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American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 11/2010, Volume 182, Issue 10, pp. 1292 - 1304
Rationale We identified a 6 year old girl with pulmonary alveolar proteinosis (PAP), impaired granulocyte macrophage colony stimulating factor (GM CSF)... 
Surfactant | Gm-csf receptor | Alveolar macrophage | Whole lung lavage | Genetic disease | alveolar macrophage | INTERLEUKIN-3 | COLONY-STIMULATING FACTOR | GM CSF | DISORDERS | COMMON BETA-SUBUNIT | DEFICIENT MICE | GM-CSF AUTOANTIBODIES | receptor | whole lung lavage | LUNG LAVAGE | GENE | RESPIRATORY SYSTEM | genetic disease | MACROPHAGE DIFFERENTIATION | surfactant | CRITICAL CARE MEDICINE | Humans | Genetic Diseases, Inborn - genetics | Child, Preschool | Infant | Male | Dyspnea - etiology | Receptors, Granulocyte Colony-Stimulating Factor - genetics | Genetic Diseases, Inborn - therapy | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Pulmonary Alveolar Proteinosis - genetics | Receptors, Granulocyte Colony-Stimulating Factor - blood | Female | Genetic Diseases, Inborn - etiology | Child | Genetic Diseases, Inborn - diagnosis | Granulocyte-Macrophage Colony-Stimulating Factor - blood | Lung - pathology | Pulmonary Alveolar Proteinosis - therapy | Genotype | Disease Progression | Pulmonary Alveolar Proteinosis - pathology | Receptors, Granulocyte Colony-Stimulating Factor - physiology | Autoantibodies - physiology | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - physiology | Pedigree | Genetic Markers - genetics | Age of Onset | Mutation | Pulmonary Alveolar Proteinosis - diagnosis | F. Pediatrics and Lung Development | GM-CSF receptor
Journal Article
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2017, Volume 57, Issue 4, pp. 448 - 458
Maintenance of tissue-specific organ lipid compositions characterizes mammalian lipid homeostasis. The lungs and liver synthesize mixed phosphatidylcholine... 
Lipidomics | Fibrotic damage | Hepatic steatosis | Pulmonary alveolar proteinosis | Lipotoxicity | COLONY-STIMULATING FACTOR | FATTY LIVER-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | fibrotic damage | pulmonary alveolar proteinosis | hepatic steatosis | CELL BIOLOGY | LIPID-ACCUMULATION | PHOSPHATIDYLCHOLINE BIOSYNTHESIS | RESPIRATORY SYSTEM | lipotoxicity | CHOLESTEROL EFFLUX | PATHWAY ENZYMES | II CELLS | CSF KNOCKOUT MICE | lipidomics | MOLECULAR-MECHANISMS | TRANSGENIC MICE | Fatty Liver - genetics | Fatty Liver - metabolism | Liver - metabolism | Fatty Liver - complications | Male | Phosphatidylcholines - metabolism | Mice, Knockout | Organ Specificity - genetics | Phosphatidylcholines - genetics | Animals | Pulmonary Alveolar Proteinosis - genetics | Pulmonary Alveoli - metabolism | Pulmonary Alveolar Proteinosis - metabolism | Female | Mice | Pulmonary Alveolar Proteinosis - etiology | Macrophages, Alveolar - metabolism | Liver diseases | Lecithin | Granulocyte-macrophage colony-stimulating factor | Colonies | Secretion | Homeostasis | Bronchus | Lipids | Surfactants | Electron microscopy | Metabolism | Apolipoproteins | Macrophages | Fatty acids | Cholesterol | Steatosis | Signal transduction | Fatty liver | Transmission electron microscopy | Granulocytes | Lungs | Rodents | Colony-stimulating factor | Alveoli | Original Research
Journal Article
Journal Article
Revue des Maladies Respiratoires, ISSN 0761-8425, 12/2014, Volume 31, Issue 10, pp. 975 - 991
Alveolar proteinosis (AP) is a rare disease characterized by alveolar accumulation of surfactant components, which impairs gas exchange. AP is classified into... 
Rituximab | Granulocyte-macrophagecolony-stimulating factor | Pulmonary alveolar proteinosis | Surfactant | Whole lung lavage
Journal Article