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Nature communications, ISSN 2041-1723, 2016, Volume 7, Issue 1, p. 11309
.... Mechanistic analyses indicate that miR-3188 directly targets mTOR to inactivate p-PI3K/p-AKT/c-JUN and induces its own expression... 
EPITHELIAL-MESENCHYMAL TRANSITION | CANCER-CELLS | ACTIVATION | INHIBITION | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | C-MYC | TUMOR-SUPPRESSOR | CISPLATIN RESISTANCE | RECEPTOR | PROMOTES | Nasopharyngeal Neoplasms - genetics | Neoplasm Transplantation | Nasopharyngeal Neoplasms - metabolism | Carcinoma | Prognosis | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | JNK Mitogen-Activated Protein Kinases - metabolism | Male | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Nasopharyngeal Neoplasms - pathology | TOR Serine-Threonine Kinases - genetics | Cell Transformation, Neoplastic - genetics | Forkhead Transcription Factors - metabolism | Aged, 80 and over | Adult | Female | JNK Mitogen-Activated Protein Kinases - genetics | Proto-Oncogene Proteins c-akt - metabolism | Nasopharyngeal Neoplasms - diagnosis | Nasopharyngeal Carcinoma | Signal Transduction | Forkhead Transcription Factors - genetics | Cell Transformation, Neoplastic - metabolism | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | Feedback, Physiological | Animals | Survival Analysis | Cell Line, Tumor | Aged | Forkhead Box Protein O1 | Mice | MicroRNAs - genetics | Cell Transformation, Neoplastic - pathology | Neoplasm Staging
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 03/2014, Volume 34, Issue 6, pp. 1100 - 1120
Journal Article
by Chan, QKY and Lam, HM and Ng, CF and Lee, AYY and Chan, ESY and Ng, HK and Ho, SM and Lau, KM
Cell Death and Differentiation, ISSN 1350-9047, 09/2010, Volume 17, Issue 9, pp. 1511 - 1523
G-protein-coupled receptor-30 (GPR30) shows estrogen-binding affinity and mediates non-genomic signaling of estrogen to regulate cell growth... 
COUPLED RECEPTOR GPR30 | ER-BETA | signal transduction | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | G | G-protein-coupled receptor-30 | G2/M | PROLIFERATION | G-PROTEIN | non-genomic estrogen signaling | ESTROGEN-RECEPTOR | 17-BETA-ESTRADIOL | CELL BIOLOGY | NUCLEAR ACCUMULATION | Y TRANSCRIPTION FACTOR | EXPRESSION | 1-[4-(6-bromobenzo[1,3]dioxol-5yl)-3a,4,5,9b-tetrahydro-3H-cyclopenta[c]quinolin-8-yl]-ethanone | Prostatic Neoplasms - metabolism | RNA, Small Interfering - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptors, G-Protein-Coupled - agonists | G2 Phase - drug effects | Quinolines - pharmacology | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Antineoplastic Agents - pharmacology | Flavonoids - pharmacology | Phosphorylation - drug effects | Prostatic Neoplasms - drug therapy | Proto-Oncogene Proteins - metabolism | Prostatic Neoplasms - pathology | Proto-Oncogene Proteins c-jun - genetics | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Proto-Oncogene Proteins c-fos - metabolism | Receptors, Estrogen - antagonists & inhibitors | Proto-Oncogene Proteins - genetics | Cyclopentanes - pharmacology | Xenograft Model Antitumor Assays | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Mice, Nude | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | Proto-Oncogene Proteins c-fos - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Receptors, G-Protein-Coupled - genetics | Cell Cycle - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2017, Volume 292, Issue 43, pp. 17919 - 17927
Journal Article
The EMBO Journal, ISSN 0261-4189, 08/2002, Volume 21, Issue 15, pp. 4104 - 4113
.... Here, we show that constitutively activated AP‐1 with robust c‐Jun and JunB overexpression is found in all tumor cells of patients with classical Hodgkin's disease. A similar AP... 
MAPK | target genes | metastasis | lymphoma | oncogenesis | Target genes | Oncogenesis | Metastasis | Lymphoma | APOPTOSIS | CYCLIN D1 EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEPATOCYTE GROWTH-FACTOR | TRANSCRIPTION | AP-1 | CELL BIOLOGY | DISEASE | ACTIVATED PROTEIN-KINASES | PROMOTER | EPSTEIN-BARR-VIRUS | STERNBERG CELLS | Proto-Oncogene Proteins c-met - biosynthesis | Proto-Oncogene Proteins c-jun - physiology | Tetradecanoylphorbol Acetate - pharmacology | Mitogens - pharmacology | DNA, Neoplasm - metabolism | Hodgkin Disease - genetics | Hodgkin Disease - pathology | Transcription Factor AP-1 - genetics | Humans | Neoplasm Proteins - physiology | Proto-Oncogene Proteins c-jun - biosynthesis | Cyclins - genetics | Recombinant Fusion Proteins - physiology | Lymphoma, Non-Hodgkin - pathology | Cyclin D2 | Cyclins - biosynthesis | Transfection | Ultraviolet Rays | Cell Transformation, Neoplastic - genetics | Cell Division | Receptors, Chemokine - biosynthesis | Transcription, Genetic | Tumor Cells, Cultured - radiation effects | Gene Expression Regulation, Neoplastic - drug effects | Reed-Sternberg Cells - drug effects | Neoplasm Proteins - genetics | Receptors, Chemokine - genetics | Gene Expression Regulation, Neoplastic - physiology | Genes, jun | MAP Kinase Signaling System - radiation effects | Lymphoma, Large B-Cell, Diffuse - pathology | Neoplasm Proteins - biosynthesis | Proto-Oncogene Proteins c-jun - genetics | Reed-Sternberg Cells - radiation effects | Receptors, CCR7 | Tumor Cells, Cultured - drug effects | Tumor Cells, Cultured - metabolism | Lymphoma, Non-Hodgkin - genetics | Proto-Oncogene Proteins c-met - genetics | Reed-Sternberg Cells - metabolism | MAP Kinase Signaling System - drug effects | Transcription Factor AP-1 - physiology | NF-kappa B - physiology | Gene Expression Regulation, Neoplastic - radiation effects | Lymphoma, Large B-Cell, Diffuse - genetics
Journal Article
Canadian Journal of Physiology and Pharmacology, ISSN 0008-4212, 2014, Volume 92, Issue 2, pp. 132 - 139
Journal Article
Molecular cancer therapeutics, ISSN 1535-7163, 12/2014, Volume 13, Issue 12, pp. 3137 - 3151
Journal Article
Nucleic acids research, ISSN 1362-4962, 2017, Volume 45, Issue 19, pp. 11249 - 11267
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 7, p. e21796
.... Methodology/Principal Findings: We provide new evidence showing that overexpression of heterodimeric AP-1 comprised of c-jun and c-fos (AP-1(c-Jun/c-Fos... 
TGF-BETA | FGF | BIOLOGY | FIBROBLAST-GROWTH-FACTOR | CELL-PROLIFERATION | BMP-4 | SPECIFICATION | EARLY XENOPUS DEVELOPMENT | TUMORIGENESIS | EMBRYOS | MESODERM INDUCTION | Embryo, Nonmammalian - cytology | Immunoprecipitation | Luciferases - metabolism | Xenopus Proteins - genetics | Transcription Factor AP-1 - genetics | Bone Morphogenetic Protein 4 - genetics | Protein Multimerization | Transcriptional Activation | Embryo, Nonmammalian - metabolism | Glycoproteins - metabolism | Activins - metabolism | Smad3 Protein - metabolism | Bone Morphogenetic Protein 4 - metabolism | Transcription Factor AP-1 - metabolism | Goosecoid Protein - genetics | Goosecoid Protein - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Smad3 Protein - genetics | In Situ Hybridization | Chromatin Immunoprecipitation | Glycoproteins - genetics | Promoter Regions, Genetic | Signal Transduction | Proto-Oncogene Proteins c-jun - genetics | RNA, Messenger - genetics | Xenopus laevis | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Proto-Oncogene Proteins c-fos - metabolism | Reverse Transcriptase Polymerase Chain Reaction | T-Box Domain Proteins - genetics | Blotting, Western | T-Box Domain Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Proto-Oncogene Proteins c-jun - metabolism | Organizers, Embryonic - metabolism | Protein Binding | Proto-Oncogene Proteins c-fos - genetics | Xenopus Proteins - metabolism | Activins - genetics | Physiological aspects | Gene expression | Bone morphogenetic proteins | Fibroblast growth factor | Transcription factors | Transcription | Genes | Gene regulation | Biochemistry | Smad3 protein | Reptiles & amphibians | Proteins | Embryogenesis | Physiology | Growth factors | c-Fos protein | Bone morphogenetic protein 4 | Cytokines | Activator protein 1 | c-Jun protein | Chordin | Noggin protein | Embryos | Mutants | Embryonic growth stage | Medicine | Signaling | Stem cells | Activin | Binding sites
Journal Article
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 4/2018, Volume 441, Issue 1, pp. 89 - 98
...MicroRNA-203 suppresses proliferation in liver cancer associated with PIK3CA, p38 MAPK, c-Jun, and GSK3 signaling Annie Zhang 1 • Jaganathan Lakshmanan 1... 
Life Sciences | Biochemistry, general | Liver cancer | c-Jun | PI3K | GSK3 | Medical Biochemistry | Oncology | MicroRNA-203 | p38-MAPK | Cardiology | PATHWAYS | ACTIVATION | METASTASIS | ANGIOGENESIS | CELL BIOLOGY | BREAST-CANCER | HEPATOCELLULAR-CARCINOMA | GROWTH | TARGET PREDICTION | TUMORIGENESIS | PROGRESSION | Cell Proliferation | Class I Phosphatidylinositol 3-Kinases - genetics | Class I Phosphatidylinositol 3-Kinases - metabolism | Liver Neoplasms - genetics | Proto-Oncogene Proteins c-jun - genetics | Humans | p38 Mitogen-Activated Protein Kinases - genetics | MicroRNAs - metabolism | RNA, Neoplasm - metabolism | Hep G2 Cells | MAP Kinase Signaling System | Proto-Oncogene Proteins c-jun - metabolism | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - metabolism | RNA, Neoplasm - genetics | Liver Neoplasms - pathology | MicroRNAs - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Glycogen Synthase Kinases - metabolism | Carcinoma, Hepatocellular - metabolism | Glycogen Synthase Kinases - genetics | MicroRNA | Synthesis | Glycogen | Genetic aspects | B cells | Gene expression | Health aspects | Protein kinases | Cancer | Cell proliferation | Transcription factors | Liver | Glycogen synthase kinase 3 | c-Jun protein | MAP kinase | Hepatocellular carcinoma | AKT protein | Phosphatidylinositol 4,5-diphosphate | Kinases | Ribonucleic acid--RNA | Western blotting | Signaling | Cell growth | Hepatocytes | MicroRNAs | miRNA | Non-coding RNA
Journal Article