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Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 6/2011, Volume 108, Issue 24, pp. 9951 - 9956
The tyrosine kinase c-Met promotes the formation and malignant progression of multiple cancers... 
Phenotypes | Cell growth | Hepatocytes | Somatic cells | Induced pluripotent stem cells | Neurons | Stem cells | Glioblastoma | Neural stem cells | Tumors | Cancer stem cell | Oct4 | Sox2 | Klf4 | Hepatocyte growth factor | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Proto-Oncogene Proteins c-met - metabolism | Homeodomain Proteins - metabolism | Humans | Glycoproteins - metabolism | Peptides - genetics | Immunoblotting | Transplantation, Heterologous | Antigens, CD - genetics | Antigens, CD - metabolism | Cellular Reprogramming | SOXB1 Transcription Factors - metabolism | Neoplasms, Experimental - pathology | Octamer Transcription Factor-3 - genetics | Flow Cytometry | Peptides - metabolism | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | RNA Interference | SOXB1 Transcription Factors - genetics | Neoplasms, Experimental - genetics | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Indoles - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Glycoproteins - genetics | Nanog Homeobox Protein | Signal Transduction | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Sulfonamides - pharmacology | AC133 Antigen | Piperazines - pharmacology | Proto-Oncogene Proteins c-myc - metabolism | Homeodomain Proteins - genetics | Proto-Oncogene Proteins c-met - genetics | Phenotype | Animals | Glioblastoma - pathology | Octamer Transcription Factor-3 - metabolism | Mice | Proto-Oncogene Proteins c-myc - genetics | Neoplasms, Experimental - metabolism | Physiological aspects | Development and progression | Cellular signal transduction | Genetic aspects | Research | Glioblastoma multiforme | Protein kinases | Transcription factors | Tumor cells | DNA damage | Embryo cells | Tumorigenicity | neurospheres | Differentiation | Protein-tyrosine kinase | glioblastoma cells | Cancer | Index Medicus | Biological Sciences | cancer stem cell | hepatocyte growth factor
Journal Article
Journal Article
Molecular oncology, ISSN 1574-7891, 01/2015, Volume 9, Issue 1, pp. 323 - 333
Journal Article
PloS one, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, pp. e0128159 - e0128159
Background c-Met, a high-affinity receptor for Hepatocyte Growth Factor (HGF), plays a critical role in tumor growth, invasion, and metastasis... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Proto-Oncogene Proteins c-met - metabolism | Receptor, Epidermal Growth Factor - genetics | Receptor, ErbB-3 - metabolism | Humans | Male | Sulfones - pharmacology | Receptor, Epidermal Growth Factor - metabolism | Carcinoma, Hepatocellular - drug therapy | Hepatocyte Growth Factor - genetics | Female | Indoles - pharmacology | Liver Neoplasms - pathology | Cell Survival - physiology | Cell Survival - drug effects | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Liver Neoplasms - drug therapy | Receptor, ErbB-3 - genetics | Hepatocyte Growth Factor - metabolism | Proto-Oncogene Proteins c-met - genetics | Signal Transduction - drug effects | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Signal Transduction - physiology | Carcinoma, Hepatocellular - metabolism | Pediatrics | Profiling | Transplants & implants | Laboratories | Lung cancer | Clinical trials | AKT protein | Oncology | Hepatocellular carcinoma | Metastasis | Kinases | Genetic screening | Metastases | Puromycin | c-Met protein | Proteins | Liver cancer | Signal transduction | Negative feedback | Epidermal growth factor | Rodents | Penicillin | Protein-tyrosine kinase receptors | Inhibition | Gefitinib | Protein-tyrosine kinase | Growth kinetics | Tyrosine | Medical research | Cell survival | Epidermal growth factor receptors | Hepatocyte growth factor | Cloning | siRNA | ErbB-3 protein | Gene expression | AC generators | Survival | 1-Phosphatidylinositol 3-kinase | Medicine | Signaling | Inhibitors | Medical prognosis | Kinetics | Tumors | Apoptosis | Index Medicus
Journal Article
Cancer research (Chicago, Ill.), ISSN 0008-5472, 03/2018, Volume 78, Issue 6, pp. 1418 - 1430
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 3/2006, Volume 103, Issue 11, pp. 4046 - 4051
Journal Article
Clinical cancer research, ISSN 1557-3265, 01/2013, Volume 19, Issue 7, pp. 1773 - 1783
Journal Article
Journal Article
Nature genetics, ISSN 1546-1718, 07/2012, Volume 44, Issue 8, pp. 852 - 860
... p.Thr790Met alteration or MET activation. Genetic or pharmacological inhibition of AXL restored sensitivity to erlotinib in these tumor models... 
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Erlotinib Hydrochloride | Proto-Oncogene Proteins c-met - metabolism | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Humans | Lung Neoplasms - metabolism | Middle Aged | Lung Neoplasms - pathology | Male | Intercellular Signaling Peptides and Proteins - metabolism | Epithelial-Mesenchymal Transition | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Adult | Female | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Proto-Oncogene Proteins - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Proto-Oncogene Proteins c-met - genetics | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | Receptor Protein-Tyrosine Kinases - genetics | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Aged | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Enzyme Activation | Mutation | Quinazolines - pharmacology | Erlotinib | Physiological aspects | Genetic aspects | Research | Lung cancer, Non-small cell | Drug therapy | Health aspects | Protein kinases | Studies | Medical research | Rodents | Lung cancer | Genomics | Kinases | Gene expression | Acquisitions & mergers | Tumors | Index Medicus
Journal Article