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Journal of lipid research, ISSN 0022-2275, 06/2012, Volume 53, Issue 6, pp. 1093 - 1105
Journal Article
The Journal of thoracic and cardiovascular surgery, ISSN 0022-5223, 2013, Volume 146, Issue 3, pp. 522 - 529
Objectives Continuous flow in the Fontan circulation results in impairment of pulmonary artery endothelial function, increased pulmonary arterial resistance, and, potentially, late failure of Fontan circulation... 
Cardiothoracic Surgery | SVC | eNOS | nitric oxide | pulmonary artery | 50% of the maximal response | PVP | PVR | maximal response | pulmonary vein pressure | endothelial nitric oxide synthase | superior vena cava | pulmonary vascular resistance | pulmonary artery pressure | Emax | PAP | SURGERY | CARDIAC & CARDIOVASCULAR SYSTEMS | RABBIT LUNGS | RESPIRATORY SYSTEM | NITRIC-OXIDE SYNTHASE | VASCULAR-RESISTANCE | VASODILATION | RELEASE | BLOOD-FLOW | HYPERTENSION | EXPRESSION | BIDIRECTIONAL CAVOPULMONARY SHUNT | Hypertension, Pulmonary - physiopathology | Endothelium, Vascular - drug effects | Fontan Procedure - adverse effects | Pulmonary Artery - surgery | Endothelium, Vascular - surgery | Vasodilation | Dose-Response Relationship, Drug | Microcirculation | Pulmonary Artery - metabolism | Pulsatile Flow - drug effects | Time Factors | Swine | Models, Animal | Nitric Oxide Synthase Type III - metabolism | Vascular Resistance | Vasodilator Agents - pharmacology | Endothelium, Vascular - physiopathology | Pulmonary Artery - physiopathology | Pulmonary Artery - drug effects | Animals | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Pulmonary Circulation - drug effects | Hypertension, Pulmonary - etiology | Pulmonary Artery - pathology | Health aspects | Children | Endothelium
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 11/2009, Volume 54, Issue 19, pp. 1787 - 1794
Objectives: Our aim was to determine the effects of high-dose (2 g) nicotinic acid (NA) on progression of atherosclerosis and measures of vascular function... 
atherosclerosis | magnetic resonance imaging | carotid | HDL cholesterol | INTIMA-MEDIA THICKNESS | CARDIAC & CARDIOVASCULAR SYSTEMS | CHOLESTEROL | SAFETY | TORCETRAPIB | ENDOTHELIAL FUNCTION | HIGH-DENSITY-LIPOPROTEIN | HIGH-RESOLUTION | DOUBLE-BLIND | NIACIN NIASPAN | CORONARY-HEART-DISEASE | Tunica Media - pathology | Tunica Media - drug effects | Humans | Middle Aged | Tunica Intima - physiopathology | Carotid Artery Diseases - physiopathology | Male | Niacin - pharmacology | Niacin - administration & dosage | Niacin - therapeutic use | Time Factors | Carotid Artery Diseases - pathology | Cholesterol, LDL - blood | Female | Carotid Artery Diseases - drug therapy | Double-Blind Method | Drug Administration Schedule | Cholesterol, HDL - drug effects | Cholesterol, LDL - drug effects | Treatment Outcome | Hypolipidemic Agents - pharmacology | Tunica Intima - drug effects | Magnetic Resonance Imaging | Confounding Factors (Epidemiology) | Tunica Intima - pathology | Hypolipidemic Agents - administration & dosage | Triglycerides - blood | Cholesterol, HDL - blood | Aged | Hypolipidemic Agents - therapeutic use | Tunica Media - physiopathology | Delayed-Action Preparations | Lipoproteins | Nuclear magnetic resonance--NMR | Pulmonary arteries | Coronary vessels | Mortality | Atherosclerosis | Cardiovascular disease | Diabetes | Acute coronary syndromes | Cholesterol
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 06/2017, Volume 56, Issue 6, pp. 1208 - 1216
Journal Article
Journal Article
American journal of physiology. Lung cellular and molecular physiology, ISSN 1522-1504, 2013, Volume 304, Issue 10, pp. L678 - L688
...) and rat monocrotaline-induced PH (prevention and treatment models). Last, the biological function of the MR was analyzed in cultured distal pulmonary artery smooth muscle cells (PASMCs... 
Pulmonary vascular remodeling | Hypoxia | Spironolactone | Aldosterone | Monocrotaline | FIBROSIS | hypoxia | OXIDATIVE STRESS | PHYSIOLOGY | aldosterone | monocrotaline | ANGIOTENSIN-II | PROLIFERATION | spironolactone | HYPERTROPHIC CARDIOMYOPATHY | pulmonary vascular remodeling | RESPIRATORY SYSTEM | GROWTH | SMOOTH-MUSCLE-CELLS | GENE-TRANSCRIPTION | Fibrosis - drug therapy | Hypoxia - drug therapy | Receptors, Mineralocorticoid - metabolism | Myocytes, Smooth Muscle - pathology | Body Weight - drug effects | Fibrosis - metabolism | Hypoxia - metabolism | Pulmonary Artery - metabolism | Spironolactone - pharmacology | Arterial Pressure - drug effects | Mineralocorticoid Receptor Antagonists - pharmacology | Vascular Resistance - drug effects | Hypertension, Pulmonary - drug therapy | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Heart Ventricles - pathology | Cardiac Output - drug effects | Rats | Aldosterone - pharmacology | Hypertension, Pulmonary - metabolism | Pulmonary Artery - drug effects | Platelet-Derived Growth Factor - metabolism | Animals | Hypoxia - pathology | Cell Proliferation - drug effects | Heart Ventricles - metabolism | Mice | Fibrosis - pathology | Hypertension, Pulmonary - pathology | Ventricular Remodeling - drug effects | Pulmonary Artery - pathology | Heart Ventricles - drug effects | Prevention | Physiological aspects | Mineralocorticoids | Smooth muscle | Muscle cells | Health aspects | Pulmonary hypertension | Pulmonary artery
Journal Article
Journal of Surgical Research, ISSN 0022-4804, 2013, Volume 184, Issue 2, pp. 951 - 958
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2012, Volume 303, Issue 9, pp. 1135 - 1142
...) to increased cardiac morbidity and mortality, especially in at risk populations. However, few studies have examined the effect of PM on baseline cardiac function in otherwise healthy individuals... 
Particulates | Oxidative stress | Air pollution | Inflammation | Pressure-volume | MATTER | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | pressure-volume | particulates | CONCENTRATED AMBIENT PARTICLES | LONG-TERM EXPOSURE | ATHEROSCLEROSIS | ISCHEMIA | inflammation | IN-VIVO | CARDIOVASCULAR-DISEASE | PARTICULATE AIR-POLLUTION | PERIPHERAL VASCULAR DISEASE | air pollution | MYOCARDIAL INJURY | oxidative stress | Cardiac Output - physiology | Heart - physiology | Male | Chlorobenzenes - administration & dosage | C-Reactive Protein - metabolism | Particulate Matter - pharmacology | Chlorobenzenes - pharmacology | Models, Animal | Ventricular Function, Left - physiology | Blood Pressure - drug effects | Blood Pressure - physiology | Free Radicals - administration & dosage | Cardiac Output - drug effects | Ventricular Function, Left - drug effects | Stroke Volume - physiology | Administration, Inhalation | Rats | Stroke Volume - drug effects | Pulmonary Artery - drug effects | Rats, Sprague-Dawley | Pulmonary Artery - physiology | Animals | Heart - drug effects | Free Radicals - pharmacology | Particulate Matter - administration & dosage | Heart | Physiological aspects | Free radicals (Chemistry) | Health aspects | Pulmonary artery | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
PloS one, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e56774
Journal Article
The FEBS journal, ISSN 1742-464X, 05/2013, Volume 280, Issue 9, pp. 2042 - 2055
...‐known antineoplastic/antiproliferative effects, little is known about BU action on vascular cell dynamics... 
cell migration | sodium butyrate | Akt | cell proliferation | pulmonary artery smooth muscle cells | HYDROXAMIC ACID | APOPTOSIS | ACTIVATION | ACETYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | CANCER | THERAPY | HISTONE DEACETYLASE INHIBITORS | HYPERTENSION | EXPRESSION | Phosphorylation | Gene Expression - drug effects | Monocrotaline | Male | Receptor, Platelet-Derived Growth Factor beta - genetics | Hypertension, Pulmonary - chemically induced | Butyric Acid - pharmacology | Acetylation | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Myocytes, Smooth Muscle - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Cell Survival - drug effects | Tissue Culture Techniques | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Rats | Familial Primary Pulmonary Hypertension | Pulmonary Artery - drug effects | Morpholines | Rats, Sprague-Dawley | Animals | Cell Cycle Checkpoints - drug effects | Histone Deacetylase Inhibitors - pharmacology | Cell Proliferation - drug effects | Protein Processing, Post-Translational | Platelet-Derived Growth Factor - physiology | Hypertension, Pulmonary - pathology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Pulmonary Artery - pathology | Cell Movement | Chromones | Smooth muscle | Esters | Platelet-derived growth factor | Pulmonary hypertension | Muscular system | Sodium | Pulmonary arteries | Cells | Cell adhesion & migration
Journal Article
Respiratory research, ISSN 1465-9921, 08/2009, Volume 10, Issue 1, pp. 76 - 76
.... The objectives of this study were to evaluate the effects of long-term exposure to cigarette smoke on endothelial function and smooth muscle-cell proliferation in pulmonary arteries of guinea pigs. Methods... 
SMOOTH-MUSCLE | DEPENDENT RELAXATION | RESPIRATORY SYSTEM | NITRIC-OXIDE SYNTHASE | DISEASE | DYSFUNCTION | MILD COPD | HYPERTENSION | HYPOXIA | EXPRESSION | EXPOSURE | Immunohistochemistry | Smoking - adverse effects | Muscle, Smooth, Vascular - metabolism | Actins - metabolism