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Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 4, pp. 1314 - 1322.e5
Background & Aims Proton pump inhibitors (PPIs) and nonsteroidal anti-inflammatory drugs (NSAIDs) are among the most commonly used classes of drugs, with the... 
Gastroenterology and Hepatology | Ulcer | Microflora | Bleeding | Acid Secretion | ULCERS | NONSTEROIDAL ANTIINFLAMMATORY DRUG | PERMEABILITY | RATS | INDOMETHACIN | LEUKOCYTE ADHERENCE | INDUCED GASTRIC DAMAGE | PATHOGENESIS | NITRIC-OXIDE | BACTERIAL OVERGROWTH | GASTROENTEROLOGY & HEPATOLOGY | Jejunum - pathology | Rats, Wistar | Colon - drug effects | Hematocrit | Peptic Ulcer - prevention & control | Male | Omeprazole - toxicity | Bifidobacterium - isolation & purification | Bifidobacterium - growth & development | Actinobacteria - drug effects | Jejunum - drug effects | Drug Interactions | Peptic Ulcer - microbiology | Time Factors | Actinobacteria - genetics | Actinobacteria - isolation & purification | Disease Models, Animal | Bifidobacterium - genetics | Jejunum - microbiology | Rats | Celecoxib | Probiotics - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Proton Pump Inhibitors - toxicity | Anti-Inflammatory Agents, Non-Steroidal - toxicity | Peptic Ulcer - pathology | Denaturing Gradient Gel Electrophoresis | 2-Pyridinylmethylsulfinylbenzimidazoles - toxicity | Gastrointestinal Hemorrhage - chemically induced | Gastrointestinal Hemorrhage - prevention & control | Bifidobacterium - drug effects | Animals | Gastrointestinal Hemorrhage - microbiology | Actinobacteria - growth & development | Pyrazoles - toxicity | Anti-Inflammatory Agents, Non-Steroidal - pharmacokinetics | Naproxen - toxicity | Sulfonamides - toxicity | Colon - microbiology | Peptic Ulcer - chemically induced | Gastrointestinal Hemorrhage - pathology | Lansoprazole | Proton pump inhibitors | Messenger RNA | Nonsteroidal anti-inflammatory drugs | Index Medicus | Abridged Index Medicus
Journal Article
Chemical Research in Toxicology, ISSN 0893-228X, 03/2015, Volume 28, Issue 3, pp. 419 - 430
Journal Article
Giornale Italiano di Farmacia Clinica, ISSN 1120-3749, 01/2018, Volume 32, Issue 1, pp. 35 - 95
Journal Article
Journal Article
Dermatitis, ISSN 1710-3568, 11/2015, Volume 26, Issue 6, pp. 287 - 290
Few published reports have described occupational contact dermatitis from proton pump inhibitor (PPI) exposure in the literature. We present an additional case... 
OMEPRAZOLE | DERMATOLOGY | Dermatitis, Contact - etiology | Air Pollutants, Occupational - toxicity | Humans | Middle Aged | Eyelids | Male | Neck | Facial Dermatoses - chemically induced | Proton Pump Inhibitors - toxicity | Dermatitis, Occupational - etiology | Index Medicus
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 05/2015, Volume 308, Issue 9, pp. G785 - G793
Nonsteroidal anti-inflammatory drugs (NSAIDs) are widely used to treat a number of conditions, and proton pump inhibitors (PPIs) are often used to prevent... 
Omeprazole | Inflammation | Indomethacin | Intestinal motility | CONSTIPATION | omeprazole | PHYSIOLOGY | RISK-FACTORS | NSAID | INJURY | PROTON PUMP INHIBITORS | FACTOR-KAPPA-B | indomethacin | SMOOTH-MUSCLE | inflammation | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | intestinal motility | BACTERIAL OVERGROWTH | GASTROENTEROLOGY & HEPATOLOGY | MOTILITY | Ileum - pathology | Jejunum - pathology | Gastrointestinal Motility - drug effects | Ileum - metabolism | Male | Omeprazole - toxicity | Jejunum - metabolism | Jejunum - drug effects | Muscle, Smooth - drug effects | Ileum - drug effects | Jejunum - physiopathology | Inflammation Mediators - metabolism | Enteritis - chemically induced | Enteritis - metabolism | Biomarkers - metabolism | Gastrointestinal Hemorrhage - physiopathology | Jejunum - microbiology | Muscle, Smooth - metabolism | Proton Pump Inhibitors - toxicity | Rats, Sprague-Dawley | Anti-Inflammatory Agents, Non-Steroidal - toxicity | Enteritis - physiopathology | Gastrointestinal Hemorrhage - chemically induced | Animals | Muscle Contraction - drug effects | Muscle, Smooth - physiopathology | Ileum - physiopathology | Indomethacin - toxicity | Ileum - microbiology | Gastrointestinal Hemorrhage - pathology | Complications and side effects | Care and treatment | Dosage and administration | Diagnosis | Muscle contraction | Risk factors | Physiology | Inhibitor drugs | Nonsteroidal anti-inflammatory drugs | Drug therapy | Small intestine | Gastrointestinal diseases | Index Medicus | Neuroregulation and Motility
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 11/2017, Volume 43, Issue 3, pp. 1037 - 1051
Journal Article
Toxicology Letters, ISSN 0378-4274, 05/2015, Volume 235, Issue 1, pp. 28 - 36
The role of the aryl hydrocarbon receptor (AhR) in hemostasis has recently gained increased attention. Here, we demonstrate, by qRT-PCR and western blot, that... 
cPLA2 | Platelet signaling | AhR non-genomic pathway | p38MAPK | Platelet priming | CPLA 2 | P38MAPK | DIOXIN | PROTON PUMP INHIBITORS | cPLA | P38 MAP KINASE | CLOPIDOGREL | MICE | TOXICOLOGY | ACTIVATED PROTEIN-KINASES | ANTIPLATELET ACTION | NONGENOMIC PATHWAY | EXPRESSION | AGGREGATION | Up-Regulation | Phosphorylation | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Humans | Peptide Fragments - pharmacology | Omeprazole - toxicity | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Receptors, Thrombin - agonists | Basic Helix-Loop-Helix Transcription Factors - metabolism | Group IV Phospholipases A2 - metabolism | Time Factors | Puromycin - pharmacology | Receptors, Aryl Hydrocarbon - metabolism | Platelet Aggregation - drug effects | p38 Mitogen-Activated Protein Kinases - metabolism | Blood Platelets - drug effects | Basic Helix-Loop-Helix Transcription Factors - agonists | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptors, Aryl Hydrocarbon - genetics | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Proton Pump Inhibitors - toxicity | Receptors, Aryl Hydrocarbon - agonists | Signal Transduction - drug effects | Blood Platelets - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Polychlorinated Dibenzodioxins - toxicity | Receptors, Thrombin - metabolism | Ligands | Platelet Activation - drug effects | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Omeprazole | Physiological aspects | Herbicides | Analysis | Dioxin | Cells | Index Medicus | Proteins | Receptors | Pathways | Aromatic compounds | Hydrocarbons | Priming | Platelets
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 2014, Volume 307, Issue 3, pp. G390 - G396
Journal Article
Toxicologic Pathology, ISSN 0192-6233, 7/2008, Volume 36, Issue 5, pp. 727 - 737
Journal Article
Journal Article
Clinical Toxicology, ISSN 1556-3650, 07/2011, Volume 49, Issue 6, pp. 457 - 563
Journal Article
Cardiovascular Therapeutics, ISSN 1755-5914, 04/2015, Volume 33, Issue 2, pp. 27 - 34
BackgroundThe safety of all proton pump inhibitors (PPIs) in patients with intrinsic cardiac disease has not been well studied. In the present study, the... 
Ventricular arrhythmias | Proton pump inhibitors | Pantoprazole | Ischemia/reperfusion | LANSOPRAZOLE | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-ISCHEMIA | OMEPRAZOLE | CORONARY-ARTERY | ANTIPLATELET THERAPY | RESPONSES | Ischemia | H+/K+-ATPASE | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | POTASSIUM | reperfusion | Proton Pump Inhibitors - pharmacology | Arrhythmias, Cardiac - chemically induced | Rats, Wistar | Myocardial Reperfusion Injury - complications | Anti-Arrhythmia Agents - pharmacology | Tachycardia, Ventricular - chemically induced | Male | 2-Pyridinylmethylsulfinylbenzimidazoles - pharmacology | Arrhythmias, Cardiac - physiopathology | Time Factors | Ventricular Premature Complexes - prevention & control | Ventricular Premature Complexes - physiopathology | Myocardial Ischemia - physiopathology | Ventricular Fibrillation - physiopathology | Myocardial Reperfusion Injury - blood | Disease Models, Animal | Ventricular Fibrillation - prevention & control | Arrhythmias, Cardiac - prevention & control | Nitric Oxide - blood | Anti-Arrhythmia Agents - toxicity | Myocardial Ischemia - blood | Heart Conduction System - drug effects | Tachycardia, Ventricular - prevention & control | Proton Pump Inhibitors - toxicity | Myocardial Reperfusion Injury - physiopathology | Ventricular Premature Complexes - chemically induced | 2-Pyridinylmethylsulfinylbenzimidazoles - toxicity | Tachycardia, Ventricular - physiopathology | Animals | Heart Conduction System - physiopathology | L-Lactate Dehydrogenase - blood | Myocardial Ischemia - complications | Ventricular Fibrillation - chemically induced | Hyperkalemia - chemically induced | Arrhythmias, Cardiac - blood | Arrhythmia | Nitric oxide | Mortality | Anti-arrhythmia drugs | Index Medicus
Journal Article