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Journal Article
Circulation, ISSN 0009-7322, 11/2009, Volume 120, Issue 20, pp. 1951 - 1960
Background-The most important determinant of longevity in pulmonary arterial hypertension is right ventricular (RV) function, but in contrast to experimental... 
Heart failure | Angiogenesis | Microcirculation | Pressure | Pulmonary heart disease | pulmonary heart disease | OVERLOAD | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | angiogenesis | pressure | microcirculation | PROGNOSTIC VALUE | MYOSIN HEAVY-CHAIN | ANTIOXIDANT RESPONSE ELEMENT | TRANSCRIPTION FACTOR NRF2 | VENTRICULAR HYPERTROPHY | CARDIAC-HYPERTROPHY | DISEASE | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | HYPERTENSION | HEMATOLOGY | Blood Pressure | Vascular Endothelial Growth Factor A - biosynthesis | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | Male | RNA, Messenger - biosynthesis | Hypertrophy, Right Ventricular - physiopathology | Pulmonary Artery - metabolism | Heme Oxygenase (Decyclizing) - biosynthesis | Hypertrophy, Right Ventricular - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Hypertrophy, Right Ventricular - metabolism | Disease Models, Animal | Gene Expression Regulation | Rats | Heart Failure - metabolism | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Heart Failure - pathology | Rats, Sprague-Dawley | Animals | Fibrosis | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right | Apoptosis | Usage | Animal models in research | Heart ventricle, Left | Physiological aspects | Diagnosis | Pulmonary hypertension | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2015, Volume 21, Issue 7, pp. 777 - 785
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2014, Volume 34, Issue 8, pp. 1704 - 1715
OBJECTIVE—Pulmonary hypertension (PH) is a progressive disease arising from remodeling and narrowing of pulmonary arteries (PAs) resulting in high pulmonary... 
Adventitia | Fibroblast | NADPH oxidase | Pulmonary artery | pulmonary artery | ACTIVATION | adventitia | PROLIFERATION | FIBROBLASTS | NAD(P)H OXIDASE | fibroblast | CHRONIC HYPOXIA | MODELS | INFLAMMATION | NOX4 | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | MOLECULAR-MECHANISMS | Up-Regulation | Antihypertensive Agents - pharmacology | Fibroblasts - enzymology | Cell Proliferation | Reactive Oxygen Species - metabolism | Humans | Monocrotaline | NADPH Oxidases - metabolism | Pyrroles | Extracellular Matrix - metabolism | Male | Dose-Response Relationship, Drug | Extracellular Matrix - genetics | Transfection | Time Factors | HEK293 Cells | Adventitia - enzymology | NADPH Oxidases - genetics | Hypertrophy, Right Ventricular - pathology | Hypertension, Pulmonary - drug therapy | Indoles | Hypertension, Pulmonary - enzymology | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Familial Primary Pulmonary Hypertension | Hypertension, Pulmonary - genetics | Hypoxia - complications | Adventitia - drug effects | NADPH Oxidase 4 | Pulmonary Artery - drug effects | Fibroblasts - pathology | Hypertrophy, Right Ventricular - enzymology | Rats, Sprague-Dawley | Pulmonary Artery - enzymology | Animals | Hypertrophy, Right Ventricular - prevention & control | Adventitia - pathology | Mice | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Cell Movement | Index Medicus
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 09/2012, Volume 367, Issue 10, pp. 913 - 921
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 12/2014, Volume 190, Issue 12, pp. e67 - e68
Journal Article
Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 2, pp. 236 - 244
Inflammatory cytokine interleukin (IL)-6 is elevated in the serum and lungs of patients with pulmonary artery hypertension (PAH). Several animal models of PAH... 
Pulmonary artery hypertension | Proliferation | Interleukin-6 | CARDIAC & CARDIOVASCULAR SYSTEMS | pulmonary artery hypertension | proliferation | UP-REGULATES EXPRESSION | interleukin-6 | VASCULAR STRUCTURE | ARTERIAL-HYPERTENSION | PLEXIFORM LESIONS | ENDOTHELIAL-CELLS | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | INFLAMMATORY CYTOKINES | HEMATOLOGY | NEOINTIMAL FORMATION | TRANSGENIC MICE | Blood Pressure | Up-Regulation | Cell Proliferation | Microtubule-Associated Proteins - metabolism | Arterioles - immunology | Hyperplasia | Repressor Proteins | Hypertension, Pulmonary - physiopathology | Muscle, Smooth, Vascular - immunology | Vascular Endothelial Growth Factor A - metabolism | Muscle, Smooth, Vascular - physiopathology | Hypertrophy, Right Ventricular - physiopathology | Time Factors | Hypertrophy, Right Ventricular - pathology | Interleukin-6 - metabolism | Proto-Oncogene Proteins - metabolism | Vascular Resistance | Inhibitor of Apoptosis Proteins | Interleukin-6 - genetics | Mice, Inbred C57BL | Hypertension, Pulmonary - immunology | Mice, Transgenic | Hypoxia - complications | Pulmonary Artery - physiopathology | Proto-Oncogene Proteins c-myc - metabolism | Endothelial Cells - immunology | Arterioles - pathology | Muscle, Smooth, Vascular - pathology | Animals | Hypertrophy, Right Ventricular - immunology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Ventricular Pressure | Proto-Oncogene Proteins c-bcl-2 | Endothelial Cells - pathology | Chronic Disease | Hypertension, Pulmonary - pathology | Transforming Growth Factor beta - metabolism | Pulmonary Artery - pathology | Ventricular Function, Right | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | Pulmonary Artery - immunology | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 10/2015, Volume 117, Issue 10, pp. 870 - 883
RATIONALE:The pathogenesis of pulmonary arterial hypertension (PAH) remains unclear. The 4 microRNAs representing the miR-143 and miR-145 stem loops are... 
microRNAs | hypertension, pulmonary | endothelium | myocytes, smooth muscle | cell movement | exosomes | MIGRATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISM | smooth muscle | PHENOTYPE | pulmonary | CANCER | myocytes | MIR-145 | GROWTH | DISEASE | PERIPHERAL VASCULAR DISEASE | CLUSTER | HEMATOLOGY | hypertension | EXPRESSION | Exosomes - metabolism | Vascular Remodeling | Muscle, Smooth, Vascular - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Hypertension, Pulmonary - physiopathology | Male | MicroRNAs - metabolism | Muscle, Smooth, Vascular - physiopathology | Case-Control Studies | Hypertension, Pulmonary - prevention & control | Pulmonary Artery - metabolism | Arterial Pressure | Transfection | Time Factors | Cattle | Female | Binding Sites | Myocytes, Smooth Muscle - metabolism | Promoter Regions, Genetic | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Cell Communication | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Mice, Knockout | Transcription Factors - metabolism | Muscle, Smooth, Vascular - pathology | Animals | MicroRNAs - genetics | Ventricular Pressure | HeLa Cells | Endothelial Cells - pathology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right | Cell Movement | Index Medicus
Journal Article
Journal Article