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Nature, ISSN 0028-0836, 2013, Volume 499, Issue 7459, pp. 500 - 503
Human infection associated with a novel reassortant avian influenza H7N9 virus has recently been identified in China(1). A total of 132 confirmed cases and 39... 
CELLS | OBSTRUCTIVE PULMONARY-DISEASE | TRANSMISSION | INCREASED EXPRESSION | HEMAGGLUTININ | SPECIFICITY | MULTIDISCIPLINARY SCIENCES | ADAPTATION | FERRETS | INFECTION | BINDING | Bronchi - virology | Chemokines - blood | Influenza A Virus, H5N1 Subtype - physiology | Receptors, Virus - metabolism | Humans | Trachea - virology | Zoonoses - virology | Lung - virology | China | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - cytology | Bronchi - metabolism | Influenza A virus - pathogenicity | Influenza A virus - immunology | Influenza, Human - virology | Bronchi - cytology | Cell Line | Influenza A virus - physiology | Birds - virology | Host Specificity | Influenza in Birds - transmission | Influenza, Human - blood | Cross Reactions - immunology | Organ Specificity | N-Acetylneuraminic Acid - metabolism | Zoonoses - transmission | N-Acetylneuraminic Acid - chemistry | Animals | Influenza Vaccines - immunology | Receptors, Virus - chemistry | Virus Replication | Influenza A Virus, H5N1 Subtype - immunology | Epithelial Cells - virology | Influenza in Birds - virology | Pulmonary Alveoli - virology | Antibodies, Viral - immunology | N-Acetylneuraminic Acid - analogs & derivatives | In Vitro Techniques | Influenza, Human - immunology | Avian influenza | Host-parasite relationships | Physiological aspects | Research | Health aspects | Chemokines | Protein binding | Pandemics | Cytokines | Viruses | Infections | Preferences | Age | Viral infections
Journal Article
Nature, ISSN 0028-0836, 01/2015, Volume 517, Issue 7536, pp. 616 - 620
Journal Article
Journal Article
The FEBS Journal, ISSN 1742-464X, 05/2018, Volume 285, Issue 9, pp. 1611 - 1634
The rapidly evolvable influenza A virus has caused pandemics linked to millions of deaths in the past century. Influenza A viruses are categorized by H... 
N‐glycans | short/long receptors | influenza viruses | human alveoli | hemagglutinins | N-glycans | PIGS | SUGAR CHAINS | SWINE | BIOCHEMISTRY & MOLECULAR BIOLOGY | long receptors | SIALIC-ACID | H1N1 | H3N2 | GLYCOLYLNEURAMINIC ACID | A(H3N2) VIRUS | short | RECEPTORS | BINDING | Influenza A virus - chemistry | Poultry Diseases - pathology | Influenza, Human - pathology | Pandemics | Swine Diseases - virology | Humans | Influenza A virus - genetics | Pulmonary Alveoli - chemistry | Sialic Acids - chemistry | Ducks | Swine Diseases - epidemiology | Hemagglutinin Glycoproteins, Influenza Virus - genetics | RNA, Viral - genetics | Disease Outbreaks | Swine | Madin Darby Canine Kidney Cells | Polysaccharides - chemistry | Influenza, Human - virology | Seasons | Poultry Diseases - epidemiology | Viral Tropism - physiology | Binding, Competitive | Carbohydrate Sequence | Amino Acid Sequence | Hemagglutinin Glycoproteins, Influenza Virus - chemistry | Influenza A virus - physiology | Pulmonary Alveoli - pathology | Orthomyxoviridae Infections - pathology | Swine Diseases - pathology | Influenza, Human - epidemiology | Influenza in Birds - epidemiology | Poultry Diseases - virology | Zoonoses | Orthomyxoviridae Infections - epidemiology | Influenza in Birds - pathology | Animals | Receptors, Virus - chemistry | Polysaccharides - physiology | Virus Replication | Dogs | Influenza in Birds - virology | Protein Binding | Pulmonary Alveoli - virology | Orthomyxoviridae Infections - virology | Amino Acid Substitution | Influenza viruses | Epidemics | Polysaccharides | Disease transmission | Analysis | Influenza | Lectins | Health aspects | Tropism | Pathogenesis | Amino acid sequence | Viruses | Outbreaks | Infections | Vaccines | Cell surface | Molecular chains | Glycan | Human populations | Respiratory tract | Proteins | Receptors | Exo-a-sialidase | Hemagglutinins | Alveoli
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2017, Volume 198, Issue 4, pp. 1616 - 1626
Human infections with highly pathogenic avian influenza A (H5N1) virus are frequently fatal but the mechanisms of disease remain ill-defined. H5N1 infection is... 
A H5N1 | NONHUMAN-PRIMATES | UNITED-STATES | EPITHELIAL-CELLS | DENDRITIC CELLS | HUMAN MACROPHAGES | DISEASE SEVERITY | IMMUNOLOGY | RHESUS MACAQUE | PATHOGENIC AVIAN INFLUENZA | IN-VIVO CHARACTERIZATION | Macrophages, Alveolar - pathology | Respiratory Distress Syndrome, Adult - physiopathology | Influenza A Virus, H5N1 Subtype - physiology | Orthomyxoviridae Infections - physiopathology | Alveolar Epithelial Cells - virology | Macaca fascicularis | Alveolar Epithelial Cells - immunology | Respiratory Distress Syndrome, Adult - immunology | Lung - virology | Alveolar Epithelial Cells - pathology | Macrophages, Alveolar - immunology | Cytokines - immunology | Disease Models, Animal | Macrophages, Alveolar - virology | Cells, Cultured | Pneumonia, Viral - immunology | Immunity, Innate - immunology | Animals | Virus Replication | Influenza A Virus, H5N1 Subtype - immunology | Respiratory Distress Syndrome, Adult - virology | Aerosols | Pulmonary Alveoli - virology | Influenza A Virus, H5N1 Subtype - pathogenicity | Orthomyxoviridae Infections - virology | Pneumonia, Viral - virology | Cytokines - biosynthesis | Lung - immunology | Orthomyxoviridae Infections - immunology | Pandemics | Epithelial cells | Viruses | Infections | Macrophages | Avian flu | Respiratory tract | Proteins | Inoculation | Primates | Alveoli | Trachea | Cytokines | Leukocytes (neutrophilic) | Exposure | Inflammation | Fatal | Inhalation | Respiratory distress syndrome | Lungs | Influenza | Replication | Interferon | Respiration | Apoptosis
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2017, Volume 19, Issue 8, pp. 904 - 914
After influenza infection, lineage-negative epithelial progenitors (LNEPs) exhibit a binary response to reconstitute epithelial barriers: activating a... 
PROGENITOR CELLS | BREAST-CANCER | INDUCIBLE FACTOR-1-ALPHA | AIRWAY STEM-CELLS | ADULT LUNG | IN-VIVO | NOTCH | MICE | BETA-CATENIN | PULMONARY-FIBROSIS | CELL BIOLOGY | Cell Proliferation | Influenza, Human - pathology | Receptors, Notch - metabolism | Humans | Male | Gene Expression Profiling | Orthomyxoviridae Infections - genetics | SOXB1 Transcription Factors - metabolism | SOXB1 Transcription Factors - genetics | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cell Transdifferentiation | Influenza, Human - virology | Single-Cell Analysis | Wnt Signaling Pathway | Disease Models, Animal | Hypoxia - virology | Tumor Suppressor Proteins - metabolism | Pulmonary Alveoli - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Orthomyxoviridae Infections - pathology | Epithelial Cells - pathology | Genotype | Mice, Transgenic | Cell Lineage | Phenotype | Hypoxia - pathology | Epithelial Cells - virology | Orthomyxoviridae Infections - virology | Cell Movement | Epithelial Cells - metabolism | Phosphoproteins - metabolism | Oxygen - metabolism | Hypoxia - metabolism | Pulmonary Alveoli - metabolism | Tumor Suppressor Proteins - genetics | Trans-Activators - genetics | Female | Influenza, Human - metabolism | Orthomyxoviridae Infections - metabolism | Cells, Cultured | Influenza A Virus, H1N1 Subtype - pathogenicity | Keratin-5 - genetics | Phosphoproteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Regeneration | Hypoxia - genetics | Animals | Influenza, Human - genetics | Keratin-5 - metabolism | Pulmonary Alveoli - virology | Trans-Activators - metabolism | Hypoxia-inducible factors | Cytokeratin | Respiratory function | Wnt protein | Transcription | Migration | Protein C | Surfactant protein C | Metaplasia | β-catenin | Signal transduction | Signaling | Clonal deletion | Lungs | Influenza | Fibrosis | Deletion | Hypoxia | Notch protein | Alveoli | Differentiation
Journal Article
PLoS pathogens, ISSN 1553-7366, 2011, Volume 7, Issue 1, p. e1001263
Journal Article