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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2014, Volume 111, Issue 52, pp. 18697 - 18702
Journal Article
Cell Metabolism, ISSN 1550-4131, 11/2013, Volume 18, Issue 5, pp. 726 - 739
Journal Article
FEBS Journal, ISSN 1742-464X, 2014, Volume 281, Issue 3, pp. 802 - 813
Long non-coding RNAs (lncRNAs), a recently characterized class of non-coding RNAs, have been shown to have important regulatory roles and are de-regulated in a... 
C-Myc | Gastric carcinoma | Proliferation | Insulin-like growth factor 2 mRNA binding protein 1 | Long noncoding RNA | MIGRATION | MANAGEMENT | proliferation | c-Myc | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | insulin-like growth factor 2 mRNA binding protein 1 | EVOLUTION | GENE | BIOLOGY | IGF2BP1 | long non-coding RNA | gastric carcinoma | EXPRESSION | PROGRESSION | Neoplasm Transplantation | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Neoplasm Proteins - antagonists & inhibitors | Stomach - metabolism | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Stomach - surgery | RNA Interference | Biomarkers, Tumor - metabolism | Biomarkers, Tumor - antagonists & inhibitors | Neoplasm Proteins - genetics | Recombinant Proteins - metabolism | Carcinoma - surgery | Recombinant Proteins - chemistry | RNA, Long Noncoding - genetics | Tumor Burden | RNA Stability | Proto-Oncogene Proteins c-myc - metabolism | Animals | Mice, Nude | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Survival Analysis | Cell Line, Tumor | Biomarkers, Tumor - genetics | Mice | Proto-Oncogene Proteins c-myc - genetics | Carcinoma - metabolism | RNA, Long Noncoding - antagonists & inhibitors | Stomach Neoplasms - surgery | RNA, Long Noncoding - metabolism | RNA-Binding Proteins - metabolism | Messenger RNA | Carcinoma | Analysis | Physiological aspects | Stomach cancer | Protein binding | Cancer | Cellular biology | Ribonucleic acid--RNA | Medical treatment | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 06/2016, Volume 534, Issue 7607, pp. 341 - 346
Chronic myeloid leukaemia (CML) arises after transformation of a haemopoietic stem cell (HSC) by the protein-tyrosine kinase BCR-ABL. Direct inhibition of... 
SURVIVAL | CD34(+) CELLS | INHIBITION | CHRONIC-PHASE | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | IMATINIB | TUMOR | CANCER | EXPRESSION | CHRONIC MYELOID-LEUKEMIA | Antigens, CD34 - metabolism | Tumor Suppressor Protein p53 - antagonists & inhibitors | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Transcriptome | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Acetamides - pharmacology | Male | Neoplasm Proteins - metabolism | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Imatinib Mesylate - therapeutic use | Acetamides - therapeutic use | Neoplastic Stem Cells - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Imidazolines - therapeutic use | Neoplastic Stem Cells - pathology | Female | Cell Death - drug effects | Hematopoietic Stem Cells - drug effects | Reproducibility of Results | Imatinib Mesylate - pharmacology | Tumor Suppressor Protein p53 - metabolism | Imidazolines - pharmacology | Hematopoietic Stem Cells - metabolism | Tumor Suppressor Protein p53 - deficiency | Proto-Oncogene Proteins c-myc - metabolism | Neoplastic Stem Cells - transplantation | Azepines - therapeutic use | Azepines - pharmacology | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Hematopoietic Stem Cells - cytology | Proto-Oncogene Proteins c-myc - deficiency | Proteomics | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Proto-Oncogene Proteins c-myc - genetics | Fusion Proteins, bcr-abl - metabolism | Molecular targeted therapy |