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by Paternoster, Lavinia and Standl, Marie and Chen, Chih-Mei and Ramasamy, Adaikalavan and Bønnelykke, Klaus and Duijts, Liesbeth and Ferreira, Manuel A and Alves, Alexessander Couto and Thyssen, Jacob P and Albrecht, Eva and Baurecht, Hansjörg and Feenstra, Bjarke and Sleiman, Patrick M A and Hysi, Pirro and Warrington, Nicole M and Curjuric, Ivan and Myhre, Ronny and Curtin, John A and Groen-Blokhuis, Maria M and Kerkhof, Marjan and Sääf, Annika and Franke, Andre and Ellinghaus, David and Fölster-Holst, Regina and Dermitzakis, Emmanouil and Montgomery, Stephen B and Prokisch, Holger and Heim, Katharina and Hartikainen, Anna-Liisa and Pouta, Anneli and Pekkanen, Juha and Blakemore, Alexandra I F and Buxton, Jessica L and Kaakinen, Marika and Duffy, David L and Madden, Pamela A and Heath, Andrew C and Montgomery, Grant W and Thompson, Philip J and Matheson, Melanie C and Le Souëf, Peter and Pourcain, Beate St and Smith, George Davey and Henderson, John and Kemp, John P and Timpson, Nicholas J and Deloukas, Panos and Ring, Susan M and Wichmann, H-Erich and Müller-Nurasyid, Martina and Novak, Natalija and Klopp, Norman and Rodríguez, Elke and McArdle, Wendy and Linneberg, Allan and Menné, Torkil and Nohr, Ellen A and Hofman, Albert and Uitterlinden, André G and van Duijn, Cornélia M and Rivadeneira, Fernando and de Jongste, Johan C and van der Valk, Ralf J P and Wjst, Matthias and Jogi, Rain and Geller, Frank and Boyd, Heather A and Murray, Jeffrey C and Kim, Cecilia and Mentch, Frank and March, Michael and Mangino, Massimo and Spector, Tim D and Bataille, Veronique and Pennell, Craig E and Holt, Patrick G and Sly, Peter and Tiesler, Carla M T and Thiering, Elisabeth and Illig, Thomas and Imboden, Medea and Nystad, Wenche and Simpson, Angela and Hottenga, Jouke-Jan and Postma, Dirkje and Koppelman, Gerard H and Smit, Henriette A and Söderhäll, Cilla and Chawes, Bo and Kreiner-Møller, Eskil and Bisgaard, Hans and Melén, Erik and Boomsma, Dorret I and Custovic, Adnan and Jacobsson, Bo and Probst-Hensch, Nicole M and Palmer, Lyle J and Glass, Daniel and Hakonarson, Hakon and Melbye, Mads and ... and The Genetics of Overweight Young Adults (GOYA) Consortium and Australian Asthma Genetics Consortium (AAGC) and the EArly Genetics & Lifecourse Epidemiology (EAGLE) Consortium and Australian Asthma Genetics Consort and EArly Genetics Lifecourse Epidemio and Genetics Overweight Young Adults G and Genetics of Overweight Young Adults (GOYA) Consortium and EArly Genetics & Lifecourse Epidemiology (EAGLE) Consortium and Institutionen för kliniska vetenskaper, Avdelningen för obstetrik och gynekologi and Sahlgrenska akademin and Göteborgs universitet and Gothenburg University and Institute of Clinical Sciences, Department of Obstetrics and Gynecology and Sahlgrenska Academy
Nature genetics, ISSN 1546-1718, 12/2011, Volume 44, Issue 2, pp. 187 - 192
Journal Article
Journal Article
American journal of human genetics, ISSN 0002-9297, 2012, Volume 90, Issue 5, pp. 784 - 795
Psoriasis is a common, immune-mediated genetic disorder of the skin and is associated with arthritis in approximately 30% of cases... 
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Haiti | Up-Regulation | Exons | Arthritis, Psoriatic - physiopathology | Humans | Child, Preschool | Molecular Sequence Data | NF-kappa B - metabolism | Gene Expression Profiling | Chemokine CCL20 | Genetic Loci | Chromosomes, Human, Pair 17 - genetics | CARD Signaling Adaptor Proteins - genetics | CARD Signaling Adaptor Proteins - metabolism | Cloning, Molecular | HEK293 Cells | Female | Membrane Proteins - metabolism | Amino Acid Sequence | Chromosomes, Human, Pair 17 - metabolism | Epidermis - metabolism | Genetic Predisposition to Disease | Membrane Proteins - genetics | Europe | Guanylate Cyclase - metabolism | Transcription Factors - genetics | Sequence Analysis, DNA | Arthritis, Psoriatic - genetics | Proteins - genetics | Transcription Factors - metabolism | Proteins - metabolism | NF-kappa B - genetics | Keratinocytes - metabolism | Pedigree | Taiwan | Guanylate Cyclase - genetics | Skin | Mutation | Genome, Human | Europeans | Gene mutations | Genetic susceptibility | Psoriasis | Genetic aspects | Research | Nucleotide sequencing | DNA sequencing | Diseases | Genomics | Medical genetics | Splicing | Susceptibility | Caspase | Keratinocytes | Epidermis | Arthritis | Inflammation | NF- Kappa B protein | CCL20 protein | Skin diseases | genomics | Age | Chemokines | Injuries | Interleukin 8
Journal Article
PloS one, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, p. e9258
Background: The Notch pathway is essential for proper epidermal differentiation during embryonic skin development. Moreover, skin specific loss of Notch... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Dermatitis, Atopic - genetics | Skin - metabolism | Humans | Receptor, Notch2 - genetics | Receptors, Notch - genetics | Myeloproliferative Disorders - genetics | Granulocyte Colony-Stimulating Factor - genetics | Dermatitis, Atopic - mortality | Flow Cytometry | Receptors, Cytokine - metabolism | Receptors, Cytokine - genetics | Granulocyte Colony-Stimulating Factor - metabolism | Skin - pathology | Immunoglobulins | Cytokines - metabolism | Mice, Inbred C57BL | Receptors, Notch - physiology | Mice, Transgenic | Survival Rate | Signal Transduction - genetics | Dermatitis, Atopic - physiopathology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Receptor, Notch2 - physiology | Animals | Mice, Nude | Models, Biological | Survival Analysis | Myeloproliferative Disorders - physiopathology | Signal Transduction - physiology | Mice | Skin - physiopathology | Receptor, Notch1 - genetics | Myeloproliferative Disorders - mortality | Receptor, Notch1 - physiology | Embryonic development | Psoriasis | Atopic dermatitis | Bone marrow | Transplantation | Skin | B cells | Syngeneic grafts | Pathogenesis | Bone marrow transplantation | Homeostasis | Lichen planus | Inactivation | Dermatitis | Thymus | Signal transduction | Embryogenesis | Biomedical materials | Immunology | Granulocytes | Granulocyte colony-stimulating factor | Lymphocytes | Rodents | Biocompatibility | Drug dosages | Osteopenia | Neutropenia | Spleen | Repressing | Deactivation | Cytokines | Developmental biology | Dermatology | Leukocytes (eosinophilic) | Neutrophils | Keratinocytes | Breast cancer | Gene expression | Myeloproliferative diseases | Hemopoiesis | Signaling | Thymic stromal lymphopoietin | Stem cells | Mast cells | Infiltration | Notch protein | Hematopoietic system | Mutation | Eosinophils
Journal Article
PloS one, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0175153
IL-1 beta is a potent player in cutaneous inflammation and central for the development of a Th17 micro-milieu in autoinflammatory diseases including psoriasis.... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Caspases - immunology | Humans | Interleukin-17 - immunology | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | S100 Proteins - immunology | Psoriasis - genetics | S100 Proteins - genetics | Th17 Cells - drug effects | Transfection | Inflammasomes - drug effects | Psoriasis - pathology | Adaptor Proteins, Signal Transducing - immunology | Apoptosis Regulatory Proteins - genetics | Skin - pathology | Skin - immunology | Th17 Cells - pathology | Interleukin-1beta - secretion | Psoriasis - immunology | S100 Calcium Binding Protein A7 | Apoptosis Regulatory Proteins - immunology | DNA - immunology | Signal Transduction | Caspases - genetics | Cells, Cultured | Gene Expression Regulation | Interleukin-17 - genetics | Interleukin-1beta - immunology | Vitamin D - pharmacology | Keratinocytes - cytology | Keratinocytes - immunology | DNA - genetics | Interleukin-17 - antagonists & inhibitors | Keratinocytes - drug effects | Inflammasomes - immunology | Adaptor Proteins, Signal Transducing - genetics | Th17 Cells - immunology | Interferon-gamma - pharmacology | Interleukins | Physiological aspects | Development and progression | Skin diseases | Genetic aspects | Inflammation | Research | Cell culture | Transcription | Interleukin | Helper cells | Lymphocytes T | Inflammatory diseases | Vitamin D | Lymphocytes | Interleukin 1 | Lesions | Deoxyribonucleic acid--DNA | Psoriasis | Cytokines | Dermatology | Keratinocytes | Caspase | Epidermis | Hazards | Gene expression | Caspase-5 | Hospitals | Biopsy | Interferon | Autoimmune diseases | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 8, p. e71690
Journal Article