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AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, ISSN 1044-1549, 12/2013, Volume 49, Issue 6, pp. 1135 - 1145
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 309, Issue 10, pp. H1679 - H1696
Normal pregnancy is associated with adaptive hemodynamic, hormonal, and vascular changes, and estrogen (E-2) may promote vasodilation during pregnancy;... 
Vascular smooth muscle | Calcium | Nitric oxide | Estrogen | Endothelium | endothelium | estrogen | nitric oxide | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | vascular smooth muscle | calcium | CA2+ ENTRY MECHANISMS | EPICARDIAL CORONARY-ARTERIES | MESENTERIC MICROVESSELS | VASCULAR SMOOTH-MUSCLE | ENDOTHELIAL NITRIC-OXIDE | GENDER-SPECIFIC REDUCTION | IN-VITRO | FEMALE RAT | PULMONARY-ARTERY ENDOTHELIUM | PERIPHERAL VASCULAR DISEASE | SIGNAL-REGULATED KINASE | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Immunohistochemistry | Mesenteric Arteries - physiology | Receptors, G-Protein-Coupled - metabolism | Calcium - metabolism | Endothelium, Vascular - drug effects | Estrogen Receptor beta - metabolism | Estrogen Receptor alpha - agonists | Potassium Channel Blockers - pharmacology | Muscle, Smooth, Vascular - drug effects | Phenols - pharmacology | Enzyme Inhibitors - pharmacology | Aorta, Thoracic - metabolism | Rats | Arteries - drug effects | Mesenteric Arteries - metabolism | Vasoconstriction - drug effects | Blotting, Western | Endothelium, Vascular - metabolism | Carotid Arteries - physiology | Vasodilation - drug effects | Adaptation, Physiological | Blood Pressure | Tetraethylammonium - pharmacology | Muscle, Smooth, Vascular - metabolism | Nitric Oxide Synthase - antagonists & inhibitors | Mesenteric Arteries - drug effects | Receptors, G-Protein-Coupled - agonists | Arteries - metabolism | Endothelium, Vascular - physiology | Vasoconstriction - physiology | Estrogen Receptor beta - agonists | Pregnancy - metabolism | Arteries - physiology | Aorta, Thoracic - physiology | Estrogen Receptor alpha - metabolism | Female | Renal Artery - metabolism | Aorta, Thoracic - drug effects | Vasodilation - physiology | Estrogens - metabolism | Muscle, Smooth, Vascular - physiology | Pregnancy - physiology | Pyrazoles - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Renal Artery - physiology | Animals | Renal Artery - drug effects | Phenols | G proteins | Research | Pregnant women | Health aspects | Membrane proteins | Pregnancy | Genomics | Protein expression | Physiology | Hormones | Vascular Biology and Microcirculation
Journal Article
Cell Proliferation, ISSN 0960-7722, 08/2016, Volume 49, Issue 4, pp. 484 - 493
Journal Article
FEBS Letters, ISSN 0014-5793, 01/2016, Volume 590, Issue 1, pp. 101 - 109
Pulmonary arterial hypertension ( PAH ) is characterized by excessive pulmonary arterial smooth muscle cells ( PASMC s) growth, partially in response to PDGF ‐... 
smooth muscle cells | Wnt signaling | vascular remodeling | pulmonary disease | PDGF | pulmonary hypertension | Proto-Oncogene Proteins c-sis - chemistry | Muscle, Smooth, Vascular - metabolism | Humans | Glycogen Synthase Kinase 3 beta | Wnt-5a Protein | Promoter Regions, Genetic - drug effects | Wnt Proteins - metabolism | Familial Primary Pulmonary Hypertension - metabolism | Pulmonary Artery - metabolism | Wnt Proteins - genetics | Protein Processing, Post-Translational - drug effects | RNA Interference | Phosphorylation - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Muscle, Smooth, Vascular - drug effects | Familial Primary Pulmonary Hypertension - pathology | Proto-Oncogene Proteins - metabolism | Recombinant Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-sis - metabolism | beta Catenin - agonists | Cells, Cultured | Proto-Oncogene Proteins - genetics | Pulmonary Artery - drug effects | Recombinant Proteins - pharmacology | Glycogen Synthase Kinase 3 - metabolism | Muscle, Smooth, Vascular - cytology | beta Catenin - metabolism | beta Catenin - genetics | Pulmonary Artery - cytology | Gene Expression Regulation - drug effects | Muscle, Smooth, Vascular - pathology | Wnt Signaling Pathway - drug effects | beta Catenin - antagonists & inhibitors | Active Transport, Cell Nucleus - drug effects | Anticoagulants - pharmacology | Genes, Reporter - drug effects | Cell Proliferation - drug effects | Wnt Proteins - antagonists & inhibitors | Pulmonary Artery - pathology
Journal Article
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2017, Volume 313, Issue 2, pp. L371 - L383
Pulmonary hypertension (PH) is a progressive disorder whose cellular pathogenesis involves enhanced smooth muscle cell (SMC) proliferation and resistance to... 
Smooth muscle cell proliferation | Pulmonary hypertension | PHYSIOLOGY | pulmonary hypertension | HOMOLOG | KAPPA-B | AP-1 | CANCER | smooth muscle cell proliferation | PATHOGENESIS | RESPIRATORY SYSTEM | PDCD4 | PPAR-GAMMA | MIR-21 | NOX4 EXPRESSION | HYPERTENSION | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | Humans | Annexin A5 - genetics | Apoptosis - genetics | Male | MicroRNAs - metabolism | PPAR gamma - metabolism | Pulmonary Artery - metabolism | Apoptosis Regulatory Proteins - genetics | Lung - metabolism | Myocytes, Smooth Muscle - drug effects | Thiazolidinediones - pharmacology | Myocytes, Smooth Muscle - metabolism | PPAR gamma - genetics | Cell Proliferation - genetics | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Cells, Cultured | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Signal Transduction - genetics | Pulmonary Artery - drug effects | Apoptosis Regulatory Proteins - metabolism | Gene Expression Regulation - drug effects | Annexin A5 - metabolism | Animals | Signal Transduction - drug effects | Lung - drug effects | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Apoptosis - physiology | RNA-Binding Proteins - metabolism | Cell proliferation | Flow cytometry | Transcription factors | MiRNA | Smooth muscle | siRNA | Muscular system | Respiratory system | Pulmonary artery | Lungs | Cell death | Derepression | MicroRNAs | Transcription activation | Rosiglitazone | Tumor suppressor genes | Hypoxia | Annexin V | Apoptosis
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2014, Volume 189, Issue 3, pp. 314 - 324
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2009, Volume 15, Issue 11, pp. 1289 - 1297
Notch receptor signaling is implicated in controlling smooth muscle cell proliferation and in maintaining smooth muscle cells in an undifferentiated state.... 
MEDICINE, RESEARCH & EXPERIMENTAL | TGF-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | VASCULAR MORPHOGENESIS | CELL BIOLOGY | CROSS-TALK | IN-VITRO | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | EXPRESSION | NEOINTIMAL FORMATION | INTRACELLULAR DOMAIN | Hypoxia - drug therapy | Microscopy, Electron, Transmission - methods | Receptors, Notch - metabolism | Humans | RNA, Messenger - metabolism | Pulmonary Artery - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Time Factors | Hypertension, Pulmonary - drug therapy | Lung - metabolism | Receptors, Notch - deficiency | Repressor Proteins - metabolism | Disease Models, Animal | Lung - pathology | Dipeptides - therapeutic use | Dipeptides - pharmacology | Enzyme Inhibitors - pharmacology | Gene Expression Regulation - physiology | Rats | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Signal Transduction - genetics | Enzyme Inhibitors - therapeutic use | Mice, Knockout | Gene Expression Regulation - drug effects | Pulmonary Artery - ultrastructure | Animals | Myocytes, Smooth Muscle | Signal Transduction - drug effects | Hypoxia - physiopathology | Receptor, Notch3 | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | In Vitro Techniques | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Cell proliferation | Molecular genetics | Development and progression | Genetic aspects | Vascular smooth muscle | Research | Properties | Health aspects | Pulmonary hypertension | Hypertension | Signal transduction | Muscular system | Cellular biology | Pulmonary arteries
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2013, Volume 49, Issue 4, pp. 609 - 618
Journal Article