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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 24, pp. 9951 - 9956
The tyrosine kinase c-Met promotes the formation and malignant progression of multiple cancers. It is well known that c-Met hyperactivation increases... 
Phenotypes | Cell growth | Hepatocytes | Somatic cells | Induced pluripotent stem cells | Neurons | Stem cells | Glioblastoma | Neural stem cells | Tumors | Cancer stem cell | Oct4 | Sox2 | Klf4 | Hepatocyte growth factor | CANCER-CELLS | MULTIDISCIPLINARY SCIENCES | FACTOR EXPRESSION | PROLIFERATION | TUMORIGENICITY | TUMOR-INITIATING CELLS | P53 | cancer stem cell | PATHWAY | hepatocyte growth factor | IN-VIVO | GROWTH-FACTOR | DIFFERENTIATION | Proto-Oncogene Proteins c-met - metabolism | Homeodomain Proteins - metabolism | Humans | Glycoproteins - metabolism | Peptides - genetics | Immunoblotting | Transplantation, Heterologous | Antigens, CD - genetics | Antigens, CD - metabolism | Cellular Reprogramming | SOXB1 Transcription Factors - metabolism | Neoplasms, Experimental - pathology | Octamer Transcription Factor-3 - genetics | Flow Cytometry | Peptides - metabolism | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | RNA Interference | SOXB1 Transcription Factors - genetics | Neoplasms, Experimental - genetics | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Indoles - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Glycoproteins - genetics | Nanog Homeobox Protein | Signal Transduction | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Sulfonamides - pharmacology | AC133 Antigen | Piperazines - pharmacology | Proto-Oncogene Proteins c-myc - metabolism | Homeodomain Proteins - genetics | Proto-Oncogene Proteins c-met - genetics | Phenotype | Animals | Glioblastoma - pathology | Octamer Transcription Factor-3 - metabolism | Mice | Proto-Oncogene Proteins c-myc - genetics | Neoplasms, Experimental - metabolism | Physiological aspects | Development and progression | Cellular signal transduction | Genetic aspects | Research | Glioblastoma multiforme | Protein kinases | Proteins | Signal transduction | Genotype & phenotype | Kinases | Gene expression | Cancer | Transcription factors | Tumor cells | DNA damage | Embryo cells | Tumorigenicity | neurospheres | Differentiation | Protein-tyrosine kinase | glioblastoma cells | Index Medicus | Biological Sciences
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 06/2010, Volume 70, Issue 12, pp. 5184 - 5193
MicroRNAs (miRNA) have rapidly emerged as modulators of gene expression in cancer in which they may have great diagnostic and therapeutic import.... 
MIGRATION | INVASIVE GROWTH | INHIBITION | METASTASIS | THERAPY | ONCOLOGY | DOWN-REGULATION | HUMAN HEPATOCELLULAR-CARCINOMA | IDENTIFICATION | MICRORNA EXPRESSION | CANCER | Proto-Oncogene Proteins c-met - metabolism | Luciferases - metabolism | Antibiotics, Antineoplastic - pharmacology | Apoptosis - drug effects | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Male | Intracellular Signaling Peptides and Proteins - metabolism | RNA, Messenger - metabolism | Carcinoma, Hepatocellular - drug therapy | Carcinoma, Hepatocellular - genetics | Aged, 80 and over | Female | Liver Neoplasms - pathology | Intracellular Signaling Peptides and Proteins - genetics | Liver Cirrhosis - genetics | Protein-Serine-Threonine Kinases - metabolism | Liver Cirrhosis - drug therapy | Liver Neoplasms - genetics | RNA, Messenger - genetics | Liver Neoplasms - drug therapy | Protein-Serine-Threonine Kinases - genetics | Cell Adhesion - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Proto-Oncogene Proteins c-met - genetics | Cell Movement - drug effects | Carcinoma, Hepatocellular - pathology | Liver Cirrhosis - pathology | Aged | Cell Proliferation - drug effects | TOR Serine-Threonine Kinases | MicroRNAs - physiology | Cell Cycle - drug effects | Doxorubicin - pharmacology | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 12/2010, Volume 70, Issue 24, pp. 10090 - 10100
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 6, pp. 2218 - 2227.e5
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2008, Volume 105, Issue 2, pp. 692 - 697
Journal Article
Cell Reports, ISSN 2211-1247, 05/2016, Volume 15, Issue 6, pp. 1175 - 1189
Journal Article
Nature Genetics, ISSN 1061-4036, 08/2012, Volume 44, Issue 8, pp. 852 - 860
Human non-small cell lung cancers (NSCLCs) with activating mutations in EGFR frequently respond to treatment with EGFRtargeted tyrosine kinase inhibitors... 
GEFITINIB | RECEPTOR TYROSINE KINASES | MET AMPLIFICATION | GROWTH | GENETICS & HEREDITY | ACQUIRED-RESISTANCE | MUTATIONS | NF-KAPPA-B | TUMORS | MUTANT EGFR | ERLOTINIB | Erlotinib Hydrochloride | Proto-Oncogene Proteins c-met - metabolism | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Humans | Lung Neoplasms - metabolism | Middle Aged | Lung Neoplasms - pathology | Male | Intercellular Signaling Peptides and Proteins - metabolism | Epithelial-Mesenchymal Transition | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Adult | Female | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Proto-Oncogene Proteins - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Proto-Oncogene Proteins c-met - genetics | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | Receptor Protein-Tyrosine Kinases - genetics | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Aged | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Enzyme Activation | Mutation | Quinazolines - pharmacology | Erlotinib | Physiological aspects | Genetic aspects | Research | Lung cancer, Non-small cell | Drug therapy | Health aspects | Protein kinases | Studies | Medical research | Rodents | Lung cancer | Genomics | Kinases | Gene expression | Acquisitions & mergers | Tumors | Index Medicus
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 04/2011, Volume 103, Issue 8, pp. 645 - 661
Background Ionizing radiation (IR) is effectively used in cancer therapy. However, in subsets of patients, a few radioresistant cancer cells survive and cause... 
STEM-CELLS | IN-VITRO | ONCOLOGY | TYROSINE KINASE | HGF | CELL INVASION | RECEPTOR | EPITHELIAL-MESENCHYMAL TRANSITIONS | NF-KAPPA-B | SCATTER-FACTOR | PROTOONCOGENE | Humans | Neoplasm Invasiveness - prevention & control | Radiation Tolerance | Receptors, Growth Factor - antagonists & inhibitors | NF-kappa B - metabolism | RNA, Messenger - metabolism | NF-kappa B - radiation effects | Receptors, Growth Factor - genetics | Proto-Oncogene Proteins c-met - radiation effects | Signal Transduction - radiation effects | Protein-Serine-Threonine Kinases - metabolism | Radiation, Ionizing | Tumor Suppressor Proteins - metabolism | DNA-Binding Proteins - radiation effects | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Cell Cycle Proteins - metabolism | Cell Cycle Proteins - radiation effects | Receptors, Growth Factor - radiation effects | Proto-Oncogene Proteins c-met - drug effects | Ataxia Telangiectasia Mutated Proteins | Phosphorylation - radiation effects | Cell Line, Tumor | Mice | RNA, Small Interfering | Tumor Suppressor Proteins - radiation effects | Receptors, Growth Factor - metabolism | Neoplasms - metabolism | Proto-Oncogene Proteins c-met - metabolism | Apoptosis - radiation effects | Up-Regulation - radiation effects | Transcription, Genetic - radiation effects | Transplantation, Heterologous | Sulfones - pharmacology | DNA-Binding Proteins - metabolism | Chromatin Immunoprecipitation | Protein-Serine-Threonine Kinases - radiation effects | Tumor Suppressor Proteins - genetics | Polymerase Chain Reaction | Cell Cycle Proteins - genetics | Indoles - pharmacology | Neoplasms - radiotherapy | Gene Expression Regulation, Neoplastic - drug effects | In Situ Nick-End Labeling | Enzyme-Linked Immunosorbent Assay | Radiation-Sensitizing Agents - pharmacology | Gene Silencing | Protein-Serine-Threonine Kinases - genetics | DNA-Binding Proteins - genetics | Cell Survival - radiation effects | Cell Movement - radiation effects | Proto-Oncogene Proteins c-met - genetics | Blotting, Northern | Animals | NF-kappa B - genetics | Protein Kinase Inhibitors - pharmacology | Neoplasms - pathology | Receptors, Growth Factor - drug effects | DNA Damage - radiation effects | Mitogen-Activated Protein Kinases - metabolism | Ionizing radiation | Physiological aspects | Genetic aspects | Cancer invasiveness | Research | Gene expression | Radiotherapy | Health aspects | Oncology | Radiation therapy | Kinases | Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 10/2017, Volume 47, Issue 4, pp. 789 - 802.e9
Inhibitors of the receptor tyrosine kinase c-MET are currently used in the clinic to target oncogenic signaling in tumor cells. We found that concomitant c-MET... 
HGF | lymph node | cancer immunotherapy | plasticity | melanoma | c-MET | T cells | neutrophils | bone marrow | therapy resistance | MIGRATION | IMMUNE-RESPONSE | LUNG | ACTIVATION | T-CELL RESPONSES | INHIBITION | HEPATOCYTE GROWTH-FACTOR | LYMPH-NODES | IMMUNOLOGY | TUMOR-ASSOCIATED NEUTROPHILS | G-CSF | Proto-Oncogene Proteins c-met - metabolism | Immunotherapy - methods | Interferon-gamma - metabolism | Signal Transduction - immunology | Proto-Oncogene Proteins c-met - immunology | Tumor Microenvironment - genetics | T-Lymphocytes - metabolism | Neoplasms, Experimental - immunology | Neutrophils - metabolism | Cell Proliferation - genetics | Mice, Inbred C57BL | Kaplan-Meier Estimate | Neoplasms, Experimental - therapy | Neutrophils - immunology | Gene Expression Profiling - methods | Gene Expression Regulation, Neoplastic - immunology | Mice, Transgenic | Signal Transduction - genetics | Mice, Knockout | Proto-Oncogene Proteins c-met - genetics | Tumor Microenvironment - immunology | Animals | Interferon-gamma - immunology | Cell Line, Tumor | T-Lymphocytes - immunology | Mice, Inbred BALB C | Neoplasms, Experimental - metabolism | Tyrosine | Care and treatment | Analysis | Immunotherapy | Phenols | Cancer | Animal models | Phosphorylation | Cytotoxicity | Lymphocytes T | Kinases | Lymph nodes | Metastases | Recruitment | c-Met protein | Vitiligo | Lymphocytes | Protein-tyrosine kinase receptors | Inhibition | Protein-tyrosine kinase | Antigens | Lymphatic system | Tumor cells | Neurons | Neutrophils | Melanoma | Leukocytes (neutrophilic) | Inflammation | Gene expression | Drainage | Serum levels | Signaling | Immunosuppression | Immune checkpoint | Infiltration | Microenvironments | Tumors | Index Medicus
Journal Article