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2006, Lung biology in health and disease, ISBN 9780824729813, Volume 216., xvi, 233
Book
Journal of Thoracic and Cardiovascular Surgery, The, ISSN 0022-5223, 2013, Volume 146, Issue 3, pp. 522 - 529
Objectives Continuous flow in the Fontan circulation results in impairment of pulmonary artery endothelial function, increased pulmonary arterial resistance,... 
Cardiothoracic Surgery | NO | SVC | eNOS | nitric oxide | pulmonary artery | 50% of the maximal response | PVP | PVR | maximal response | pulmonary vein pressure | PA | endothelial nitric oxide synthase | superior vena cava | pulmonary vascular resistance | pulmonary artery pressure | EC | Emax | PAP | SURGERY | CARDIAC & CARDIOVASCULAR SYSTEMS | RABBIT LUNGS | RESPIRATORY SYSTEM | NITRIC-OXIDE SYNTHASE | VASCULAR-RESISTANCE | VASODILATION | RELEASE | BLOOD-FLOW | HYPERTENSION | EXPRESSION | BIDIRECTIONAL CAVOPULMONARY SHUNT | Hypertension, Pulmonary - physiopathology | Endothelium, Vascular - drug effects | Fontan Procedure - adverse effects | Pulmonary Artery - surgery | Endothelium, Vascular - surgery | Vasodilation | Dose-Response Relationship, Drug | Microcirculation | Pulmonary Artery - metabolism | Pulsatile Flow - drug effects | Time Factors | Swine | Models, Animal | Nitric Oxide Synthase Type III - metabolism | Vascular Resistance | Vasodilator Agents - pharmacology | Endothelium, Vascular - physiopathology | Pulmonary Artery - physiopathology | Pulmonary Artery - drug effects | Animals | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Pulmonary Circulation - drug effects | Hypertension, Pulmonary - etiology | Pulmonary Artery - pathology | Health aspects | Children | Endothelium | Index Medicus | Abridged Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 02/2016, Volume 22, Issue 2, pp. 154 - 162
Although the lung can undergo self-repair after injury, fibrosis in chronically injured or diseased lungs can occur at the expense of regeneration. Here we... 
MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | ANGIOGENESIS | MACROPHAGE REGULATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | NOTCH | SIGNALING PROMOTES | CELL BIOLOGY | TO-MESENCHYMAL TRANSITION | ENDOTHELIAL-CELLS | DISEASE | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | Antibiotics, Antineoplastic - toxicity | Receptors, Notch - metabolism | Humans | Calcium-Binding Proteins - antagonists & inhibitors | Capillaries - drug effects | Hydrochloric Acid - toxicity | Smad3 Protein - metabolism | Pulmonary Circulation - physiology | Intercellular Signaling Peptides and Proteins - metabolism | Pulmonary Artery - metabolism | Receptors, CXCR - metabolism | Serrate-Jagged Proteins | Lung Injury - metabolism | Lung - metabolism | Membrane Proteins - metabolism | Pulmonary Fibrosis - metabolism | Capillaries - metabolism | Endothelial Cells - physiology | Wnt Signaling Pathway | Bleomycin - toxicity | Fibroblasts - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Lung - pathology | Endothelial Cells - metabolism | RNA, Small Interfering - pharmacology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Pulmonary Artery - drug effects | Lung - physiology | Regeneration - physiology | Macrophages - metabolism | Regeneration - drug effects | Animals | Membrane Proteins - antagonists & inhibitors | Smad3 Protein - drug effects | Fibroblasts - drug effects | Lung - drug effects | Fibrosis | Fluorescent Antibody Technique | Macrophages - drug effects | Mice | Pulmonary Circulation - drug effects | Oligopeptides - pharmacology | Receptors, CXCR - agonists | Endothelial Cells - drug effects | Physiological aspects | Regeneration (Biology) | Lung diseases | Blood vessels | Angiogenesis | Pulmonary fibrosis | Cellular biology | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 01/2013, Volume 127, Issue 1, pp. 115 - 125
Background-Evidence suggestive of endoplasmic reticulum (ER) stress in the pulmonary arteries of patients with pulmonary arterial hypertension has been... 
hypertension, pulmonary | metabolism | mitochondria | endoplasmic reticulum stress | vascular diseases | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | CLINICAL-TRIAL | CFTR FUNCTION | CHEMICAL CHAPERONES | ORAL SODIUM PHENYLBUTYRATE | HUMAN PROSTATE-CANCER | PRIMARY BILIARY-CIRRHOSIS | ARTERIAL-HYPERTENSION | IN-VIVO | PERIPHERAL VASCULAR DISEASE | CYSTIC-FIBROSIS | Cholagogues and Choleretics - pharmacology | Hypoxia - drug therapy | Apoptosis - drug effects | Male | Hypertension, Pulmonary - prevention & control | Hypoxia - metabolism | Antineoplastic Agents - metabolism | Pulmonary Circulation - physiology | Taurochenodeoxycholic Acid - pharmacology | Phenylbutyrates - metabolism | Phenylbutyrates - pharmacology | Antineoplastic Agents - pharmacology | Hypertension, Pulmonary - drug therapy | Disease Models, Animal | Cholagogues and Choleretics - metabolism | Endoplasmic Reticulum Stress - drug effects | Models, Cardiovascular | Taurochenodeoxycholic Acid - metabolism | Mice, Inbred C57BL | Rats | Hypertension, Pulmonary - metabolism | Mitochondria - drug effects | Rats, Sprague-Dawley | Activating Transcription Factor 6 - metabolism | Animals | Signal Transduction - drug effects | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | Pulmonary Circulation - drug effects | Apoptosis - physiology | Mitochondria - physiology | Chronic Disease | Endoplasmic Reticulum Stress - physiology | Care and treatment | Research | Molecular chaperones | Endoplasmic reticulum | Pulmonary hypertension | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Anesthesiology, ISSN 0003-3022, 11/1999, Volume 91, Issue 5, pp. 1408 - 1414
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 03/2012, Volume 110, Issue 5, pp. 727 - 738
RATIONALE:Stroke is the third most common cause of death in industrialized countries. The main therapeutic target is the ischemic penumbra, potentially... 
ischemic penumbra | collateral blood flow | ischemic stroke | nitric oxide inhalation | CARDIAC & CARDIOVASCULAR SYSTEMS | PLATELET-AGGREGATION | FOCAL CEREBRAL-ISCHEMIA | S-NITROSOHEMOGLOBIN | FLOW | RESPIRATORY-DISTRESS-SYNDROME | L-ARGININE | SUBARACHNOID HEMORRHAGE | PERIPHERAL VASCULAR DISEASE | RED-BLOOD-CELLS | HEMATOLOGY | HYPOXIC PULMONARY VASOCONSTRICTION | Arterioles - physiology | Neurons - pathology | Nitric Oxide - pharmacology | Humans | Male | Nitric Oxide - therapeutic use | Stroke - physiopathology | Stroke - pathology | Brain - blood supply | Regional Blood Flow - drug effects | Models, Animal | Vasodilation - physiology | Cell Survival - physiology | Collateral Circulation - drug effects | Cell Survival - drug effects | Collateral Circulation - physiology | Stroke - prevention & control | Mice, Inbred C57BL | Administration, Inhalation | Brain Ischemia - physiopathology | Brain Ischemia - prevention & control | Nitric Oxide - administration & dosage | Arterioles - drug effects | Animals | Brain Ischemia - pathology | Sheep | Mice | Vasodilation - drug effects | Regional Blood Flow - physiology | Animal models | Oxygen | Stroke | Cell survival | Arterioles | Inhalation | Reperfusion | Ischemia | Microscopy | Cerebral blood flow | Perfusion | Nitric oxide | Brain injury | Index Medicus | Brain | Brain Ischemia | pathology | Collateral Circulation | Vasodilation | administration & dosage | pharmacology | Neurologi | Administration | Cell Survival | Neurons | physiology | drug effects | prevention & control | Nitric Oxide | Regional Blood Flow | Inbred C57BL | Neurology | physiopathology | blood supply | Animal | Models | therapeutic use
Journal Article
Circulation: Heart Failure, ISSN 1941-3289, 09/2012, Volume 5, Issue 5, pp. 627 - 634
Background-TRV120027 is a novel beta-arrestin biased ligand of the angiotensin II type 1 receptor; it antagonizes canonical G-protein-mediated coupling while,... 
Heart failure | Receptors | Animal models of human disease | Cardiovascular pharmacology | β-arrestin | Angiotensin II | PATHWAYS | heart failure | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | animal models of human disease | receptors | BETA-ARRESTIN-2 | beta-arrestin | KINASE | ADRENERGIC-RECEPTOR | cardiovascular pharmacology | BLOOD-PRESSURE | INHIBITION | angiotensin II | Furosemide - administration & dosage | Heart Failure - physiopathology | Urodynamics - drug effects | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Arrestins - metabolism | Atrial Natriuretic Factor - blood | Glomerular Filtration Rate - drug effects | Kidney - metabolism | Time Factors | Arterial Pressure - drug effects | Vascular Resistance - drug effects | Renal Circulation - drug effects | Receptor, Angiotensin, Type 1 - drug effects | Drug Therapy, Combination | Kidney - physiopathology | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Disease Models, Animal | Heart - physiopathology | Diuretics - administration & dosage | Kidney - drug effects | Natriuresis - drug effects | Diuretics - pharmacology | Heart Failure - metabolism | Cardiac Pacing, Artificial | Heart Failure - drug therapy | Diuresis - drug effects | Animals | Signal Transduction - drug effects | beta-Arrestins | Receptor, Angiotensin, Type 1 - metabolism | Dogs | Pulmonary Wedge Pressure - drug effects | Heart - drug effects | Ligands | Oligopeptides - administration & dosage | Infusions, Intravenous | Furosemide - pharmacology | Oligopeptides - pharmacology | Index Medicus
Journal Article
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 06/2011, Volume 183, Issue 12, pp. 1723 - 1729
Journal Article