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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 01/2007, Volume 104, Issue 4, pp. 1342 - 1347
Journal Article
Cardiovascular research, ISSN 1755-3245, 12/2015, Volume 108, Issue 3, pp. 377 - 386
Neointimal hyperplasia is a common feature of fibro-proliferative vascular disease and characterizes initial stages of atherosclerosis. Neointimal lesions... 
ARTERIAL INJURY | MYOCARDIN | PULMONARY-HYPERTENSION | Neointimal hyperplasia | ATHEROSCLEROSIS | CADHERIN EXPRESSION | SMOOTH-MUSCLE-CELL | FLOW | Fibrosis | ERK5 | IN-VIVO | GROWTH | Shear stress | GENE-EXPRESSION | Endothelial-to-mesenchymal transition | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Male | Aortic Diseases - metabolism | Neointima | Transfection | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Swine | MAP Kinase Kinase 5 - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Carotid Artery Diseases - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | MAP Kinase Kinase 5 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Aorta, Thoracic - physiopathology | Plaque, Atherosclerotic | Regional Blood Flow | Mechanotransduction, Cellular | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Carotid Arteries - physiopathology | Carotid Artery Diseases - genetics | Human Umbilical Vein Endothelial Cells - pathology | Carotid Arteries - pathology | Endothelial Cells - pathology | Index Medicus
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 07/2008, Volume 100, Issue 1, pp. 101 - 109
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 10/2016, Volume 311, Issue 4, pp. H944 - H957
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2012, Volume 302, Issue 3, pp. 688 - 696
...) alleviates ROS generation, maintains MMP/TIMP balance, and regresses interstitial fibrosis, we used a mouse model of pulmonary artery constriction (PAC... 
Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Folic Acid - pharmacology | Reactive Oxygen Species - metabolism | Hypertension, Pulmonary - physiopathology | Male | Autophagy - physiology | Autophagy - drug effects | Hypertrophy, Right Ventricular - drug therapy | Hypertrophy, Right Ventricular - physiopathology | Matrix Metalloproteinase 9 - metabolism | Pulmonary Artery - metabolism | Hypertrophy, Right Ventricular - pathology | Hypertension, Pulmonary - drug therapy | Hypertrophy, Right Ventricular - metabolism | Heart Ventricles - pathology | Disease Models, Animal | Biomarkers - metabolism | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Ventricular Remodeling - physiology | Vitamin B Complex - pharmacology | Tissue Inhibitor of Metalloproteinases - metabolism | Matrix Metalloproteinase 13 - metabolism | Animals | Heart Ventricles - physiopathology | Heart Ventricles - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Hypertension, Pulmonary - pathology | Ventricular Remodeling - drug effects | Pulmonary Artery - pathology | Autophagy (Cytology) | Physiological aspects | Development and progression | Research | Health aspects | Metalloproteins | Pulmonary hypertension | Heart ventricle, Right | Proteins | Oxidative stress | Cardiovascular disease | Autophagy | Rodents | Index Medicus | Muscle Mechanics and Ventricular Function | matrix metalloproteinase | mitophagy | p62 | LC3A | tissue inhibitor of metalloproteinase
Journal Article
Molecular and cellular biochemistry, ISSN 1573-4919, 12/2018, Volume 455, Issue 1-2, pp. 169 - 183
Abnormal proliferation of pulmonary artery smooth muscle cells (PASMCs) plays a dominant role in the development of pulmonary arterial hypertension (PAH... 
Life Sciences | Biochemistry, general | Medical Biochemistry | Oncology | Pulmonary arterial hypertension | Cardiology | AMP-activated protein kinase | Pulmonary artery smooth muscle cell | Carnitine palmitoyltransferase 1 | p21 | Life Sciences & Biomedicine | Science & Technology | Cell Biology | AMP-Activated Protein Kinases - metabolism | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Signal Transduction | Myocytes, Smooth Muscle - pathology | Tumor Suppressor Protein p53 - metabolism | Rats | Male | Hypertension, Pulmonary - metabolism | Rats, Sprague-Dawley | Carnitine O-Palmitoyltransferase - antagonists & inhibitors | Muscle, Smooth, Vascular - pathology | Pulmonary Artery - metabolism | Animals | Carnitine O-Palmitoyltransferase - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Hypertension, Pulmonary - drug therapy | Hypertension, Pulmonary - pathology | Myocytes, Smooth Muscle - metabolism | Pulmonary Artery - pathology | Ethylenediaminetetraacetic acid | Proteins | Platelet-derived growth factor | Smooth muscle | Fatty acids | Adenosine triphosphate | Pulmonary hypertension | Carnitine | Cell proliferation | Phosphorylation | p53 Protein | Monocrotaline | Arteries | Time dependence | Mitochondria | Cell cycle | Oxidation | Growth factors | Hypertension | Pulmonary arteries | Muscles | Metabolism | Palmitoyltransferase | Platelet-derived growth factor BB | Pulmonary artery | Carnitine palmitoyltransferase | Cyclin-dependent kinase inhibitor p21 | Lungs | Catabolism | ATP | Veins & arteries | Index Medicus
Journal Article
Journal of Vascular Research, ISSN 1018-1172, 10/2009, Volume 46, Issue 6, pp. 561 - 571
... 30, 2009 Effects of Pathological Flow on Pulmonary Artery Endothelial Production of Vasoactive Mediators and Growth Factors Min Li a, b Kurt R. Stenmark a Robin... 
Research Paper | Vascular remodeling | Shear stress | Endothelin | Vascular tone | Nitric oxide | Pulmonary hypertension | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Endothelial Growth Factors - metabolism | Phosphorylation | Thromboxane B2 - metabolism | Cell Proliferation | Cadherins - metabolism | Muscle, Smooth, Vascular - metabolism | Pulsatile Flow | Coculture Techniques | Actins - metabolism | Myocytes, Smooth Muscle - pathology | Hypertension, Pulmonary - physiopathology | Stress, Mechanical | Vascular Endothelial Growth Factor A - metabolism | Myosin Heavy Chains - metabolism | Antigens, CD - metabolism | Pulmonary Artery - metabolism | Cattle | Vasodilator Agents - metabolism | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Myocytes, Smooth Muscle - metabolism | Animals, Newborn | Endothelial Cells - metabolism | Endothelin-1 - metabolism | Cells, Cultured | Hypertension, Pulmonary - metabolism | Prostaglandins F - metabolism | Mechanotransduction, Cellular | Muscle, Smooth, Vascular - pathology | Animals | Vasoconstrictor Agents - metabolism | Endothelial Cells - pathology | Nitric Oxide - metabolism | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Index Medicus
Journal Article