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Cell reports (Cambridge), ISSN 2211-1247, 2014, Volume 8, Issue 1, pp. 75 - 83
Journal Article
by Chan, QKY and Lam, HM and Ng, CF and Lee, AYY and Chan, ESY and Ng, HK and Ho, SM and Lau, KM
Cell Death and Differentiation, ISSN 1350-9047, 09/2010, Volume 17, Issue 9, pp. 1511 - 1523
G-protein-coupled receptor-30 (GPR30) shows estrogen-binding affinity and mediates non-genomic signaling of estrogen to regulate cell growth... 
COUPLED RECEPTOR GPR30 | ER-BETA | signal transduction | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | G | G-protein-coupled receptor-30 | G2/M | PROLIFERATION | G-PROTEIN | non-genomic estrogen signaling | ESTROGEN-RECEPTOR | 17-BETA-ESTRADIOL | CELL BIOLOGY | NUCLEAR ACCUMULATION | Y TRANSCRIPTION FACTOR | EXPRESSION | 1-[4-(6-bromobenzo[1,3]dioxol-5yl)-3a,4,5,9b-tetrahydro-3H-cyclopenta[c]quinolin-8-yl]-ethanone | Prostatic Neoplasms - metabolism | RNA, Small Interfering - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptors, G-Protein-Coupled - agonists | G2 Phase - drug effects | Quinolines - pharmacology | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Antineoplastic Agents - pharmacology | Flavonoids - pharmacology | Phosphorylation - drug effects | Prostatic Neoplasms - drug therapy | Proto-Oncogene Proteins - metabolism | Prostatic Neoplasms - pathology | Proto-Oncogene Proteins c-jun - genetics | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Proto-Oncogene Proteins c-fos - metabolism | Receptors, Estrogen - antagonists & inhibitors | Proto-Oncogene Proteins - genetics | Cyclopentanes - pharmacology | Xenograft Model Antitumor Assays | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Mice, Nude | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | Proto-Oncogene Proteins c-fos - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Receptors, G-Protein-Coupled - genetics | Cell Cycle - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Nature (London), ISSN 1476-4687, 2002, Volume 416, Issue 6882, pp. 744 - 749
Journal Article
Molecular Endocrinology, ISSN 0888-8809, 01/2012, Volume 26, Issue 1, pp. 37 - 51
Journal Article
Scientific reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 1480 - 11
...) rats, thus contributing to Muller cell gliosis, characterized by upregulated expression of glial fibrillary acidic protein (GFAP... 
ACTIVATED PROTEIN-KINASE | MESSENGER-RNA | POTASSIUM CHANNELS | MULTIDISCIPLINARY SCIENCES | GANGLION-CELLS | MEMBRANE CONDUCTANCE | INTRAOCULAR-PRESSURE | OCULAR HYPERTENSION MODEL | GLIAL-CELLS | EXPERIMENTAL GLAUCOMA | NUCLEUS-ACCUMBENS | Retina - drug effects | Glial Fibrillary Acidic Protein - genetics | Retina - metabolism | Nitriles - pharmacology | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Ependymoglial Cells - metabolism | Gliosis - chemically induced | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Ocular Hypertension - metabolism | Glial Fibrillary Acidic Protein - metabolism | Ocular Hypertension - drug therapy | Gliosis - pathology | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | JNK Mitogen-Activated Protein Kinases - genetics | Ependymoglial Cells - pathology | Disease Models, Animal | Barium Compounds - administration & dosage | Butadienes - pharmacology | Gliosis - genetics | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Ocular Hypertension - pathology | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - metabolism | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Potassium Channels, Inwardly Rectifying - genetics | Ependymoglial Cells - drug effects | Rats, Sprague-Dawley | Ocular Hypertension - genetics | Cyclic AMP Response Element-Binding Protein - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Chlorides - administration & dosage | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cyclic AMP Response Element-Binding Protein - metabolism | Proto-Oncogene Proteins c-fos - genetics | Gliosis - drug therapy | Protein Kinase Inhibitors - pharmacology | Chronic Disease | Potassium Channels, Inwardly Rectifying - metabolism | Retina - pathology | Mitogen-Activated Protein Kinase 1 - metabolism | Transcription factors | Potassium channels (inwardly-rectifying) | P elements | Glial fibrillary acidic protein | c-Jun protein | Extracellular signal-regulated kinase | MAP kinase | Retina | Injection | Cyclic AMP response element-binding protein | Kinases | Proteins | Signal transduction | Gliosis | Protein kinase | Rodents | c-Fos protein | Glutamic acid receptors (metabotropic)
Journal Article
Aging Cell, ISSN 1474-9718, 02/2019, Volume 18, Issue 1, pp. e12872 - n/a
Journal Article
PloS one, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e80873
Numerous studies have reported that inflammatory cytokines are important mediators for osteoclastogenesis, thereby causing excessive bone resorption and... 
REHMANNIA-GLUTINOSA | ACTIVATION | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | RECEPTOR | DIFFERENTIATION | MOLECULAR-BASIS | CELL | MICE LACKING | OSTEOPOROSIS | BONE-RESORPTION | Interleukin-6 - antagonists & inhibitors | RANK Ligand - metabolism | Reactive Oxygen Species - metabolism | NF-kappa B - metabolism | Osteoclasts - cytology | Bone Marrow Cells - drug effects | Female | Cell Differentiation | Interleukin-1beta - biosynthesis | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-1beta - antagonists & inhibitors | Phenols - pharmacology | Proto-Oncogene Proteins c-fos - antagonists & inhibitors | Cell Survival - drug effects | Glucosides - pharmacology | NF-kappa B - antagonists & inhibitors | Ovariectomy | Signal Transduction | Bone Marrow Cells - cytology | NFATC Transcription Factors - metabolism | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | Antioxidants - pharmacology | Macrophages - cytology | Osteoclasts - metabolism | Mice, Inbred ICR | Bone Resorption - prevention & control | RANK Ligand - genetics | Macrophages - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | NF-kappa B - genetics | RANK Ligand - antagonists & inhibitors | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Interleukin-6 - biosynthesis | Macrophages - drug effects | Proto-Oncogene Proteins c-fos - genetics | Mice | NFATC Transcription Factors - antagonists & inhibitors | Tumor Necrosis Factor-alpha - biosynthesis | Bone Marrow Cells - metabolism | Osteoclasts - drug effects | Tumor Necrosis Factor-alpha - antagonists & inhibitors | NFATC Transcription Factors - genetics | Osteoprogenitor cells | Transcription factors | Phosphorylation | Interleukin | Arthritis | Activation | Kinases | Macrophages | Interleukin 6 | Antioxidants | Bone resorption | Osteoporosis | Bone growth | Bone marrow | Tumor necrosis factor-TNF | Biocompatibility | Inhibition | Pretreatment | Bone loss | TRANCE protein | c-Fos protein | NF-κB protein | Cytokines | c-Jun protein | Pharmacology | Inflammation | Dentistry | Tumor necrosis factor-α | Orthodontics | Medicine | Osteoclastogenesis | Stem cells | Ligands | Osteoclasts | Laboratory animals | Differentiation
Journal Article