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European Respiratory Journal, ISSN 0903-1936, 05/2017, Volume 49, Issue 5, pp. 1700545 - 1700545
In 1980, the question “Is there a fibrotic gene?” was posed in an editorial [1] commenting on a report about pulmonary fibrosis in monozygotic twins. Over the... 
INTERSTITIAL LUNG-DISEASE | MORTALITY | DRIVEN | TELOMERE DYSFUNCTION | PNEUMONIA | RESPIRATORY SYSTEM | Pneumonia | Lung diseases | Hypersensitivity | Genes | Protein C | Genomes | Surfactant protein C | Pulmonary fibrosis | Pneumonitis | Fibrosis | Mutation | Alveoli | Alveolitis | DNA sequencing
Journal Article
Lancet, The, ISSN 0140-6736, 2009, Volume 373, Issue 9678, pp. 1891 - 1904
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2010, Volume 207, Issue 8, pp. 1589 - 1597
Uncontrolled extracellular matrix production by fibroblasts in response to tissue injury contributes to fibrotic diseases, such as idiopathic pulmonary... 
MEDICINE, RESEARCH & EXPERIMENTAL | PHOSPHATASE | MECHANISMS | GROWTH-FACTOR | IMMUNOLOGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | MICRORNA | Oligonucleotides - genetics | Pulmonary Fibrosis - therapy | Idiopathic Pulmonary Fibrosis - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Actins - metabolism | MicroRNAs - metabolism | Pulmonary Fibrosis - genetics | Idiopathic Pulmonary Fibrosis - metabolism | Actins - genetics | Antisense Elements (Genetics) - therapeutic use | Collagen - genetics | Lung - metabolism | Phosphorylation - drug effects | Smad7 Protein - genetics | Extracellular Matrix Proteins - metabolism | Transforming Growth Factor beta1 - pharmacology | Fibroblasts - metabolism | Smad7 Protein - metabolism | Cell Line | Lung - pathology | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Smad2 Protein - metabolism | Mice, Transgenic | Pulmonary Fibrosis - pathology | Transforming Growth Factor beta1 - genetics | Fibroblasts - pathology | Fibronectins - metabolism | Collagen - metabolism | Animals | Fibroblasts - drug effects | Antisense Elements (Genetics) - genetics | Idiopathic Pulmonary Fibrosis - pathology | Pulmonary Fibrosis - chemically induced | Bleomycin - pharmacology | Fibronectins - genetics | Mice | MicroRNAs - genetics | Brief Definitive Report
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2015, Volume 308, Issue 4, pp. L344 - L357
Pathological fibrosis is driven by a feedback loop in which the fibrotic extracellular matrix is both a cause and consequence of fibroblast activation.... 
Plasminogen activator inhibitor 1 | Extracellular matrix | Mechanotransduction | Hippo | Idiopathic pulmonary fibrosis | LUNG | DOMAIN | PHYSIOLOGY | mechanotransduction | HIPPO PATHWAY | idiopathic pulmonary fibrosis | TRANSCRIPTIONAL COACTIVATOR | plasminogen activator inhibitor 1 | GROWTH-FACTOR-BETA | MECHANICAL MEMORY | MATRIX STIFFNESS | INHIBITION | RESPIRATORY SYSTEM | DIFFERENTIATION | IDIOPATHIC PULMONARY-FIBROSIS | extracellular matrix | NIH 3T3 Cells | Humans | Male | Intracellular Signaling Peptides and Proteins - metabolism | Pulmonary Fibrosis - genetics | Phosphoproteins - metabolism | Serpin E2 - biosynthesis | Gene Knockdown Techniques | Female | Lung - metabolism | Pulmonary Fibrosis - metabolism | Plasminogen Activator Inhibitor 1 - genetics | Intracellular Signaling Peptides and Proteins - genetics | Mechanotransduction, Cellular - genetics | Fibroblasts - metabolism | Lung - pathology | Pulmonary Fibrosis - pathology | Phosphoproteins - genetics | Fibroblasts - pathology | Plasminogen Activator Inhibitor 1 - biosynthesis | Serpin E2 - genetics | Animals | Transforming Growth Factor beta - genetics | Adaptor Proteins, Signal Transducing - genetics | Mice, Inbred NOD | Mice | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Transforming Growth Factor beta - metabolism | Transcriptional coactivators | Fibroblasts | Genetic aspects | Pulmonary fibrosis | Health aspects | Oncogenes | Call for Papers
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 04/2011, Volume 364, Issue 16, pp. 1503 - 1512
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 03/2014, Volume 66, Issue 3, pp. 714 - 725
Journal Article
Respirology, ISSN 1323-7799, 02/2019, Volume 24, Issue 2, pp. 97 - 98
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2009, Volume 119, Issue 4, pp. 772 - 787
Idiopathic pulmonary fibrosis (IPF) is characterized by distorted lung architecture and loss of respiratory function. Enhanced (myo)fibroblast activation, ECM... 
EPITHELIAL-MESENCHYMAL TRANSITION | PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | LUNG FIBROSIS | CELLS | APOPTOSIS | TISSUE GROWTH-FACTOR | CCN FAMILY | MECHANISMS | EXPRESSION | INTERSTITIAL PNEUMONIAS | Oncogene Proteins - genetics | Up-Regulation | Idiopathic Pulmonary Fibrosis - genetics | Epithelial Cells - metabolism | Humans | Middle Aged | Male | Pulmonary Fibrosis - genetics | Wnt Proteins - metabolism | Oncogene Proteins - physiology | CCN Intercellular Signaling Proteins | Idiopathic Pulmonary Fibrosis - etiology | Pulmonary Alveoli - metabolism | Adult | Female | Intracellular Signaling Peptides and Proteins - genetics | Pulmonary Fibrosis - etiology | Bleomycin - toxicity | Disease Models, Animal | Pulmonary Alveoli - pathology | Mice, Inbred C57BL | Oncogene Proteins - pharmacology | Epithelial Cells - pathology | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Recombinant Proteins - pharmacology | beta Catenin - metabolism | Animals | Models, Biological | Proto-Oncogene Proteins - physiology | Pulmonary Fibrosis - physiopathology | Cell Proliferation - drug effects | Idiopathic Pulmonary Fibrosis - physiopathology | Mice | Intracellular Signaling Peptides and Proteins - physiology | Cellular proteins | Pulmonary fibrosis | Genes | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Nature Genetics, ISSN 1061-4036, 05/2015, Volume 47, Issue 5, pp. 512 - 517
Journal Article