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Cellular Signalling, ISSN 0898-6568, 01/2013, Volume 25, Issue 1, pp. 150 - 158
JNK signaling functions to induce defense mechanisms that protect organisms against a variety of different situations. The sestrin 2 gene, a p53-regulated... 
Excisanin A | JNK/c-Jun | Sestrin 2 | Autophagy | TARGET | GADD45 | APOPTOSIS | N-TERMINAL KINASE | ENDOPLASMIC-RETICULUM STRESS | KAPPA-B | CELL BIOLOGY | GROWTH | LEUKEMIA-CELLS | RABDOSIA-RUBESCENS | TRANSCRIPTION FACTOR | Phosphorylation | Mitogen-Activated Protein Kinase 8 - antagonists & inhibitors | Microtubule-Associated Proteins - metabolism | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Mitogen-Activated Protein Kinase 9 - genetics | Autophagy - drug effects | Proto-Oncogene Proteins c-jun - antagonists & inhibitors | Chromatin Immunoprecipitation | RNA Interference | Mitogen-Activated Protein Kinase 8 - genetics | Nuclear Proteins - genetics | Diterpenes - pharmacology | Mitogen-Activated Protein Kinase 9 - metabolism | Proto-Oncogene Proteins c-jun - genetics | Mitogen-Activated Protein Kinase 8 - metabolism | Nuclear Proteins - metabolism | Enzyme Activation - drug effects | Up-Regulation - drug effects | MAP Kinase Signaling System - drug effects | Autophagy-Related Protein 5 | Proto-Oncogene Proteins c-jun - metabolism | Mitogen-Activated Protein Kinase 9 - antagonists & inhibitors | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | RNA, Small Interfering - metabolism | Tumor proteins | Analysis | Cancer | Index Medicus | Therapy | Cellular | Pathways | Genes | Activation | Psychological effects | Signalling
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 05/2014, Volume 447, Issue 4, pp. 672 - 677
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/1998, Volume 273, Issue 29, pp. 18623 - 18632
Journal Article
Molecular Cell, ISSN 1097-2765, 2001, Volume 7, Issue 3, pp. 517 - 528
Decreased Fas expression during tumor progression often results in a loss of Fas-ligand (FasL)-mediated apoptosis. Human and mouse melanoma exhibit an inverse... 
PERIPHERAL-BLOOD CELLS | CONSTITUTIVE ACTIVATION | IFN-GAMMA | ORIENTATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | DOWN-REGULATION | UV-INDUCED APOPTOSIS | MALIGNANT-MELANOMA | CYCLE PROGRESSION | EXPRESSION | CELL BIOLOGY | Transcriptional Activation - genetics | Membrane Glycoproteins - metabolism | Apoptosis - radiation effects | Humans | RNA, Messenger - metabolism | fas Receptor - biosynthesis | fas Receptor - metabolism | Promoter Regions, Genetic - genetics | Transcription Factor AP-1 - metabolism | Proto-Oncogene Proteins c-jun - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Radiation Tolerance - radiation effects | Ultraviolet Rays | Melanoma - genetics | Protein Binding - radiation effects | Trans-Activators - genetics | fas Receptor - genetics | Fibroblasts | Response Elements - genetics | Tumor Cells, Cultured | DNA-Binding Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-jun - genetics | RNA, Messenger - genetics | Gene Expression Regulation | Melanoma - radiotherapy | Radiation Tolerance - genetics | Up-Regulation - genetics | DNA - metabolism | Melanoma - pathology | DNA-Binding Proteins - genetics | DNA - genetics | Fas Ligand Protein | Animals | Proto-Oncogene Proteins c-jun - metabolism | Trans-Activators - metabolism | Mice | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Transcription, Genetic - genetics | Sequence Deletion - genetics | c-Jun protein | FasL protein | Index Medicus
Journal Article
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 12/2016, Volume 101, pp. 129 - 142
Inflammatory breast cancer (IBC) is the most aggressive form of breast cancer. Treatment options are limited and the mechanisms underlying its aggressiveness... 
Intermittent hypoxia | Oxidative stress | NF-κB | Reactive oxygen species | ROS | Tenascin-C | Inflammatory breast cancer | ANGIOGENESIS | KEY REGULATOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLONGED HYPOXIA | MOTIF OVER-REPRESENTATION | NF-kappa B | NUCLEAR-FACTOR | REDOX REGULATION | CYCLING HYPOXIA | ENDOCRINOLOGY & METABOLISM | TRANSCRIPTIONAL REGULATION | Oxygen - pharmacology | Interleukin-1beta - pharmacology | Epithelial Cells - metabolism | Tenascin - genetics | Nitriles - pharmacology | Oxidative Stress | Epithelial Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | JNK Mitogen-Activated Protein Kinases - metabolism | NF-KappaB Inhibitor alpha - genetics | NF-kappa B - metabolism | Mammary Glands, Human - metabolism | Sulfones - pharmacology | Tenascin - metabolism | Proto-Oncogene Proteins c-jun - antagonists & inhibitors | Cell Hypoxia | Matrix Metalloproteinase 9 - metabolism | Neoplasm Metastasis | Cyclooxygenase 2 - genetics | Matrix Metalloproteinase 9 - genetics | NF-E2-Related Factor 2 - genetics | JNK Mitogen-Activated Protein Kinases - genetics | Phosphorylation - drug effects | Mammary Glands, Human - drug effects | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction | Proto-Oncogene Proteins c-jun - genetics | Epithelial Cells - pathology | Mammary Glands, Human - pathology | Anthracenes - pharmacology | NF-kappa B - genetics | Proto-Oncogene Proteins c-jun - metabolism | NF-E2-Related Factor 2 - metabolism | NF-KappaB Inhibitor alpha - metabolism | Acetylcysteine - pharmacology | Cyclooxygenase 2 - metabolism | Cell Line, Tumor | Index Medicus
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 2010, Volume 142, Issue 3, pp. 392 - 403
Excitotoxicity is one of the main features responsible for neuronal cell death after acute brain injury and in several neurodegenerative disorders, for which... 
c-Jun | Neuroprotection | Tf-lipoplexes | siRNA | Kainate | Excitotoxicity | RNA INTERFERENCE | DISEASE GENES | APOPTOSIS | BRAIN-INJURY | N-TERMINAL KINASE | MAP KINASES | CHEMISTRY, MULTIDISCIPLINARY | CELL-DEATH | SIGNALING PATHWAY | HYPOXIC-ISCHEMIC INJURY | PHARMACOLOGY & PHARMACY | CEREBRAL-ISCHEMIA | Immunohistochemistry | Neuroprotective Agents - therapeutic use | Neurons - pathology | Gene Silencing - drug effects | Humans | Seizures - drug therapy | Seizures - metabolism | Quaternary Ammonium Compounds - chemistry | Drug Carriers - chemistry | Proto-Oncogene Proteins c-jun - antagonists & inhibitors | Cholesterol - chemistry | Neuroprotective Agents - pharmacology | Seizures - pathology | Drug Compounding | Fatty Acids, Monounsaturated - chemistry | Neurons - metabolism | Cell Culture Techniques | Kainic Acid - toxicity | Neurons - drug effects | Neuroprotective Agents - administration & dosage | Cell Survival - drug effects | Proto-Oncogene Proteins c-jun - genetics | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Cells, Cultured | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Protein Transport | Animals | RNA, Small Interfering - therapeutic use | Transferrin - chemistry | Mice | Liposomes | Glutamic Acid - toxicity | RNA, Small Interfering - administration & dosage | Index Medicus | Antibodies
Journal Article