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Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7692, pp. 373 - 377
Both microbial and host genetic factors contribute to the pathogenesis of autoimmune diseases(1-4). There is accumulating evidence that microbial species that... 
INTESTINAL INFLAMMATION | RESPONSES | IMMUNITY | MULTIDISCIPLINARY SCIENCES | IN-VIVO | DISEASE | HUMAN MICROBIOTA | MICE | DIFFERENTIATION | EXPRESSION | ANTIGEN | Bioengineering | T-Lymphocytes, Regulatory - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - antagonists & inhibitors | Homeostasis | Colitis - pathology | Male | Intestines - immunology | T-Lymphocytes, Regulatory - immunology | Forkhead Transcription Factors - metabolism | Helicobacter hepaticus - pathogenicity | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | T-Lymphocytes, Regulatory - cytology | Female | Th17 Cells - cytology | Colitis - immunology | Helicobacter hepaticus - genetics | Interleukin-10 - deficiency | Helicobacter hepaticus - immunology | Immune Tolerance | Host-Pathogen Interactions | Animals | Colitis - microbiology | Intestines - microbiology | Proto-Oncogene Proteins c-maf - deficiency | Th17 Cells - immunology | Interleukin-10 - biosynthesis | Proto-Oncogene Proteins c-maf - metabolism | Mice | Interleukin-10 - immunology | Host-bacteria relationships | Inflammatory bowel diseases | Helicobacter infections | Physiological research | Research | T cells | Immunological tolerance | Pathogenesis | Helper cells | Lymphocytes T | Inactivation | Small intestine | Accumulation | Microbiota | Microorganisms | Immunology | Lymphocytes | Rodents | Bacteria | Foxp3 protein | Genetic factors | Deactivation | Immunoregulation | T cell receptors | c-Maf protein | Inflammatory bowel disease | Large intestine | Interleukin 10 | Colitis | Autoimmune diseases | Differentiation | Colonization
Journal Article
The Journal of Immunology, ISSN 0022-1767, 05/2009, Volume 182, Issue 10, pp. 6226 - 6236
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 9, pp. 1857 - 1874
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2014, Volume 289, Issue 33, pp. 22980 - 22990
Programmed cell death protein 4 (PDCD4) is a tumor suppressor and has also been shown to suppress production of the immunomodulatory cytokine IL-10. The... 
MAMMALIAN TARGET | RAPAMYCIN | INNATE INFLAMMATORY RESPONSE | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUPPRESSOR PDCD4 | LIPOPOLYSACCHARIDE | KINASE-AKT | TRANSLATION | EXPRESSION | IL-10 | RNA-Binding Proteins - genetics | Transcription, Genetic - drug effects | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Proteolysis - drug effects | TOR Serine-Threonine Kinases - genetics | Apoptosis Regulatory Proteins - genetics | Repressor Proteins - metabolism | Cell Line | Proto-Oncogene Proteins c-maf - genetics | Gene Expression Regulation - physiology | Repressor Proteins - genetics | Macrophages - cytology | Apoptosis Regulatory Proteins - metabolism | Gene Expression Regulation - drug effects | Phosphatidylinositol 3-Kinases - genetics | Macrophages - metabolism | Transcription, Genetic - physiology | Promoter Regions, Genetic - physiology | Interleukin-10 - genetics | Lipopolysaccharides - pharmacology | Twist-Related Protein 1 - genetics | Interleukin-10 - biosynthesis | Proto-Oncogene Proteins c-maf - metabolism | Apoptosis - physiology | Twist-Related Protein 1 - metabolism | RNA-Binding Proteins - metabolism | c-maf | mTOR Complex (mTORC) | Immunology | Interleukin | PDCD4 | Inflammation | Rapamycin | Transcription Factor | Twist2 | Lipopolysaccharide (LPS)
Journal Article
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2012, Volume 122, Issue 9, pp. 3281 - 3294
CD4 T follicular helper (TFH) cells interact with and stimulate the generation of antigen-specific B cells. TFH cell interaction with B cells correlates with... 
BINDING PROTEIN-ALPHA | MEDICINE, RESEARCH & EXPERIMENTAL | GERMINAL CENTER B | C/EBP-ALPHA | ACTIVATION | COLONY-STIMULATING FACTOR | PROLIFERATION ARREST | IMMUNE-RESPONSES | IMMUNODEFICIENCY VIRUS-INFECTION | RECEPTOR | DIFFERENTIATION | Cell Proliferation | Lymph Nodes - pathology | Oligonucleotide Array Sequence Analysis | Transcriptome | Macaca mulatta | Antibodies, Viral - blood | T-Lymphocytes, Helper-Inducer - immunology | Cell Death | Receptors, Lysosphingolipid - genetics | Receptors, Lysosphingolipid - metabolism | Simian Acquired Immunodeficiency Syndrome - pathology | B-Lymphocytes - metabolism | Simian Immunodeficiency Virus - immunology | Gene Expression | T-Lymphocytes, Helper-Inducer - metabolism | Cytokines - metabolism | Proto-Oncogene Proteins c-maf - genetics | Lymph Nodes - metabolism | Lymph Nodes - virology | Germinal Center - pathology | Transcription Factors - genetics | Simian Acquired Immunodeficiency Syndrome - blood | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Simian Immunodeficiency Virus - physiology | Host-Pathogen Interactions | Transcription Factors - metabolism | Animals | B-Lymphocytes - immunology | Simian Acquired Immunodeficiency Syndrome - immunology | Germinal Center - virology | Proto-Oncogene Proteins c-maf - metabolism | CD4 Antigens - metabolism | Genetic aspects | Research | Simian immunodeficiency virus | Gene expression | CD4 lymphocytes
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2012, Volume 188, Issue 8, pp. 3734 - 3744
Journal Article