPTOR | 17-BETA-ESTRADIOL | CELL BIOLOGY | NUCLEAR ACCUMULATION | Y TRANSCRIPTION FACTOR | EXPRESSION | 1-[4-(6-bromobenzo[1,3]dioxol-5yl)-3a,4,5,9b-tetrahydro-3H-cyclopenta[c]quinolin-8-yl]-ethanone | Prostatic Neoplasms - metabolism | RNA, Small Interfering - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptors, G-Protein-Coupled - agonists | G2 Phase - drug effects | Quinolines - pharmacology | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Antineoplastic Agents - pharmacology | Flavonoids - pharmacology | Phosphorylation - drug effects | Prostatic Neoplasms - drug therapy | Proto-Oncogene Proteins - metabolism | Prostatic Neoplasms - pathology | Proto-Oncogene Proteins c-jun - genetics | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Proto-Oncogene Proteins c-fos - metabolism | Receptors, Estrogen - antagonists & inhibitors | Proto-Oncogene Proteins - genetics | Cyclopentanes - pharmacology | Xenograft Model Antitumor Assays | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Mice, Nude | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | Proto-Oncogene Proteins c-fos - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Receptors, G-Protein-Coupled - genetics | Cell Cycle - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Molecular Endocrinology, ISSN 0888-8809, 01/2012, Volume 26, Issue 1, pp. 37 - 51
Journal Article
Nature (London), ISSN 1476-4687, 2002, Volume 416, Issue 6882, pp. 744 - 749
Journal Article
Journal Article
Aging Cell, ISSN 1474-9718, 02/2019, Volume 18, Issue 1, pp. e12872 - n/a
Journal Article