X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (4295) 4295
Publication (603) 603
Book Review (533) 533
Book Chapter (24) 24
Conference Proceeding (9) 9
Dissertation (7) 7
Book / eBook (3) 3
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (4183) 4183
humans (3360) 3360
animals (2396) 2396
mice (1498) 1498
oncology (1372) 1372
cell line, tumor (1311) 1311
apoptosis (1288) 1288
cell biology (1240) 1240
proto-oncogene proteins c-myc - metabolism (1181) 1181
cancer (1004) 1004
c-myc (997) 997
biochemistry & molecular biology (963) 963
expression (930) 930
proto-oncogene proteins c-myc - genetics (868) 868
signal transduction (833) 833
female (800) 800
apoptosis - drug effects (763) 763
proteins (701) 701
male (695) 695
phosphorylation (694) 694
cell proliferation - drug effects (597) 597
gene expression (589) 589
proto-oncogene proteins c-akt - metabolism (585) 585
cell proliferation (559) 559
activation (543) 543
research (530) 530
antineoplastic agents - pharmacology (525) 525
signal transduction - drug effects (517) 517
article (467) 467
cells, cultured (444) 444
proto-oncogene proteins - metabolism (437) 437
gene expression regulation, neoplastic (435) 435
rats (421) 421
cell line (414) 414
cell cycle (403) 403
proto-oncogene proteins c-myc - antagonists & inhibitors (389) 389
tumor cells, cultured (384) 384
genetic aspects (380) 380
down-regulation (377) 377
proto-oncogene proteins - genetics (374) 374
gene-expression (373) 373
proliferation (373) 373
proto-oncogene proteins c-bcl-2 - metabolism (364) 364
growth (362) 362
tumors (361) 361
kinases (360) 360
blotting, western (351) 351
cells (333) 333
rna interference (329) 329
analysis (321) 321
transfection (318) 318
phosphatidylinositol 3-kinases - metabolism (317) 317
physiological aspects (314) 314
mutation (312) 312
health aspects (308) 308
inhibition (306) 306
multidisciplinary sciences (304) 304
in-vivo (300) 300
mice, nude (299) 299
enzyme inhibitors - pharmacology (295) 295
protein (294) 294
research article (291) 291
xenograft model antitumor assays (286) 286
hematology (284) 284
gene (283) 283
protein kinase inhibitors - pharmacology (279) 279
genetics & heredity (278) 278
pathway (272) 272
transcription factors (270) 270
differentiation (265) 265
rna, messenger - metabolism (263) 263
myc (261) 261
reverse transcriptase polymerase chain reaction (256) 256
cell survival - drug effects (255) 255
tumor suppressor protein p53 - metabolism (254) 254
cell growth (252) 252
transcription (252) 252
transcription factors - metabolism (252) 252
gene expression regulation, neoplastic - drug effects (250) 250
rna, messenger - genetics (249) 249
genes (240) 240
akt (238) 238
care and treatment (238) 238
biology (237) 237
p53 (232) 232
proto-oncogene proteins c-bcl-2 - genetics (229) 229
gene expression regulation (227) 227
leukemia (225) 225
dose-response relationship, drug (224) 224
medicine (222) 222
protein binding (216) 216
breast cancer (214) 214
in-vitro (212) 212
nuclear proteins - metabolism (211) 211
phosphatidylinositol 3-kinases - antagonists & inhibitors (210) 210
proto-oncogene proteins c-akt - antagonists & inhibitors (207) 207
time factors (207) 207
apoptosis - genetics (206) 206
apoptosis - physiology (206) 206
nf-kappa-b (206) 206
more...
Library Location Library Location
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Cell, ISSN 0092-8674, 2011, Volume 146, Issue 6, pp. 904 - 917
Journal Article
FEBS Journal, ISSN 1742-464X, 2014, Volume 281, Issue 3, pp. 802 - 813
Long non-coding RNAs (lncRNAs), a recently characterized class of non-coding RNAs, have been shown to have important regulatory roles and are de-regulated in a... 
C-Myc | Gastric carcinoma | Proliferation | Insulin-like growth factor 2 mRNA binding protein 1 | Long noncoding RNA | MIGRATION | MANAGEMENT | proliferation | c-Myc | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | insulin-like growth factor 2 mRNA binding protein 1 | EVOLUTION | GENE | BIOLOGY | IGF2BP1 | long non-coding RNA | gastric carcinoma | EXPRESSION | PROGRESSION | Neoplasm Transplantation | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Neoplasm Proteins - antagonists & inhibitors | Stomach - metabolism | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Stomach - surgery | RNA Interference | Biomarkers, Tumor - metabolism | Biomarkers, Tumor - antagonists & inhibitors | Neoplasm Proteins - genetics | Recombinant Proteins - metabolism | Carcinoma - surgery | Recombinant Proteins - chemistry | RNA, Long Noncoding - genetics | Tumor Burden | RNA Stability | Proto-Oncogene Proteins c-myc - metabolism | Animals | Mice, Nude | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Survival Analysis | Cell Line, Tumor | Biomarkers, Tumor - genetics | Mice | Proto-Oncogene Proteins c-myc - genetics | Carcinoma - metabolism | RNA, Long Noncoding - antagonists & inhibitors | Stomach Neoplasms - surgery | RNA, Long Noncoding - metabolism | RNA-Binding Proteins - metabolism | Messenger RNA | Carcinoma | Analysis | Physiological aspects | Stomach cancer | Protein binding | Cancer | Cellular biology | Ribonucleic acid--RNA | Medical treatment | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 06/2016, Volume 534, Issue 7607, pp. 341 - 346
Chronic myeloid leukaemia (CML) arises after transformation of a haemopoietic stem cell (HSC) by the protein-tyrosine kinase BCR-ABL. Direct inhibition of... 
CD34(+) CELLS | INHIBITION | MICROARRAY | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | IMATINIB | TUMOR | COMBINATION | CANCER | EXPRESSION | CHRONIC MYELOID-LEUKEMIA | Antigens, CD34 - metabolism | Tumor Suppressor Protein p53 - antagonists & inhibitors | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Transcriptome | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Acetamides - pharmacology | Male | Neoplasm Proteins - metabolism | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Imatinib Mesylate - therapeutic use | Acetamides - therapeutic use | Neoplastic Stem Cells - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Imidazolines - therapeutic use | Neoplastic Stem Cells - pathology | Female | Cell Death - drug effects | Hematopoietic Stem Cells - drug effects | Reproducibility of Results | Imatinib Mesylate - pharmacology | Tumor Suppressor Protein p53 - metabolism | Imidazolines - pharmacology | Hematopoietic Stem Cells - metabolism | Tumor Suppressor Protein p53 - deficiency | Proto-Oncogene Proteins c-myc - metabolism | Neoplastic Stem Cells - transplantation | Azepines - therapeutic use | Azepines - pharmacology | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Hematopoietic Stem Cells - cytology | Proto-Oncogene Proteins c-myc - deficiency | Proteomics | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Proto-Oncogene Proteins c-myc - genetics | Fusion Proteins, bcr-abl - metabolism | Molecular targeted therapy | Cancer cells | Stem cells | Development and progression | Genetic aspects | Chronic myeloid leukemia | Tumor proteins | Properties | Health aspects | Methods | Proteins | Transcription factors | Leukemia | Cell cycle | Kinases | Cancer therapies | Apoptosis | Index Medicus
Journal Article
Journal of Cancer Research and Clinical Oncology, ISSN 0171-5216, 3/2013, Volume 139, Issue 3, pp. 437 - 445
Journal Article
Genes and Development, ISSN 0890-9369, 05/2013, Volume 27, Issue 10, pp. 1101 - 1114
Tumorigenesis results from dysregulation of oncogenes and tumor suppressors that influence cellular proliferation, differentiation, apoptosis, and/or... 
Huwe1 | c-Myc | Mule | Ras | p21 | Miz1 | DNA-DAMAGE | KERATINOCYTE GROWTH | DEVELOPMENTAL BIOLOGY | CELL BIOLOGY | BASE EXCISION-REPAIR | HUWE1 UBIQUITIN LIGASE | NEGATIVE REGULATION | GENETICS & HEREDITY | ARF TUMOR-SUPPRESSOR | DIFFERENTIATION | MIZ-1 | HUMAN CANCER | Protein Inhibitors of Activated STAT - deficiency | Tetradecanoylphorbol Acetate - pharmacology | 9,10-Dimethyl-1,2-benzanthracene - pharmacology | Male | Protein Inhibitors of Activated STAT - metabolism | Cyclin-Dependent Kinase Inhibitor p15 - biosynthesis | Oncogene Protein p21(ras) - metabolism | Cyclin-Dependent Kinase Inhibitor p16 | Oncogene Protein p21(ras) - antagonists & inhibitors | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cell Transformation, Neoplastic - genetics | Cyclin-Dependent Kinase Inhibitor p15 - genetics | Nuclear Proteins - deficiency | Protein Inhibitors of Activated STAT - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Nuclear Proteins - genetics | Protein Inhibitors of Activated STAT - antagonists & inhibitors | Skin Neoplasms - pathology | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Signal Transduction | Down-Regulation | Cells, Cultured | Ubiquitin-Protein Ligases - metabolism | Nuclear Proteins - metabolism | Skin Neoplasms - chemically induced | Proto-Oncogene Proteins c-myc - metabolism | Mice, Knockout | Skin Neoplasms - metabolism | Keratinocytes - pathology | Animals | Tumor Suppressor Protein p53 | Keratinocytes - drug effects | Keratinocytes - metabolism | Nuclear Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - deficiency | Skin Neoplasms - genetics | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Ubiquitin-Protein Ligases - deficiency | Mice | Proto-Oncogene Proteins c-myc - genetics | Cyclin-Dependent Kinase Inhibitor p15 - metabolism | Genes, ras | Ubiquitin-Protein Ligases - genetics | Oncogene Protein p21(ras) - genetics | Carcinogenesis | Ras genes | Analysis | Index Medicus | Research Paper
Journal Article
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 04/2017, Volume 24, Issue 4, pp. 649 - 659
Journal Article
The EMBO Journal, ISSN 0261-4189, 07/2007, Volume 26, Issue 14, pp. 3332 - 3345
The c‐Myc oncoprotein promotes cell growth by enhancing ribosomal biogenesis through upregulation of RNA polymerases I‐, II‐, and III‐dependent transcription.... 
ribosomal proteins | transcription | cell cycle | TRRAP | L11 | C‐Myc | C-Myc | Ribosomal proteins | Transcription | Cell cycle | MYC/MAX/MAD NETWORK |